Physiological responses of anglo-nubian and baladi goats and their crossbreds to water deprivation under sub-tropical conditions

1989 ◽  
Vol 22 (3-4) ◽  
pp. 295-304 ◽  
Author(s):  
A.Hassan Gamal Eldin
2019 ◽  
pp. 1-11
Author(s):  
Ariana Lopes Correia de Paiva ◽  
Josiel Ferreira ◽  
Talyta Lins Nunes ◽  
Andressa Nunes Mouta ◽  
Naftali Silva Fernandes ◽  
...  

1991 ◽  
Vol 261 (5) ◽  
pp. R1171-R1175 ◽  
Author(s):  
C. C. Barney ◽  
J. S. Williams ◽  
D. H. Kuiper

Dehydration can be brought about by either water deprivation or by heat exposure (thermal dehydration). Angiotensin II has been shown to have a role in water deprivation-induced thirst. The current study was designed to determine whether angiotensin II is involved in thirst caused by thermal dehydration. Male Sprague-Dawley strain rats were dehydrated by exposure to a 40 degree C environment for 2-4 h or by water deprivation for 44 h. Water deprivation but not heat exposure significantly increased plasma renin activity. Neither ureteric ligation nor nephrectomy significantly altered water intake after thermal dehydration. Captopril, an inhibitor of angiotensin converting enzyme, given at a dose of 100 mg/kg ip, significantly decreased water intake in water-deprived rats but not in thermally dehydrated rats. Angiotensin II therefore does not appear to play a role in the control of water intake of thermally dehydrated rats. The physiological responses to dehydration in rats are dependent on the way in which the dehydration is brought about.


2020 ◽  
Vol 52 (6) ◽  
pp. 3575-3582
Author(s):  
Guilherme Resende de Almeida ◽  
Luciano Hauschild ◽  
Alícia Zem Fraga ◽  
Thayssa de Oliveira Littiere ◽  
Vinicius Eduardo Moreira ◽  
...  

2005 ◽  
Vol 288 (6) ◽  
pp. R1791-R1799 ◽  
Author(s):  
Linda Rinaman ◽  
Regis R. Vollmer ◽  
Joseph Karam ◽  
Donnesha Phillips ◽  
Xia Li ◽  
...  

Evidence in rats suggests that central oxytocin (OT) signaling pathways contribute to suppression of food intake during dehydration (i.e., dehydration anorexia). The present study examined water deprivation-induced dehydration anorexia in wild-type and OT −/− mice. Mice were deprived of food alone (fasted, euhydrated) or were deprived of both food and water (fasted, dehydrated) for 18 h overnight. Fasted wild-type mice consumed significantly less chow during a 60-min refeeding period when dehydrated compared with their intake when euhydrated. Conversely, fasting-induced food intake was slightly but not significantly suppressed by dehydration in OT −/− mice, evidence for attenuated dehydration anorexia. In a separate experiment, mice were deprived of water (but not food) overnight for 18 h; then they were anesthetized and perfused with fixative for immunocytochemical analysis of central Fos expression. Fos was elevated similarly in osmo- and volume-sensitive regions of the basal forebrain and hypothalamus in wild-type and OT −/− mice after water deprivation. OT-positive neurons expressed Fos in dehydrated wild-type mice, and vasopressin-positive neurons were activated to a similar extent in wild-type and OT −/− mice. Conversely, significantly fewer neurons within the hindbrain dorsal vagal complex were activated in OT −/− mice after water deprivation compared with activation in wild-type mice. These findings support the view that OT-containing projections from the hypothalamus to the hindbrain are necessary for the full expression of compensatory behavioral and physiological responses to dehydration.


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