Chronic Myeloid Leukemia Presenting as Portal Vein Thrombosis with Esophageal Variceal Bleed—A Rare Presentation

2015 ◽  
Vol 5 ◽  
pp. S78
Author(s):  
Rahul Sainani
Keyword(s):  
Chronic Myeloid Leukemia ◽  
Portal Vein ◽  
Portal Vein Thrombosis ◽  
Myeloid Leukemia ◽  
Rare Presentation ◽  
Esophageal Variceal ◽  
Vein Thrombosis ◽  
Variceal Bleed

scholarly journals Splenic and portal vein thrombosis following laparoscopic splenectomy in a pediatric patient with chronic myeloid leukemia

2006 ◽  
Vol 124 (5) ◽  
pp. 275-277 ◽  
Author(s):  
Henrique Manoel Lederman ◽  
Evan Fieldston
Keyword(s):  
Magnetic Resonance ◽  
Chronic Myeloid Leukemia ◽  
Portal Vein ◽  
Portal Vein Thrombosis ◽  
Laparoscopic Splenectomy ◽  
Myeloid Leukemia ◽  
Rare Complication ◽  
Marrow Transplant ◽  
Vein Thrombosis ◽  
Increased Risk

CONTEXT: Splenic or portal vein thrombosis is a rare complication following splenectomy. CASE REPORT: We report a case of splenic and portal venous thrombosis in a 10-year-old girl with chronic myeloid leukemia who underwent laparoscopic splenectomy prior to bone marrow transplant. Clinical suspicion of such thrombosis should be high for patients who have had splenectomy. The diagnosis is confirmed by Doppler ultrasound or contrast-enhanced computed tomography; magnetic resonance imaging magnetic resonance angiography or arteriography can also be used. Proposals for postoperative screening protocols are discussed. Patients with primary myeloproliferative disorders are at increased risk of portal vein thrombosis, independent of surgical intervention, perhaps due to platelet dysfunction resulting from abnormalities of pluripotent stem cells. Marked splenomegaly (with larger draining veins) is thought to increase the risk of thrombosis.

Massive Splenic Infarction and Portal Vein Thrombosis in Children With Chronic Myeloid Leukemia

2014 ◽  
Vol 36 (7) ◽  
pp. e471-e472 ◽  
Author(s):  
Tekin Aksu ◽  
Arzu Y. Erdem ◽  
Ali Fettah ◽  
Dilek Kaçar ◽  
Zekai Avci ◽  
...  
Keyword(s):  
Chronic Myeloid Leukemia ◽  
Portal Vein ◽  
Portal Vein Thrombosis ◽  
Myeloid Leukemia ◽  
Splenic Infarction ◽  
Vein Thrombosis

Relationship between underlying causes of HCC and its complications.

2019 ◽  
Vol 37 (15_suppl) ◽  
pp. e13123-e13123
Author(s):  
Daniel Kyung ◽  
Stuthi Perimbeti ◽  
Bolanle Adepoju ◽  
Shaun Bryan Hanson ◽  
Michael Joseph McCormack ◽  
...  
Keyword(s):  
Hepatic Encephalopathy ◽  
Portal Vein ◽  
Portal Vein Thrombosis ◽  
Bivariate Analysis ◽  
Chi Square ◽  
Esophageal Variceal ◽  
Esophageal Variceal Bleeding ◽  
Vein Thrombosis ◽  
The Us ◽  
Chi Square Test

e13123 Background: Hepatocellular carcinoma (HCC) remains the 4th leading cause of cancer mortality in the US. While mortality varies by stage, the presence of certain HCC-related complications contribute to increased mortality. In this study, we assessed whether the underlying cause for HCC influenced the frequency of common complications. Methods: The National Inpatient Sample between 1999 and 2014 was analyzed using ICD-9 codes. The ICD-9 code for HCC was used to extract all admissions from the years 1999-2014 and weighted to approximate the full inpatient population of the US over the 16 year interval. The prevalence of HCC-related complications were calculated for portal vein thrombosis (PVT), erythrocytosis, peritonitis, esophageal variceal bleeding (EVB), portal hypertension (Portal HTN) and hepatic encephalopathy (HE). Bivariate analysis using Chi square test was performed to compare the percentages of each complication with underlying risk factors for HCC (HBV, HCV, NASH and alcohol). Results: Total of 131,115 admissions (weighted = 648,732) was identified to have HCC. Conclusions: Portal HTN was the most common complication associated with HCV and NASH, whereas hepatic encephalopathy was most frequently seen with alcohol and PVT with HBV, respectively. Alcohol was associated with the highest rate of HCC-complications with the exception of portal vein thrombosis. HBV was associated with the lowest frequencies of complications except for PVT, for which it was the highest. Future studies might look at whether disease modifying measures such as cessation of alcohol and eradication of active HBV, HCV would impact the natural history of HCC.[Table: see text][Table: see text]

