Acoustic Reflex and Exchange Rate for White Noise Short Stimuli

Author(s):  
G. Rossi
1985 ◽  
Vol 99 (9) ◽  
pp. 857-863 ◽  
Author(s):  
Giovanni Rossi ◽  
Paolo Solero ◽  
M. Rolando

AbstractFor the purpose of this study, acoustic signals were generated by an Amplaid MK VI. An Amplaid 702 impedence meter was connected to its averaging section and to its computer. The stimuli were bursts of unfiltered white noise (UWN) and of narrow band white noise (NBWN; 30 db./oct/slope; central frequencies 1,000, 2,000, 4,000 Hz.) lasting 3–1,000 msec, at intensity of 105 db. SPL p.e. The following parameters were evaluated: stapedius contraction latency, amplitude, duration and recruitment time. It was found that latency was independent of the spectrum of the stimulus and its duration. Amplitude and recruitment time, on the other hand, were related to spectrum and duration, while duration of contraction was directly related to the duration of the stimulus only.


1974 ◽  
Vol 39 (1) ◽  
pp. 11-22 ◽  
Author(s):  
James Jerger ◽  
Phillip Burney ◽  
Larry Mauldin ◽  
Betsy Crump

Acoustic reflex thresholds for pure tones and white noise were used to predict severity of audiometric loss in 1043 ears with sensorineural hearing loss. Both severity and slope of loss were predicted in an additional 113 ears. Prediction was usually quite accurate. Serious errors occurred in only 4% of cases. These findings have important implications for the auditory evaluation of babies and young children.


1974 ◽  
Vol 83 (3) ◽  
pp. 406-412 ◽  
Author(s):  
Ernst-August Schnieder

After exposure to white noise (SPL > 90 dB) the cochlear half life (τ) is increased by 100%. After exposure to white noise (90 dB<SPL<105 dB) the concentration of lactate in the perilymph increases significantly, while the blood level remains constant. Both results are capable of supporting the hypothesis that exposure to noise of a critical intensity (which appears to lie beween 90 and 100 dB) reduces cochlear blood flow. This reduction in turn diminishes the exchange rate of perilymph and reduces the oxygen supply to the sensory cells. The subsequent hypoxidosis of the organ of Corti may cause metabolic breakdown in, and on prolonged exposure, degeneration of the sensory cells. Both results are further capable of supporting the hypothesis outlined in Part I, that perilymph is the medium of transport to and from the cells of the organ of Corti.


1977 ◽  
Vol 42 (4) ◽  
pp. 514-519 ◽  
Author(s):  
Dennis A. Abahazi ◽  
Herbert J. Greenberg

Acoustic reflex thresholds elicited using clinical acoustic impedance techniques were obtained on normal infants between one month and one year of age. Reflex activating signals of 500, 1000, 2000 Hz, low-pass, high-pass, and white noise were used. Less intensity was required to elicit the acoustic reflex to the noise than to the pure tones, with the 500-Hz tone requiring the greatest intensity. Inference of auditory thresholds from the acoustic reflex indicated the presence of normal hearing in the infant group.


1977 ◽  
Vol 32 (3) ◽  
pp. 761 ◽  
Author(s):  
Dennis E. Logue ◽  
Richard James Sweeney

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