Interaction of gibberellins A3 and A4 in cucumber hypocotyl growth

1963 ◽  
Vol 50 (10) ◽  
pp. 381-382 ◽  
Author(s):  
William K. Purves ◽  
Peter G. Lorber
Keyword(s):  
Hypocotyl Growth ◽  
Cucumber Hypocotyl

The Roles of Gibberellin and Auxin in Cucumber Hypocotyl Growth

1965 ◽  
Vol 18 (2) ◽  
pp. 462-473 ◽  
Author(s):  
Masayuki Katsumi ◽  
Bernard O. Phinney ◽  
William K. Purves
Keyword(s):  
Hypocotyl Growth ◽  
Cucumber Hypocotyl

scholarly journals Promotion of Cucumber Hypocotyl Growth by Two New Gibberellins

Nature ◽  
1961 ◽  
Vol 189 (4758) ◽  
pp. 74-74 ◽  
Author(s):  
P. W. BRIAN ◽  
H. G. HEMMING
Keyword(s):  
Hypocotyl Growth ◽  
Cucumber Hypocotyl

scholarly journals INDUCTION OF CHILLING-TOLERANCE IN CUCUMBER (CUCUMIS SATIVUS L.) SEEDLINGS BY ENDOGENOUS AND APPLIED ETHANOL

HortScience ◽  
1995 ◽  
Vol 30 (2) ◽  
pp. 188f-189
Author(s):  
Chaim Frenkel ◽  
Amnon Erez
Keyword(s):  
Cold Tolerance ◽  
Cucumis Sativus ◽  
Membrane Lipids ◽  
Chilling Injury ◽  
Volatile Anesthetics ◽  
Chilling Tolerance ◽  
Marginal Effect ◽  
Hypocotyl Growth ◽  
Cucumis Sativus L ◽  
Cucumber Hypocotyl

Five-day-old etiolated cucumber (Cucumis sativus L. cv. Marketmore) seedlings held at 2C for 72 hours develop chilling injury resulting in desiccation and collapse of the hypocotyl tissues and eventual plant death. Hypoxia-induced accumulation of ethanol and acetaldehyde led to tolerance to subsequent chilling as evidenced by continued hypocotyl growth and freedom from injury. Arrest of volatile accumulation by applied bisulfite negated the development of hypoxia-induced chilling tolerance in seedlings. In seedlings held in normoxia, cold tolerance was induced by applied ethanol vapors, whereas acetaldehyde had a marginal effect, suggesting that hypoxia-induced cold tolerance may arise from the accumulation and activity of ethanol. Cold tolerance was induced by exposure to gaseous n-propanol and n-butanol vapors and other volatile anesthetics, including chloroform and halothane, indicating that ethanol activity may stem in part from an anesthetic effect that causes disorder of membrane lipids. However, development of cold tolerance in ethanol-enriched tissues was time-dependent, suggesting an association with biosynthetic event(s). Ethanol did not change the fatty acid composition in cucumber hypocotyl membranes.

Wall extensibility during hypocotyl growth: A hypothesis to explain elastic-induced wall loosening

1995 ◽  
Vol 95 (2) ◽  
pp. 296-303
Author(s):  
Hans G. Edelmann
Keyword(s):  
Hypocotyl Growth ◽  
Wall Loosening ◽  
Wall Extensibility

Effects of soil resistance on hypocotyl growth in soybeans

1977 ◽  
Author(s):  
Karl Howard Knittle
Keyword(s):  
Soil Resistance ◽  
Hypocotyl Growth

Effects of soil resistance on hypocotyl growth in soybeans

1977 ◽  
Author(s):  
Karl Howard Knittle
Keyword(s):  
Soil Resistance ◽  
Hypocotyl Growth

scholarly journals A family of pathogen-induced cysteine-rich transmembrane proteins is involved in plant disease resistance

Planta ◽  
2021 ◽  
Vol 253 (5) ◽  
Author(s):  
Marciel Pereira Mendes ◽  
Richard Hickman ◽  
Marcel C. Van Verk ◽  
Nicole M. Nieuwendijk ◽  
Anja Reinstädler ◽  
...  
Keyword(s):  
Arabidopsis Thaliana ◽  
Plasma Membrane ◽  
Disease Resistance ◽  
Plant Disease Resistance ◽  
Transmembrane Proteins ◽  
Series Data ◽  
Immune Gene ◽  
Biological Processes ◽  
Hypocotyl Growth ◽  
Enhanced Resistance

Abstract Main conclusion Overexpression of pathogen-induced cysteine-rich transmembrane proteins (PCMs) in Arabidopsis thaliana enhances resistance against biotrophic pathogens and stimulates hypocotyl growth, suggesting a potential role for PCMs in connecting both biological processes. Abstract Plants possess a sophisticated immune system to protect themselves against pathogen attack. The defense hormone salicylic acid (SA) is an important player in the plant immune gene regulatory network. Using RNA-seq time series data of Arabidopsis thaliana leaves treated with SA, we identified a largely uncharacterized SA-responsive gene family of eight members that are all activated in response to various pathogens or their immune elicitors and encode small proteins with cysteine-rich transmembrane domains. Based on their nucleotide similarity and chromosomal position, the designated Pathogen-induced Cysteine-rich transMembrane protein (PCM) genes were subdivided into three subgroups consisting of PCM1-3 (subgroup I), PCM4-6 (subgroup II), and PCM7-8 (subgroup III). Of the PCM genes, only PCM4 (also known as PCC1) has previously been implicated in plant immunity. Transient expression assays in Nicotiana benthamiana indicated that most PCM proteins localize to the plasma membrane. Ectopic overexpression of the PCMs in Arabidopsis thaliana resulted in all eight cases in enhanced resistance against the biotrophic oomycete pathogen Hyaloperonospora arabidopsidis Noco2. Additionally, overexpression of PCM subgroup I genes conferred enhanced resistance to the hemi-biotrophic bacterial pathogen Pseudomonas syringae pv. tomato DC3000. The PCM-overexpression lines were found to be also affected in the expression of genes related to light signaling and development, and accordingly, PCM-overexpressing seedlings displayed elongated hypocotyl growth. These results point to a function of PCMs in both disease resistance and photomorphogenesis, connecting both biological processes, possibly via effects on membrane structure or activity of interacting proteins at the plasma membrane.

The Role of the Cotyledons in Gibberellin- and Auxin-Induced Elongation of the Cucumber Hypocotyl

1965 ◽  
Vol 18 (2) ◽  
pp. 550-556 ◽  
Author(s):  
Masayuki Katsumi ◽  
William K. Purves ◽  
Bernard O. Phinney ◽  
Jiro Kato
Keyword(s):  
Cucumber Hypocotyl
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