Simultaneous arterial and urinary bladder pressure recordings in multiple system atrophy and in spinal disorders with detrusor hyperreflexia

1997 ◽  
Vol 7 (6) ◽  
pp. 299-304 ◽  
Author(s):  
T. Petersen ◽  
C. J. Mathias ◽  
M. Alam ◽  
V. Chandiramani ◽  
C. J. Fowler
Keyword(s):  
Urinary Bladder ◽  
Multiple System Atrophy ◽  
Bladder Pressure ◽  
Spinal Disorders ◽  
Urinary Bladder Pressure ◽  
Detrusor Hyperreflexia

scholarly journals Measurement of Urinary Bladder Pressure: A Comparison of Methods

Sensors ◽  
2018 ◽  
Vol 18 (7) ◽  
pp. 2128 ◽  
Author(s):  
Ingelin Clausen ◽  
Lars W. Tvedt ◽  
Thomas Glott
Keyword(s):  
Urinary Bladder ◽  
Bladder Pressure ◽  
Comparison Of Methods ◽  
Urinary Bladder Pressure

scholarly journals Chiral selective effects of doxazosin enantiomers on blood pressure and urinary bladder pressure in anesthetized rats

2006 ◽  
Vol 27 (11) ◽  
pp. 1423-1430 ◽  
Author(s):  
Shi-ping MA ◽  
Lei-ming REN ◽  
Ding ZHAO ◽  
Zhong-ning ZHU ◽  
Miao WANG ◽  
...  
Keyword(s):  
Blood Pressure ◽  
Urinary Bladder ◽  
Bladder Pressure ◽  
Anesthetized Rats ◽  
Urinary Bladder Pressure ◽  
Selective Effects

Is Urinary Bladder Pressure a Sensitive Indicator of Intra-Abdominal Pressure?

Endoscopy ◽  
1998 ◽  
Vol 30 (09) ◽  
pp. 778-780 ◽  
Author(s):  
S. Yol ◽  
A. Kartal ◽  
Ş. Tavli ◽  
Y. Tatkan
Keyword(s):  
Urinary Bladder ◽  
Sensitive Indicator ◽  
Bladder Pressure ◽  
Abdominal Pressure ◽  
Urinary Bladder Pressure

scholarly journals Gastric and urinary bladder pressures correlate with intra-abdominal pressure in patients with morbid obesity

2021 ◽  
Author(s):  
Shadi Hamoud ◽  
Siham Abdelgani ◽  
Michal Mekel ◽  
Safa Kinaneh ◽  
Ahmad Mahajna
Keyword(s):  
Morbid Obesity ◽  
Urinary Bladder ◽  
Tidal Volume ◽  
Supine Position ◽  
Inspiratory Pressure ◽  
Bladder Pressure ◽  
Abdominal Pressure ◽  
Urinary Bladder Pressure ◽  
Respiratory Parameters ◽  
Gastric Pressure

AbstractIntra-abdominal pressure (IAP) affects cardio-respiratory and hemodynamic parameters and can be measured directly or indirectly by measuring gastric or urinary bladder pressure. The aim of this study was to investigate the correlation between IAP, gastric pressure and urinary bladder pressure in patients with morbid obesity, at normal and elevated levels of IAP in two positions. As well, to examine the effects of increasing IAP and patient's position on hemodynamic and respiratory parameters. Twelve patients undergoing laparoscopic bariatric surgery were included. IAP, gastric pressure, and urinary bladder pressure were measured while patients were in the supine position and after 45° anti-Trendelenburg tilt. Mean inspiratory pressure, peak inspiratory pressure, and tidal volume were recorded and assessed. In supine position; directly measured IAP was 9.1 ± 1.8 mmHg, compared to 10 ± 3.6 and 8.9 ± 2.9 mmHg in the stomach and bladder, respectively. Increasing IAP to 15 mmHg resulted in an increased gastric pressure of 17 ± 3.8 mmHg, and urinary bladder pressure of 14.8 ± 3.9 mmHg. Gastric and urinary bladder pressures strongly correlated with IAP (R = 0.875 and 0.847, respectively). With 45° anti-Trendelenburg tilt; directly measured IAP was 9.4 ± 2.2 mmHg, and pressures of 10.8 ± 3.8 mmHg and 9.2 ± 3.8 mmHg were measured in the stomach and the bladder, respectively. Increasing IAP to 15 mmHg resulted in elevating gastric and bladder pressures to 16.6 ± 5.3 and 13.3 ± 4 mmHg, respectively. Gastric and urinary bladder pressures had good correlation with IAP (R = 0.843 and 0.819, respectively). Changing patient position from supine to 45° anti-Trendelenburg position resulted in decreased mean and peak inspiratory pressures, and increased tidal volume. Basal IAP is high in patients with morbid obesity. IAP shows positive correlation to gastric and urinary bladder pressures at both normal and elevated levels of IAP. Anti-Trendelenburg tilt of mechanically ventilated morbidly obese patients resulted in favorable effects on respiratory parameters.Trial Registration: The study was retrospectively registered in the NIH registry. Registration number is pending.

