Increased expression of glutamic acid decarboxylase mRNA in rat substantia nigra after an ibotenic acid lesion in the caudate-putamen

Nils Lindefors; Stefan Brené; Håkan Persson

doi:10.1016/0169-328x(90)90029-d

Increased expression of glutamic acid decarboxylase mRNA in rat substantia nigra after an ibotenic acid lesion in the caudate-putamen

Molecular Brain Research ◽

10.1016/0169-328x(90)90029-d ◽

1990 ◽

Vol 7 (3) ◽

pp. 207-212 ◽

Cited By ~ 17

Author(s):

Nils Lindefors ◽

Stefan Brené ◽

Håkan Persson

Keyword(s):

Substantia Nigra ◽

Glutamic Acid ◽

Glutamic Acid Decarboxylase ◽

Ibotenic Acid ◽

Acid Decarboxylase ◽

Caudate Putamen ◽

Rat Substantia Nigra

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Chronic administration of amphetamine increases glutamic acid decarboxylase activity in the rat substantia nigra

Neuroscience Letters ◽

10.1016/0304-3940(90)90014-z ◽

1990 ◽

Vol 109 (3) ◽

pp. 315-320 ◽

Cited By ~ 1

Author(s):

M. Pérez-de la Mora ◽

D. López-Quiroz ◽

J. Méndez-Franco ◽

R. Drucker-Colín

Keyword(s):

Substantia Nigra ◽

Glutamic Acid ◽

Glutamic Acid Decarboxylase ◽

Chronic Administration ◽

Acid Decarboxylase ◽

Decarboxylase Activity ◽

Rat Substantia Nigra

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Short-term dopaminergic regulation of GABA release in dopamine deafferented caudate-putamen is not directly associated with glutamic acid decarboxylase gene expression

Neuroscience Letters ◽

10.1016/0304-3940(91)90761-h ◽

1991 ◽

Vol 128 (1) ◽

pp. 66-70 ◽

Cited By ~ 23

Author(s):

William T. O'Connor ◽

Nils Lindefors ◽

Stefan Brené ◽

Mario Herrera-Marschitz ◽

Håkan Persson ◽

...

Keyword(s):

Gene Expression ◽

Glutamic Acid ◽

Glutamic Acid Decarboxylase ◽

Gaba Release ◽

Acid Decarboxylase ◽

Short Term ◽

Caudate Putamen ◽

Dopaminergic Regulation

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Comparative effects of acute or chronic administration of levodopa to 6-hydroxydopamine-lesioned rats on the expression of glutamic acid decarboxylase in the neostriatum and GABAA receptors subunits in the substantia nigra, pars reticulata

Neuroscience ◽

10.1016/j.neuroscience.2004.12.032 ◽

2005 ◽

Vol 132 (3) ◽

pp. 833-842 ◽

Cited By ~ 34

Author(s):

J. Katz ◽

K.M. Nielsen ◽

J.-J. Soghomonian

Keyword(s):

Substantia Nigra ◽

Glutamic Acid ◽

Glutamic Acid Decarboxylase ◽

Gabaa Receptors ◽

Chronic Administration ◽

Substantia Nigra Pars Reticulata ◽

Acid Decarboxylase ◽

6 Hydroxydopamine

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Micro topography of Tyrosine Hydroxylase, Glutamic Acid Decarboxylase, and Choline Acetyltransferase in the Substantia Nigra and Ventral Tegmental Area of Control and Parkinsonian Brains

Journal of Neurochemistry ◽

10.1111/j.1471-4159.1981.tb04672.x ◽

1981 ◽

Vol 37 (5) ◽

pp. 1218-1227 ◽

Cited By ~ 55

Author(s):

F. Javoy-Agid ◽

A. Ploska ◽

Y. Agid

Keyword(s):

Tyrosine Hydroxylase ◽

Substantia Nigra ◽

Glutamic Acid ◽

Ventral Tegmental Area ◽

Choline Acetyltransferase ◽

Glutamic Acid Decarboxylase ◽

Acid Decarboxylase ◽

Ventral Tegmental ◽

Micro Topography

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EFFECTS OF DEPOLARIZATION ON COFACTOR REGULATION OF GLUTAMIC ACID DECARBOXYLASE IN SUBSTANTIA NIGRA SYNAPTOSOMES

