P5. 1,25-Dihydroxyvitamin D3 increases bone mineral content in the isolated avian ulna

Bone ◽  
1992 ◽  
Vol 13 (1) ◽  
pp. 108-109
Author(s):  
JS Price ◽  
N Wong ◽  
AEG Goodship
1984 ◽  
Vol 247 (1) ◽  
pp. F1-F13 ◽  
Author(s):  
F. L. Coe ◽  
D. A. Bushinsky

The mechanisms responsible for hypercalciuria may involve intestinal calcium transport, renal tubule calcium reabsorption, and the regulation of bone mineral content. Both parathyroid hormone and 1,25-dihydroxyvitamin D3 (1,25(OH)2D3) may alter urine calcium. For these reasons, understanding the pathogenesis of hypercalciuria in patients has proven to be difficult. We present here an analysis of pathways that regulate systemic calcium homeostasis and of the various mechanisms that have been proposed to explain normocalcemic hypercalciuria in humans. Available evidence seems to implicate disordered regulation of 1,25(OH)2D3 as a basis for at least one common form of hypercalciuria.


2013 ◽  
Author(s):  
N Hangartner Thomas ◽  
F Short David ◽  
Gilsanz Vicente ◽  
J Kalkwarf Heidi ◽  
M Lappe Joan ◽  
...  

2017 ◽  
Author(s):  
Saara Valkama ◽  
Heli Viljakainen ◽  
Elisa Holmlund-Suila ◽  
Jenni Rosendahl ◽  
Helena Hauta-alus ◽  
...  

2014 ◽  
Author(s):  
Julian A Waung ◽  
J H Duncan Bassett ◽  
Graham R Williams

1988 ◽  
Vol 59 (2) ◽  
pp. 173-175 ◽  
Author(s):  
Torbjorn Ahl ◽  
Hans-Erik Sjoberg ◽  
Nils Dalen

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