Role of 3D reconstructed images in primary pulmonary vein stenosis

Author(s):  
Bharath V ◽  
Milind Padmakar Hote

2012 ◽  
Vol 48 (8) ◽  
pp. 838-840 ◽  
Author(s):  
A. Walsh ◽  
G. Canny ◽  
C.J. McMahon ◽  
J.M. Redmond ◽  
P. McNally


Heart Rhythm ◽  
2005 ◽  
Vol 2 (5) ◽  
pp. S276
Author(s):  
Hsuan-Ming Tsao ◽  
Mei-Han Wu ◽  
Higa Satoshi ◽  
Kun-Tai Lee ◽  
Ching-Tai Tai ◽  
...  


2002 ◽  
Vol 39 ◽  
pp. 410
Author(s):  
Ahmed N. Alomrani ◽  
Michael R. Nihill ◽  
Ronald G. Grifka ◽  
Colin J. McMahon ◽  
Charles E. Mullins ◽  
...  


Author(s):  
Juan J Portero-Portaz ◽  
Juan G Córdoba-Soriano ◽  
Arsenio Gallardo-López ◽  
Jesús M Jiménez-Mazuecos

Abstract Background pulmonary veins stenosis after atrial fibrillation radiofrequency ablation is an uncommon complication. When it occurs, percutaneous treatment is the preferred approach. There is a lack of standardized procedures, and when stenting restenosis is relatively common. Case summary we present a young patient with recurrence of pulmonary vein stenosis after a first percutaneous treatment. The recurrence of haemoptysis and dyspnoea after treatment in pulmonary vein stenosis allowed the diagnosis of significant stenosis again in our patient. In the Heart Team we opted for new percutaneous treatment, using IVUS to optimize the final result. Discussion nowadays, percutaneous approach is preferred and among percutaneous treatments for pulmonary veins stenosis, and stenting has shown better results than ballon angioplasty. Despite this, experience in instent restenosis is limited, and there is a lack of adequate and specific material for its approach. In this case we present the possible role of the IVUS and the drug-coated ballon angioplasty in this entity.



Children ◽  
2021 ◽  
Vol 8 (2) ◽  
pp. 128
Author(s):  
Abbas H. Zaidi ◽  
Jessica M. Yamada ◽  
David T. Miller ◽  
Kerry McEnaney ◽  
Christina Ireland ◽  
...  

Pulmonary vein stenosis (PVS) is a rare, frequently lethal disease with heterogeneous phenotypes and an unclear etiology. Limited studies have reported associations between PVS and congenital heart disease (CHD), chronic lung disease (CLD), and/or prematurity; however, to date, there have been no studies that report detailed clinical syndromic phenotypes and the potential role of genetics in PVS. An existing registry of multivessel PVS patients seen at Boston Children’s Hospital (BCH) was queried between August 2006 and January 2017 for all existing genetic testing data on these patients. PVS was defined as an intraluminal pulmonary venous obstruction in ≥2 vessels with mean pressure gradients > 4 mmHg. One-hundred-and-fifty-seven patients (46% female, with a median age at PVS diagnosis of 3 months) formed the cohort. Seventy-one (45%) patients had available genetic testing information. Of the 71 patients, a likely genetic diagnosis was found in 23 (32%) patients: 13 (57%) were diagnosed with Trisomy 21 (T21), five (22%) with Smith–Lemli–Opitz Syndrome, five (22%) had other pathologic genetic disease, and 24 (33%) had variants of unknown significance. The majority of 13 patients with T21 and PVS had common atrioventricular canal (CAVC) (10, 77%) and all had severe pulmonary hypertension (PHTN), which led to their PVS diagnosis. In our study, PVS was associated with T21, the majority of whom also had CAVC and PHTN. Therefore, complete assessment of the pulmonary veins should be considered for all T21 patients, especially those with CAVC presenting with PHTN. Furthermore, prospective standardized genetic testing with detailed clinical phenotyping may prove informative about potential genetic etiologies of PVS.



2017 ◽  
Vol 14 (3) ◽  
pp. 178-183
Author(s):  
Т.V. Rogova ◽  
R.М. Кurganov ◽  
G.S. Netalieva ◽  
A.I. Kim


Sign in / Sign up

Export Citation Format

Share Document