Journal of Neurochemistry ◽

10.1111/j.1471-4159.1979.tb05185.x ◽

1979 ◽

Vol 33 (2) ◽

pp. 533-539 ◽

Cited By ~ 43

Author(s):

Leonard P. Miller ◽

Judith R. Walters

Keyword(s):

Substantia Nigra ◽

Glutamic Acid ◽

Glutamic Acid Decarboxylase ◽

Acid Decarboxylase

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Transplants of fetal substantia nigra regulate glutamic acid decarboxylase gene expression in host striatal neurons

Molecular Brain Research ◽

10.1016/0169-328x(91)90097-h ◽

1991 ◽

Vol 10 (4) ◽

pp. 359-362 ◽

Cited By ~ 19

Author(s):

Jose Segovia ◽

Rafael Castro ◽

Vicente Notario ◽

Karen Gale

Keyword(s):

Gene Expression ◽

Substantia Nigra ◽

Glutamic Acid ◽

Glutamic Acid Decarboxylase ◽

Acid Decarboxylase ◽

Striatal Neurons

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Dopamine receptor blockade increases dopamine D2 receptor and glutamic acid decarboxylase mRNAs in mouse substantia nigra

European Journal of Pharmacology Molecular Pharmacology ◽

10.1016/0922-4106(94)90022-1 ◽

1994 ◽

Vol 269 (1) ◽

pp. 25-33 ◽

Cited By ~ 8

Author(s):

Zheng-Hong Qin ◽

Benjamin Weiss

Keyword(s):

Substantia Nigra ◽

Glutamic Acid ◽

Dopamine Receptor ◽

Glutamic Acid Decarboxylase ◽

Dopamine D2 Receptor ◽

D2 Receptor ◽

Acid Decarboxylase ◽

Receptor Blockade ◽

Dopamine D2

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On the origin of substance P and glutamic acid decarboxylase (GAD) in the substantia nigra

Brain Research ◽

10.1016/0006-8993(77)91034-4 ◽

1977 ◽

Vol 135 (2) ◽

pp. 315-323 ◽

Cited By ~ 275

Author(s):

Michael J. Brownstein ◽

Edmund A. Mroz ◽

Marcel L. Tappaz ◽

Susan E. Leeman

Keyword(s):

Substance P ◽

Substantia Nigra ◽

Glutamic Acid ◽

Glutamic Acid Decarboxylase ◽

Acid Decarboxylase

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Glutamic acid decar☐ylase (GAD) activity in the rat substantia nigra after discrete bilateral kainic acid-inducedlesions of the caudate-putamen and globus pallidus: correlation with locomotor activity

Brain Research ◽

10.1016/0006-8993(81)90254-7 ◽

1981 ◽

Vol 213 (2) ◽

pp. 460-466 ◽

Cited By ~ 4

Author(s):

Usama M.H. Al-Shabibi ◽

John A. Davies

Keyword(s):

Locomotor Activity ◽

Substantia Nigra ◽

Glutamic Acid ◽

Kainic Acid ◽

Globus Pallidus ◽

Caudate Putamen ◽

Gad Activity ◽

Rat Substantia Nigra

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The importance of islet antigen-2 and glutamic acid decarboxylase antibodies in proper diagnosis of diabetes mellitus at the age ≥30 and <70 years

Endocrine Abstracts ◽

10.1530/endoabs.49.ep434 ◽

2017 ◽

Author(s):

Aiste Kondrotiene ◽

Evalda Danyte ◽

Rimantas Zalinkevicius ◽

Rasa Verkauskiene

Keyword(s):

Diabetes Mellitus ◽

Glutamic Acid ◽

Glutamic Acid Decarboxylase ◽

Acid Decarboxylase ◽

Glutamic Acid Decarboxylase Antibodies ◽

Proper Diagnosis ◽

Islet Antigen ◽

Diagnosis Of Diabetes Mellitus

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