The role of free radicals and eicosanoids in the pathogenetic mechanism underlying ischemic brain edema

1989 ◽  
Vol 10 (2) ◽  
pp. 101-133 ◽  
Author(s):  
Takao Asano ◽  
Tohru Koide ◽  
Osamu Gotoh ◽  
Hiroo Joshita ◽  
Tetsu Hanamura ◽  
...  
Keyword(s):  
Free Radicals ◽  
Brain Edema ◽  
Pathogenetic Mechanism ◽  
Ischemic Brain ◽  
Ischemic Brain Edema

The Role of Catecholamine-Induced Lipid Peroxidation and Histamine in Ischemic Brain Edema

1989 ◽  
pp. 818-820 ◽  
Author(s):  
H. Morooka ◽  
H. Sasayama ◽  
K. Sakai ◽  
S. Namba ◽  
A. Nishimoto
Keyword(s):  
Lipid Peroxidation ◽  
Brain Edema ◽  
Ischemic Brain ◽  
Ischemic Brain Edema

Role of Prostaglandins in Experimental Ischemic Brain Edema in Hypertensive Rats: TXA2 Priority in Hypertensive Rats

Brain Edema ◽  
1985 ◽  
pp. 392-395
Author(s):  
S. Sugimoto ◽  
A. Terashi ◽  
Y. Katayama ◽  
M. Mizoguchi ◽  
T. Soeda ◽  
...  
Keyword(s):  
Brain Edema ◽  
Hypertensive Rats ◽  
Ischemic Brain ◽  
Ischemic Brain Edema

The Role of Monoamines and Cyclic AMP in Ischemic Brain Edema

1976 ◽  
pp. 203-214 ◽  
Author(s):  
K. Kogure ◽  
P. Scheinberg ◽  
H. Kishikawa ◽  
R. Busto
Keyword(s):  
Cyclic Amp ◽  
Brain Edema ◽  
Ischemic Brain ◽  
Ischemic Brain Edema

scholarly journals The role of bradykinin in mediating ischemic brain edema in rats.

Stroke ◽  
1993 ◽  
Vol 24 (4) ◽  
pp. 571-575 ◽  
Author(s):  
T Kamiya ◽  
Y Katayama ◽  
F Kashiwagi ◽  
A Terashi
Keyword(s):  
Brain Edema ◽  
Ischemic Brain ◽  
Ischemic Brain Edema

scholarly journals Cortical Tissue Pressure Gradients in Early Ischemic Brain Edema

1986 ◽  
Vol 6 (1) ◽  
pp. 1-7 ◽  
Author(s):  
Shizuo Hatashita ◽  
Julian T. Hoff
Keyword(s):  
Pressure Gradient ◽  
Brain Edema ◽  
Fluid Pressure ◽  
Pressure Gradients ◽  
Tissue Pressure ◽  
Ischemic Brain ◽  
Ischemic Brain Edema ◽  
Ventricular Fluid Pressure ◽  
Severe Degree

We examined the role of ischemic brain edema, tissue pressure gradients, and regional CBF (rCBF) in adjacent regions of cerebral cortex in cats with middle cerebral artery (MCA) occlusion (MCAO). Tissue pressure, rCBF, and water content were measured from gray matter in the central core and the peripheral margin of the MCA territory over 6 h after MCAO. Ventricular fluid pressure and CSF pressure were recorded. Tissue pressure in the ischemic core, with a flow of ∼5 ml/100 g/min, increased more than that in the periphery where flow was ∼19 ml/100 g/min. Tissue pressure rose progressively to 14.8 ± 1.0 mm Hg in the core over 6 h after MCAO, establishing a significant pressure gradient between that tissue and the lateral ventricle nearby or the subarachnoid space in the middle fossa within the first 3 h. The increase in tissue pressure was linearly related to the amount of edema fluid that developed until the edema reached a severe degree. This study shows that a hydrostatic tissue pressure gradient within ischemic cortex is associated with ischemic brain edema. The magnitude of the gradient that develops is related to the severity of ischemic edema in that tissue.

The role of hydrostatic pressure in ischemic brain edema

1981 ◽  
Vol 9 (3) ◽  
pp. 273-282 ◽  
Author(s):  
Kyuya Kogure ◽  
Raul Busto ◽  
Peritz Scheinberg
Keyword(s):  
Hydrostatic Pressure ◽  
Brain Edema ◽  
Ischemic Brain ◽  
Ischemic Brain Edema
Sign in / Sign up

Export Citation Format

Share Document