Exposure and dose to particulate matter inside the subway system of Athens, Greece

2017 ◽  
Vol 10 (8) ◽  
pp. 1015-1028 ◽  
Author(s):  
Eleni Mammi-Galani ◽  
Konstantinos Eleftheriadis ◽  
Luis Mendes ◽  
Mihalis Lazaridis
2015 ◽  
Vol 511 ◽  
pp. 711-722 ◽  
Author(s):  
Vânia Martins ◽  
Teresa Moreno ◽  
María Cruz Minguillón ◽  
Fulvio Amato ◽  
Eladio de Miguel ◽  
...  

2021 ◽  
pp. 111620
Author(s):  
Jianlin Ren ◽  
Junjie He ◽  
Xiangfei Kong ◽  
Wei Xu ◽  
Yiting Kang ◽  
...  

2008 ◽  
Vol 105 (12) ◽  
pp. 586-595
Author(s):  
M. Dormann ◽  
B. Vanderheyden ◽  
D. Steyls

1972 ◽  
Vol 27 (02) ◽  
pp. 263-271 ◽  
Author(s):  
I. A. Cooper ◽  
P Cochrane ◽  
B. G. Firkin ◽  
K. J. Pinkard

SummaryIt has been suggested that human platelets possess the ability to phagocytose particulate matter similar to the polymorphonuclear leukocyte. However some difference of opinion has arisen regarding this contention, particularly as differences have been demonstrated with regard to the observed metabolic changes occurring in platelets related to such a process.The experiments reported in this paper were designed to observe the aerobic and anaerobic metabolism in human platelets during and following interiorization of two different particles, viz. polystyrene latex and thorotrast. The results of these experiments show a marked difference between both types of particles with regard to observable metabolic changes despite the rapid interiorization of both types of material. Some alteration occurs in both aerobic and anaerobic metabolism a considerable time after interiorization of latex, whereas no alteration could be demonstrated after interiorization of thorotrast. It is suggested that the interiorization of particulate matter is by some process other than phagocytosis and that observed metabolic changes related to latex may be due to a release reaction.


1961 ◽  
Vol 06 (01) ◽  
pp. 025-036 ◽  
Author(s):  
James W. Hampton ◽  
William E. Jaques ◽  
Robert M. Bird ◽  
David M. Selby

Summary1. Infusions containing particulate matter, viz. whole amniotic fluid, amniotic fluid sediment, and glass beads, produce in dogs changes in both early and late phases of the clotting reaction. These changes are associated with the development of pulmonary hypertension.2. When dogs were given an active fibrinolysin followed by an infusion of whole amniotic fluid, the alterations in the clotting mechanism were either delayed or did not appear. No pulmonary hypertension developed in these animals.3. We infer that infusions containing particulate matter will produce in dogs both pulmonary hypertension and changes in the clotting mechanism. Although these are independent changes, both are as closely related to the damage to the pulmonary vessels as they are to the biological nature of the infusions.


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