Changes in discharge rate of epileptogenic foci during active and passive changes in urinary bladder pressure in cats

1977 ◽  
Vol 57 (2) ◽  
pp. 532-548
Author(s):  
R.C. Dill ◽  
J.A. Romiti ◽  
J.A. Kusske ◽  
R.W. Porter
Keyword(s):  
Urinary Bladder ◽  
Discharge Rate ◽  
Bladder Pressure ◽  
Urinary Bladder Pressure ◽  
Epileptogenic Foci

scholarly journals Gastric and Urinary Bladder Pressures Correlate with Intra-abdominal Pressure in Patients with Morbid Obesity

2020 ◽  
Author(s):  
Shadi Hamoud ◽  
Siham Abdelgani ◽  
Michal Mekel ◽  
Safa Kinaneh ◽  
Ahmad Mahajna
Keyword(s):  
Morbid Obesity ◽  
Urinary Bladder ◽  
Tidal Volume ◽  
Supine Position ◽  
Inspiratory Pressure ◽  
Bladder Pressure ◽  
Abdominal Pressure ◽  
Urinary Bladder Pressure ◽  
Respiratory Parameters ◽  
Gastric Pressure

Abstract Background Intra-abdominal pressure (IAP) affects cardio-respiratory and hemodynamic parameters and can be measured directly or indirectly by measuring gastric or urinary bladder pressure. The aim of this study was to investigate the correlation between IAP, gastric pressure and urinary bladder pressure in patients with morbid obesity, at normal and elevated levels of IAP in two positions. As well, to examine the effects of increasing IAP and patient's position on hemodynamic and respiratory parameters. Methods Twelve patients undergoing bariatric surgery were included. IAP, gastric pressure, and urinary bladder pressure were measured while patients were in the supine position and after 45o anti-Trendelenburg tilt. Mean inspiratory pressure, peak inspiratory pressure, and tidal volume were recorded and assessed. Results In supine position; directly measured IAP was 9.1 ± 1.8 mmHg, compared to 10 ± 3.6 and 8.9 ± 2.9 mmHg in the stomach and bladder, respectively. Increasing IAP to 15 mmHg resulted in an increased gastric pressure of 17 ± 3.8 mmHg, and urinary bladder pressure of 14.8 ± 3.9 mmHg. Gastric and urinary bladder pressures strongly correlated with IAP (R = 0.875 and 0.847, respectively). With 45o anti-Trendelenburg tilt; directly measured IAP was 9.4 ± 2.2 mmHg, and pressures of 10.8 ± 3.8 mmHg and 9.2 ± 3.8 mmHg were measured in the stomach and the bladder, respectively. Increasing IAP to 15 mmHg resulted in elevating gastric and bladder pressures to 16.6 ± 5.3 and 13.3 ± 4 mmHg, respectively. Gastric and urinary bladder pressures had good correlation with IAP (R = 0.843 and 0.819, respectively). Changing patient position from supine to 45o anti-Trendelenburg position resulted in decreased mean and peak inspiratory pressures, and increased tidal volume. Conclusions Basal IAP is high in patients with morbid obesity. IAP shows positive correlation to gastric and urinary bladder pressures at both normal and elevated levels of IAP. Anti-Trendelenburg tilt of mechanically ventilated morbidly obese patients resulted in favorable effects on respiratory parameters.

