Can sperm whales tell us how to protect the brain against hypoxia and increased intracranial pressure?

1997 ◽  
Vol 20 (4) ◽  
pp. 295-295
Author(s):  
Dag Moskopp
Keyword(s):  
Intracranial Pressure ◽  
Sperm Whales ◽  
Increased Intracranial Pressure ◽  
The Brain

scholarly journals SURVIVAL OF POLIOMYELITIC VIRUS IN THE BRAIN OF THE RABBIT

1918 ◽  
Vol 27 (3) ◽  
pp. 443-447 ◽  
Author(s):  
Harold L. Amoss
Keyword(s):  
Intracranial Pressure ◽  
Pressure Effects ◽  
Rabbit Brain ◽  
High Dilution ◽  
Increased Intracranial Pressure ◽  
Intracerebral Inoculation ◽  
Pathological Lesions ◽  
Active Virus ◽  
The Brain

Suspensions of the central nervous tissues of monkeys, containing the active filterable virus of poliomyelitis, may be injected into the brain of rabbits without setting up symptoms, provided the volume of injection does not cause dangerous increased intracranial pressure. Aside from the pressure effects which develop quickly, no other symptoms or pathological lesions are produced by the suspensions. The active virus of poliomyelitis survives in the brain of rabbits for 4 days, as determined by tests in the monkey, into which the excised site of injection in the rabbit brain is reinoculated. It cannot be detected by this test after the expiration of 7 days. The virus of poliomyelitis is unadapted to the rabbit, and neither induces lesions nor survives long in the central nervous organs of that animal. In this respect it differs from certain streptococci cultivated from poliomyelitic tissues. A monkey immunized to streptococcus cultivated from human poliomyelitic nervous tissues yielded a serum which agglutinated the streptococcus in high dilution, but was without neutralizing action on the filtered virus; and the streptococcus-immune monkey was not protected against the effects of an intracerebral inoculation of the filtered virus. The experiments recorded provide additional reasons for concluding that the streptococcus cultivated from cases of poliomyelitis differs essentially from the filterable virus and is not the microbic cause of epidemic poliomyelitis.

Multiple small cavernous angiomas of the brain with increased intracranial pressure

1978 ◽  
Vol 4 (4) ◽  
pp. 376-378 ◽  
Author(s):  
Richard S. A. Tindall ◽  
Joel B. Kirkpatrick ◽  
Fred Sklar
Keyword(s):  
Intracranial Pressure ◽  
Increased Intracranial Pressure ◽  
Cavernous Angiomas ◽  
The Brain

scholarly journals Central neurocytomas of uncommon locations: report of two cases

2006 ◽  
Vol 64 (4) ◽  
pp. 1015-1018 ◽  
Author(s):  
Francinaldo Lobato Gomes ◽  
Luciano Ricardo França ◽  
Samuel Tau Zymberg ◽  
Sérgio Cavalheiro
Keyword(s):  
Differential Diagnosis ◽  
Intracranial Pressure ◽  
Obstructive Hydrocephalus ◽  
Signs And Symptoms ◽  
Pineal Region ◽  
Additional Treatment ◽  
Third Ventriculostomy ◽  
Increased Intracranial Pressure ◽  
The Brain ◽  
Uncommon Location

We report two patients with central neurocytomas at an uncommon location in the brain. The first, a 58-year-old man presenting with signs and symptoms of increased intracranial pressure, had a tumor located at the pineal region. The second, a 21-year-old woman with tumor in the aqueductal region had worsening migraine-like headaches and diplopia. Both patients had obstructive hydrocephalus treated by neuroendoscopic third ventriculostomy and biopsy of the tumors. No additional treatment was done. We conclude that neurocytomas should be considered in the differential diagnosis of tumors located in the pineal and aqueductal regions.

scholarly journals Pseudotumor Cerebri : A Rare Presentation of Systemic Lupus Erythematosus

2017 ◽  
Vol 34 (2) ◽  
pp. 104-107
Author(s):  
Aparna Das ◽  
Jiban Chandra Das ◽  
Abdullah Al Ahmad ◽  
Md Azizul Kahhar
Keyword(s):  
Intracranial Pressure ◽  
Lupus Erythematosus ◽  
Pseudotumor Cerebri ◽  
Systemic Lupus ◽  
Increased Intracranial Pressure ◽  
Radiological Findings ◽  
Rare Presentation ◽  
Complex Deposition ◽  
The Brain ◽  
Csf Absorption

Raised intracranial pressure in the absence of an intracranial mass or hydrocephalus (BIH or pseudotumor cerebri) has been described in association with many conditions including SLE. Several pathogenic pathways tie BIH with SLE as thrombotic obliteration of cerebral arteriolar and venous systems and immune complex deposition within the arachnoid villi that are responsible for cerebrospinal fluid (CSF) absorption. The diagnosis of BIH was confirmed by increased intracranial pressure in the absence of any abnormal radiological findings of the brain. We report a young woman with SLE complicated by BIH which resolved with corticosteroid therapy and osmotic diuretics.J Bangladesh Coll Phys Surg 2016; 34(2): 104-107

Cerebral radiation necrosis simulating a brain tumor

1974 ◽  
Vol 40 (2) ◽  
pp. 267-271 ◽  
Author(s):  
E. Fletcher Eyster ◽  
Surl L. Nielsen ◽  
Glenn E. Sheline ◽  
Charles B. Wilson
Keyword(s):  
Brain Tumor ◽  
Intracranial Pressure ◽  
Frontal Lobe ◽  
Radiation Necrosis ◽  
Increased Intracranial Pressure ◽  
Content Type ◽  
Cerebral Radiation Necrosis ◽  
Tentorial Herniation ◽  
The Brain ◽  
Fine Print

✓ Two years after undergoing irradiation for a malignant ethmoid tumor, a 50-year-old man developed signs of increased intracranial pressure, an expanding right frontal lobe mass, and tentorial herniation. Operation revealed a mass that grossly appeared to be a glioma, but microscopically proved to be radiation necrosis of the brain.

Osmotic Therapy

2018 ◽  
Author(s):  
Eelco F. M. Wijdicks ◽  
Sarah L. Clark
Keyword(s):  
Intensive Care ◽  
Intracranial Pressure ◽  
Intensive Care Units ◽  
Hypertonic Saline ◽  
Volume Status ◽  
Close Monitoring ◽  
Increased Intracranial Pressure ◽  
Fluid Compartments ◽  
Osmotic Agents ◽  
The Brain

Osmotic drugs are ubiquitously used in neurocritically ill patients. Mannitol and hypertonic saline are readily available in emergency departments and intensive care units to reduce intracranial pressure. Mannitol depletes and hypertonic saline expands the volume status. Hyperosmolar fluids increase the intravascular osmolality, draw water from the brain, reduce or temporize shift, and reduce globally increased intracranial pressure from any cause. These osmotic fluids change fluid compartments and cannot be used indiscriminately. Particularly when they are administered regularly, close monitoring is needed and target goals should be set. This chapter discusses triggers for the use of these osmotic agents, how to judge their effect, and how to dose adequately.

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