Placoid Retinopathies: Acute Posterior Multifocal Placoid Pigment Epitheliopathy, Serpiginous Choroiditis, Relentless Placoid Chorioretinitis, and Persistent Placoid Maculopathy

2020 ◽  
pp. 1-31
Author(s):  
Tara J. Schaab ◽  
Rukhsana G. Mirza ◽  
Lee M. Jampol
Keyword(s):  
Serpiginous Choroiditis ◽  
Relentless Placoid Chorioretinitis

scholarly journals Relentless placoid chorioretinitis: A case report

2016 ◽  
Vol 144 (9-10) ◽  
pp. 527-530
Author(s):  
Ljiljana Obradovic ◽  
Svetlana Jovanovic ◽  
Nenad Petrovic ◽  
Suncica Sreckovic ◽  
Zorica Jovanovic
Keyword(s):  
Case Report ◽  
Clinical Laboratory ◽  
Clinical Course ◽  
Laboratory Diagnostics ◽  
Intermediate Form ◽  
Serpiginous Choroiditis ◽  
Clinical Center ◽  
Retinal Distribution ◽  
The Right ◽  
Relentless Placoid Chorioretinitis

Introduction. Relentless placoid chorioretinitis is an entity which belongs to the group of an atypical intermediate form of primary inflammatory choriocapillaropathies, resembling both acute posterior multifocal placoid pigment epitheliopathy and serpiginous choroiditis, but the retinal distribution and clinical course are not the same. Because of this similarity this entity was termed ?AMPPiginous?. This entity was first described by Jones et al. in 2000. The aim of our case report is to present a very specific case where the clinical course was progressive, with loss of vision in the affected eye. Case Outline. A 31-year-old man, with no previous ophthalmic diseases, was hospitalized at the Clinic of Ophthalmology, Clinical Center Kragujevac, because of a reduction of vision in the right eye, and scotoma and metamorphopsia in the left eye. The clinical course of retinal lesions in the left eye resembled the changes observed in acute posterior multifocal placoid pigment epitheliopathy, and the right eye changes were between acute posterior multifocal placoid pigment epitheliopathy and serpiginous choroiditis. The diagnosis of relentless placoid chorioretinitis was confirmed after clinical, laboratory, immunological, virological, and angiography examinations. Conclusion. The progressive clinical course of the disease, complemented by multimodal imaging and extensive laboratory diagnostics, has led us to the diagnosis of relentless placoid chorioretinitis. The combined anti-inflammatory and immunomodulatory therapy led to the stabilization of visual acuity of the left eye as opposed to the right, where there has been no recovery.

Macular serpiginous choroiditis secondary to tuberculosis

2020 ◽  
Vol 43 (5) ◽  
pp. 449
Author(s):  
M. Tijani ◽  
N. Albaroudi ◽  
N. Boutimzine ◽  
O. Cherkaoui
Keyword(s):  
Serpiginous Choroiditis

REVIEW

2012 ◽  
Vol 16 (01) ◽  
pp. 30-40
Keyword(s):  
Human Cancer ◽  
Serpiginous Choroiditis

Human Cancer Viruses: Past, Present and Future. Serpiginous Choroiditis: An Update.

Sequential imaging in a case of relentless placoid chorioretinitis

2021 ◽  
Vol 1 (4) ◽  
pp. 675
Author(s):  
Apoorva Ayachit ◽  
Madan Joshi ◽  
Nishita Yadav ◽  
Shrinivas Joshi ◽  
Guruprasad Ayachit
Keyword(s):  
Relentless Placoid Chorioretinitis

Riding the wave: challenges in the management of serpiginous choroiditis

2014 ◽  
Vol 42 (7) ◽  
pp. 601-602
Author(s):  
Jane M Wells ◽  
Justine R Smith
Keyword(s):  
Serpiginous Choroiditis

scholarly journals Management of Uveitis-Related Choroidal Neovascularization: From the Pathogenesis to the Therapy

2014 ◽  
Vol 2014 ◽  
pp. 1-6 ◽  
Author(s):  
Enzo D’Ambrosio ◽  
Paolo Tortorella ◽  
Ludovico Iannetti
Keyword(s):  
Choroidal Neovascularization ◽  
Intravitreal Bevacizumab ◽  
Posterior Uveitis ◽  
Age Related Macular Degeneration ◽  
Vascular Endothelial ◽  
Intravitreal Ranibizumab ◽  
Serpiginous Choroiditis ◽  
Age Related ◽  
Stromal Cell Derived Factor ◽  
Endothelial Growth Factor

Inflammatory choroidal neovascularization is a severe but uncommon complication of uveitis, more frequent in posterior uveitis such as punctate inner choroidopathy, multifocal choroiditis, serpiginous choroiditis, and Vogt-Koyanagi-Harada syndrome. Its pathogenesis is supposed to be similar to the wet age related macular degeneration: hypoxia, release of vascular endothelial growth factor, stromal cell derived factor 1-alpha, and other mediators seem to be involved in the uveitis-related choroidal neovascularization. A review on the factors implicated so far in the pathogenesis of inflammatory choroidal neovascularization was performed. Also we reported the success rate of single studies concerning the therapies of choroidal neovascularization secondary to uveitis during the last decade: photodynamic therapy, intravitreal bevacizumab, and intravitreal ranibizumab, besides steroidal and immunosuppressive therapy. Hereby a standardization of the therapeutic approach is proposed.

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