scholarly journals Effects of surgically induced weight loss on urinary bladder pressure, sagittal abdominal diameter and obesity co-morbidity

1998 ◽  
Vol 22 (3) ◽  
pp. 230-235 ◽  
Author(s):  
H Sugerman ◽  
A Windsor ◽  
M Bessos ◽  
J Kellum ◽  
H Reines ◽  
...  
Keyword(s):  
Weight Loss ◽  
Urinary Bladder ◽  
Bladder Pressure ◽  
Sagittal Abdominal Diameter ◽  
Urinary Bladder Pressure ◽  
Co Morbidity ◽  
Surgically Induced

scholarly journals Electroacupuncture decreases the urinary bladder pressure in patients with acute gastrointestinal injury

2015 ◽  
Vol 14 (1) ◽  
pp. 34-39 ◽  
Author(s):  
H.J. Yu ◽  
J.G. Zhu ◽  
P. Shen ◽  
L.H. Shi ◽  
Y.C. Shi ◽  
...  
Keyword(s):  
Urinary Bladder ◽  
Bladder Pressure ◽  
Gastrointestinal Injury ◽  
Urinary Bladder Pressure ◽  
Acute Gastrointestinal Injury

Urodynamic properties and neurotransmitter dependence of urinary bladder contractility in the BK channel deletion model of overactive bladder

2005 ◽  
Vol 289 (3) ◽  
pp. F604-F610 ◽  
Author(s):  
K. S. Thorneloe ◽  
A. L. Meredith ◽  
A. M. Knorn ◽  
R. W. Aldrich ◽  
M. T. Nelson
Keyword(s):  
Urinary Bladder ◽  
Overactive Bladder ◽  
Bk Channels ◽  
Bk Channel ◽  
Bladder Pressure ◽  
Bladder Smooth Muscle ◽  
Medical Issues ◽  
Urinary Bladder Smooth Muscle ◽  
Important Therapeutic Target

Overactive bladder and incontinence are major medical issues, which lack effective therapy. Previously, we showed (Meredith AL, Thornloe KS, Werner ME, Nelson MT, and Aldrich RW. J Biol Chem 279: 36746–36752, 2004) that the gene mSlo1 encodes large-conductance Ca2+-activated K+ (BK) channels of urinary bladder smooth muscle (UBSM) and that ablation of mSlo1 leads to enhanced myogenic and nerve-mediated contractility and increased urination frequency. Here, we examine the in vivo urodynamic consequences and neurotransmitter dependence in the absence of the BK channel. The sensitivity of contractility to nerve stimulation was greatly enhanced in UBSM strips from Slo−/− mice. The stimulation frequency required to obtain a 50% maximal contraction was 8.3 ± 0.9 and 19.1 ± 1.8 Hz in Slo−/− and Slo +/+ mice, respectively. This enhancement is at least partially due to alterations in UBSM excitability, as muscarinic-induced Slo−/− contractility is elevated in the absence of neuronal activity. Muscarinic-induced Slo−/− contractility was mimicked by blocking BK channels with iberiotoxin (IBTX) in Slo +/+ strips, whereas IBTX had no effect on Slo−/− strips. IBTX also enhanced purinergic contractions of Slo +/+ UBSM but was without effect on purinergic contractions of Slo−/− strips. In vivo bladder pressure and urine output measurements (cystometry) were performed on conscious, freely moving mice. Slo−/− mice exhibited increased bladder pressures, pronounced pressure oscillations, and urine dripping. Our results indicate that the BK channel in UBSM has a very significant role in urinary function and dysfunction and as such likely represents an important therapeutic target.

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