Renal Cystic Diseases

2020 ◽  
pp. 319-323
Author(s):  
Alexandra Rehfuss ◽  
Christina Ching ◽  
Daniel DaJusta ◽  
Molly Fuchs
Keyword(s):  
Renal Cystic Diseases

Renal Neoplasms in Patients With Renal Cystic Diseases

1999 ◽  
Vol 161 (4) ◽  
pp. 1404-1404
Author(s):  
E.A.P. Reichard ◽  
M.A. Roubidoux ◽  
N.R. Dunnick
Keyword(s):  
Renal Neoplasms ◽  
Renal Cystic Diseases

Renal Cystic Diseases in Children — Classification

1979 ◽  
Vol 5 (5) ◽  
pp. 289-293
Author(s):  
J. Bruezière
Keyword(s):  
Renal Cystic Diseases

scholarly journals VIP17/MAL expression modulates epithelial cyst formation and ciliogenesis

2012 ◽  
Vol 303 (8) ◽  
pp. C862-C871 ◽  
Author(s):  
Vinita Takiar ◽  
Kavita Mistry ◽  
Monica Carmosino ◽  
Nicole Schaeren-Wiemers ◽  
Michael J. Caplan
Keyword(s):  
Epithelial Cells ◽  
Primary Cilium ◽  
Three Dimensional ◽  
3D Culture ◽  
Cyst Formation ◽  
Unknown Etiology ◽  
Development In Vitro ◽  
Renal Cystic Diseases

The polarized organization of epithelial cells is required for vectorial solute transport and may be altered in renal cystic diseases. Vesicle integral protein of 17 kDa (VIP17/MAL) is involved in apical vesicle transport. VIP17/MAL overexpression in vivo results in renal cystogenesis of unknown etiology. Renal cystogenesis can occur as a consequence of defects of the primary cilium. To explore the role of VIP17/MAL in renal cystogenesis and ciliogenesis, we examined the polarization and ciliary morphology of wild-type and VIP17/MAL overexpressing Madin-Darby canine kidney renal epithelial cells grown in two-dimensional (2D) and three-dimensional (3D) cyst culture. VIP17/MAL is apically localized when expressed in cells maintained in 2D and 3D culture. VIP17/MAL overexpressing cells produce more multilumen cysts compared with controls. While the distributions of basolateral markers are not affected, VIP17/MAL expression results in aberrant sorting of the apical marker gp135 to the primary cilium. VIP17/MAL overexpression is also associated with shortened or absent cilia. Immunofluorescence analysis performed on kidney sections from VIP17/MAL transgenic mice also demonstrates fewer and shortened cilia within dilated lumens ( P < 0.01). These studies demonstrate that VIP17/MAL overexpression results in abnormal cilium and cyst development, in vitro and in vivo, suggesting that VIP17/MAL overexpressing mice may develop cysts secondary to a ciliary defect.

Renal Neoplasms in Patients With Renal Cystic Diseases

1999 ◽  
pp. 1404
Author(s):  
E. A. P. Reichard ◽  
M. A. Roubidoux ◽  
N. R. Dunnick
Keyword(s):  
Renal Neoplasms ◽  
Renal Cystic Diseases

Renal cystic diseases

1997 ◽  
Vol 7 (8) ◽  
pp. 1267-1275 ◽  
Author(s):  
H. S. Thomsen ◽  
E. Levine ◽  
J. W. Meilstrup ◽  
M. A. Van Slyke ◽  
K. A. Edgar ◽  
...  
Keyword(s):  
Renal Cystic Diseases

scholarly journals Spectrum of Preneoplastic and Neoplastic Cystic Lesions of the Kidney

2012 ◽  
Vol 136 (4) ◽  
pp. 400-409 ◽  
Author(s):  
Ying-Bei Chen ◽  
Satish K. Tickoo
Keyword(s):  
English Language ◽  
Adult Population ◽  
Renal Cysts ◽  
Clinical History ◽  
Renal Tumors ◽  
Cystic Lesions ◽  
Renal Neoplasms ◽  
Clinicopathologic Characteristics ◽  
Cystic Component ◽  
Renal Cystic Diseases

Context.—Cystic lesions of the kidney may be accompanied by a range of neoplasms with distinct prognoses and future risks of developing additional tumors. In addition, some renal tumors, with or without accompanying renal cysts, may show a prominent cystic component. In the adult population, neoplasms occurring in a background of renal cystic diseases and cystic renal neoplasms often pose diagnostic challenges because of their many overlapping features. Objective.—To review the clinicopathologic characteristics of common entities in the spectrum of neoplastic and potential preneoplastic cystic lesions encountered in adults, with an emphasis on renal cystic diseases associated with tumor development and on renal neoplasms with predominantly cystic morphology. Data Sources.—The relevant English-language literature was reviewed, accompanied by the authors' experience at their practicing institution. Conclusions.—The presence of multiple renal cysts, both acquired and syndromic, can be associated with a variety of renal tumors. The morphology of the cysts and associated tumor types can help predict the genetic or acquired basis of the lesions, and particularly in specimens with no accompanying pertinent clinical history, such potential associations should be suggested in surgical pathology reports.

Renal Cystic Diseases

2006 ◽  
Vol 13 (1) ◽  
pp. 26-56 ◽  
Author(s):  
Michele Bisceglia ◽  
Carlos A Galliani ◽  
Christof Senger ◽  
Carmine Stallone ◽  
Adalberto Sessa
Keyword(s):  
Renal Cystic Diseases

Renal cystic diseases and ciliopathies

2012 ◽  
pp. 319-333
Author(s):  
Lesley Rees ◽  
Paul A. Brogan ◽  
Detlef Bockenhauer ◽  
Nicholas J.A. Webb
Keyword(s):  
Renal Cystic Diseases

Expression of Epidermal Growth Factor Receptor and Proliferative Activity of Cyst Epithelium in Human Renal Cystic Diseases

2006 ◽  
Vol 76 (3) ◽  
pp. 269-273 ◽  
Author(s):  
Eva Compérat ◽  
Sophie Ferlicot ◽  
Philippe Camparo ◽  
Pascal Eschwege ◽  
Nathalie Ba ◽  
...  
Keyword(s):  
Epidermal Growth Factor Receptor ◽  
Growth Factor ◽  
Epidermal Growth Factor ◽  
Proliferative Activity ◽  
Growth Factor Receptor ◽  
Epidermal Growth ◽  
Renal Cystic Diseases
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