Causes of Cardiac Failure and Timing of Transplantation

2018 ◽  
pp. 1-18
Author(s):  
Seth A. Hollander
Keyword(s):  
Cardiac Failure

Pathogenesis and management of sodium and water retention in cardiac failure and cirrhosis

2001 ◽  
Vol 21 (2) ◽  
pp. 157-172 ◽  
Author(s):  
Robert W. Schrier ◽  
Andrey K. Gurevich ◽  
Melissa A. Cadnapaphornchai
Keyword(s):  
Cardiac Failure ◽  
Water Retention

An unusual case of biventricular cardiac failure

2001 ◽  
Vol 56 (2) ◽  
pp. 127-129
Author(s):  
Patrick SCHOETERS ◽  
Herbert DE RAEDT ◽  
Luc COLEMONT
Keyword(s):  
Cardiac Failure ◽  
Unusual Case

Surgical Intervention in Chronic Postinfarction Cardiac Failure

Circulation ◽  
1969 ◽  
Vol 39 (5s1) ◽  
Author(s):  
MAUREEN A. HARMAN ◽  
WILLIAM A. BAXLEY ◽  
WILLIAM B. JONES ◽  
HAROLD T. DODGE ◽  
STERLING EDWARDS
Keyword(s):  
Cardiac Failure ◽  
Surgical Intervention

Regulation of Cardiac Output in Cardiac Failure

2015 ◽  
pp. 221-230
Author(s):  
R. Gourgon ◽  
J. P. Merillon ◽  
Y. Pansard ◽  
R. Prasquier ◽  
J. Y. Baglin ◽  
...  
Keyword(s):  
Cardiac Output ◽  
Cardiac Failure

scholarly journals Disrupting the LINC complex by AAV mediated gene transduction prevents progression of Lamin induced cardiomyopathy

2021 ◽  
Vol 12 (1) ◽  
Author(s):  
Ruth Jinfen Chai ◽  
Hendrikje Werner ◽  
Peter Yiqing Li ◽  
Yin Loon Lee ◽  
Khaing Thet Nyein ◽  
...  
Keyword(s):  
Cardiac Failure ◽  
Viral Vector ◽  
Nuclear Periphery ◽  
Dominant Negative ◽  
Gene Transduction ◽  
Linc Complex ◽  
Specific Expression ◽  
Common Cause ◽  
Complex Protein ◽  
One Year

AbstractMutations in the LaminA gene are a common cause of monogenic dilated cardiomyopathy. Here we show that mice with a cardiomyocyte-specific Lmna deletion develop cardiac failure and die within 3–4 weeks after inducing the mutation. When the same Lmna mutations are induced in mice genetically deficient in the LINC complex protein SUN1, life is extended to more than one year. Disruption of SUN1’s function is also accomplished by transducing and expressing a dominant-negative SUN1 miniprotein in Lmna deficient cardiomyocytes, using the cardiotrophic Adeno Associated Viral Vector 9. The SUN1 miniprotein disrupts binding between the endogenous LINC complex SUN and KASH domains, displacing the cardiomyocyte KASH complexes from the nuclear periphery, resulting in at least a fivefold extension in lifespan. Cardiomyocyte-specific expression of the SUN1 miniprotein prevents cardiomyopathy progression, potentially avoiding the necessity of developing a specific therapeutic tailored to treating each different LMNA cardiomyopathy-inducing mutation of which there are more than 450.

TETANY COMPLICATED BY CARDIAC FAILURE

1935 ◽  
Vol 1 (7) ◽  
pp. 212-213 ◽  
Author(s):  
R. W. Gibson ◽  
F. E. Gibson
Keyword(s):  
Cardiac Failure

scholarly journals Pharmacology of Cardiac Failure

1959 ◽  
Vol 52 (12) ◽  
pp. 1057-1060
Author(s):  
J. H. Burn
Keyword(s):  
Cardiac Failure
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