Percutaneous balloon valvuloplasty via the right internal jugular vein for valvular pulmonic stenosis with severe right ventricular failure

1989 ◽  
Vol 117 (3) ◽  
pp. 684-685 ◽  
Author(s):  
Assad Chaara ◽  
Layla Zniber ◽  
Naima El Haitem ◽  
Mohamed Benomar
2020 ◽  
Vol 8 (2) ◽  
pp. e001050
Author(s):  
Ilaria Petruccione ◽  
Pamela J Murison ◽  
Hayley Rebecca McDonald ◽  
Patricia Pawson

A 4-year old male miniature schnauzer with severe pulmonic stenosis was presented for percutaneous balloon valvuloplasty. The dog had been managed medically with atenolol for a month prior to the procedure. Clinical examination was unremarkable except for auscultation of a grade V/VI left-sided systolic murmur. The dog was premedicated with pethidine. Anaesthesia was induced using diazepam and etomidate and maintained using sevoflurane in oxygen. Angiography was uneventful, but when the guidewire was threaded through the right side of the heart to allow insertion of the balloon catheter, marked desaturation with visible cyanosis developed. This resolved on withdrawal of the guidewire and catheter but recurred each time the guidewire and catheter were repositioned. Balloon valvuloplasty was eventually successful in reducing the pressure gradient across the stenotic valve from 102 to 52 mmHg. Hypoxaemia did not recur during recovery and the dog was discharged the following day.


2021 ◽  
Vol 21 (1) ◽  
Author(s):  
Jose R. Navas-Blanco ◽  
Justin Miranda ◽  
Victor Gonzalez ◽  
Asif Mohammed ◽  
Oscar D. Aljure

Abstract Background The World Symposium of Pulmonary Hypertension in 2018, updated the definition of pulmonary hypertension (PH) as mean pulmonary artery pressures (PAP) > 20 mmHg. Pulmonary venous hypertension secondary to left-heart disease, constitutes the most common cause of PH, and the determination of a co-existent pre-capillary (primary) PH becomes paramount, particularly at the moment of evaluating and managing patients with heart failure. Pulmonary artery pressures above the systemic pressures define supra-systemic PH and generally leads to frank right ventricular failure and high mortality. Case presentation We present the perioperative management of a patient with rheumatic mitral valve disease, initially found to have severe PH due to pulmonary venous hypertension, who underwent percutaneous mitral balloon valvuloplasty complicated with mitral chordae rupture, severe mitral regurgitation and supra-systemic PH. Multiple medical therapies and an intra-aortic balloon pump were used as means of non-surgical management of this complication. Conclusions This case report illustrates the perioperative implications of combined pre- and post-capillary PH and supra-systemic PH, as this has not been widely discussed in previous literature. A thorough literature review of the clinical characteristics of PH, methods to determine co-existent pre- and post-capillary PH components, as well as concomitant right ventricular failure is presented. Severe PH has known detrimental effects on the hemodynamic status of patients, which can ultimately lead to a decrease in effective cardiac output and poor tissue perfusion.


1991 ◽  
Vol 32 (4) ◽  
pp. 165-169 ◽  
Author(s):  
S. E. Brownlie ◽  
M. A. Cobb ◽  
J. Chambers ◽  
G. Jackson ◽  
S. Thomas

2008 ◽  
Vol 51 (22) ◽  
pp. 2163-2172 ◽  
Author(s):  
Jennifer Cowger Matthews ◽  
Todd M. Koelling ◽  
Francis D. Pagani ◽  
Keith D. Aaronson

ESC CardioMed ◽  
2018 ◽  
pp. 2493-2495
Author(s):  
Joanne A. Groeneveldt ◽  
Anton Vonk Noordegraaf ◽  
Frances S. de Man

In pulmonary arterial hypertension, afterload on the right ventricle is increased. Due to several adaptive mechanisms, the right ventricle is able to cope with a three- to fivefold increase in afterload. However, when adaptive mechanisms are no longer sufficient to compensate for this increase, the patient will develop right ventricular dysfunction and failure. This chapter provides an overview of mechanisms currently considered as having important roles in right ventricular adaptation and right ventricular failure.


2019 ◽  
Vol 14 (3-4) ◽  
pp. 76-77
Author(s):  
Dubravka Šušnjar ◽  
Sandra Jakšić Jurinjak ◽  
Ilko Vuksanović ◽  
Mario Udovičić ◽  
Josip Vincelj ◽  
...  

2012 ◽  
Vol 23 (1) ◽  
pp. 86-90 ◽  
Author(s):  
Mark Puhlman

Left ventricular assist devices (LVADs) have become accepted as treatment for heart failure as a result of improvements in diagnosing and treating left ventricular failure and limited donor availability. In the Pivotal Study of the HeartMate II in the bridge to transplantation population, the incidence of right ventricular failure without the implantation of a right ventricular assist device was 14%, with an additional 6% of the participants ill enough that they required implantation of a right ventricular assist device. This complication increases mortality, cost, and length of stay. This article reviews the screening of LVAD candidates for the probability of right ventricular failure postoperatively, the evaluation of right ventricular function in LVAD candidates, and the optimal management of the right ventricle during the perioperative care of LVAD patients.


2010 ◽  
Vol 13 (1) ◽  
pp. 21
Author(s):  
Mustafa Pac ◽  
Aysenur Pac ◽  
Tugcin Bora Polat ◽  
Sevket Balli ◽  
Nesrin Turhan ◽  
...  

Right ventricular dysplasia is usually discovered by the presence of ventricular arrhythmia. As arrhythmia is an epiphenomenon, the first presentation of some cases can be primarily heart failure. We describe an adolescent girl who presented with progressive right heart failure and whose hallmark was fibrofatty replacement of ventricular muscle, especially of the right side, without ventricular arrhythmia. The patient was successfully treated by orthotopic heart transplantation.


2014 ◽  
Vol 25 (3) ◽  
pp. 580-583 ◽  
Author(s):  
Laurence Vaujois ◽  
Nicolaas van Doesburg ◽  
Marie-Josée Raboisson

AbstractUhl’s anomaly is an evolutive disease leading to terminal right ventricular failure. The most difficult differential diagnosis at presentation is the Ebstein disease. We describe the evolution of a foetus with Uhl’s anomaly from 21 to 30 weeks of gestation, with progressive reduction in the right ventricular anterior myocardium suggestive of apoptosis, leading to foetal demise.


1996 ◽  
Vol 335 (1) ◽  
pp. 21-25 ◽  
Author(s):  
Chuan-Rong Chen ◽  
Tsung O. Cheng ◽  
Tao Huang ◽  
Ying-Ling Zhou ◽  
Ji-Yan Chen ◽  
...  

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