3201 Acute Portal Vein Thrombosis Presenting With Atypical Abdominal Pain: A Rare Presentation

2019 ◽  
Vol 114 (1) ◽  
pp. S1710-S1710
Author(s):  
Tejinder Randhawa ◽  
Ishaan Vohra ◽  
Yazan Abu Omar ◽  
Ricky Patel ◽  
Estefania Flores
Keyword(s):  
Abdominal Pain ◽  
Portal Vein ◽  
Portal Vein Thrombosis ◽  
Rare Presentation ◽  
Vein Thrombosis

scholarly journals Recurrent esophagogastric variceal bleeding due to portal vein thrombosis caused by protein S deficiency

2018 ◽  
Vol 06 (11) ◽  
pp. E1283-E1288 ◽  
Author(s):  
Haoxiong Zhou ◽  
Jieying Xuan ◽  
Xianyi Lin ◽  
Yunwei Guo
Keyword(s):  
Portal Vein ◽  
Portal Vein Thrombosis ◽  
Variceal Bleeding ◽  
Endoscopic Therapy ◽  
Protein S ◽  
Protein S Deficiency ◽  
Esophageal Variceal ◽  
Vein Thrombosis ◽  
S Deficiency

Abstract Background and study aims Esophagogastric variceal bleeding (EGVB) is common in patients with portal vein thrombosis (PVT). Hereditary deficiencies in natural anticoagulant proteins, such as protein S, might contribute to PVT. However, recurrent EGVB caused by PVT in patients with protein S deficiency is seldom reported. Herein, we present the case of a 38-year-old man with protein S deficiency complicated with PVT. The patient suffered recurrent EGVB for 7 years. He underwent splenectomy plus pericardial revascularization and sequential endoscopic therapy, including one gastric variceal obturation (GVO) procedure and two esophageal variceal ligations (EVL) to eradicate the varices. Rivaroxaban was administrated to reduce risk of thrombotic events. The patient is currently well without rebleeding after 1 year of follow-up. To our knowledge there is no consensus on management of recurrent EGVB on the basis of thrombophilia complicated with PVT. According to our practice, sequential endoscopic therapy combined with anticoagulant appears to be effective and safe.

scholarly journals S2907 Portal Vein Thrombosis in the COVID-19 Era: A Rare Presentation

2021 ◽  
Vol 116 (1) ◽  
pp. S1204-S1204
Author(s):  
Arnold Forlemu ◽  
Shehroz Aslam ◽  
Zaid Ansari ◽  
Brett Hughes ◽  
Keng-Yu Chuang
Keyword(s):  
Portal Vein ◽  
Portal Vein Thrombosis ◽  
Rare Presentation ◽  
Vein Thrombosis

scholarly journals Successful management of portal vein thrombosis in a Yorkshire Terrier with protein-losing enteropathy

2020 ◽  
Vol 16 (1) ◽  
Author(s):  
Yumi Sakamoto ◽  
Kumiko Ishigaki ◽  
Chieko Ishikawa ◽  
Tomohiro Nakayama ◽  
Kazushi Asano ◽  
...  
Keyword(s):  
Portal Vein ◽  
Portal Vein Thrombosis ◽  
Low Dose ◽  
Factor Xa ◽  
Albumin Concentration ◽  
Rare Presentation ◽  
Protein Losing Enteropathy ◽  
Vein Thrombosis ◽  
Portal Vein Thrombus ◽  
Yorkshire Terrier

Abstract Background Portal vein thrombosis (PVT) is a rare presentation in dogs with protein-losing enteropathy (PLE). Rivaroxaban, an oral, selective, direct factor Xa inhibitor, has not been reported to be administrated for canine PVT and the effect is unclear in dogs with PLE. Case presentation An 11-year-old Yorkshire Terrier presented with moderate ascites. The dog had severe hypoalbuminemia (1.2 g/dL), and a portal vein thrombus was confirmed on computed tomographic angiography (CTA). On endoscopic examination, it became apparent that the hypoalbuminemia was caused by PLE, which was consequent of lymphatic dilation and lymphoplasmacytic enteritis. Therefore, the dog was initially treated with oral administrations of spironolactone and clopidogrel, with dietary fat restriction. However, a follow-up CTA showed no changes in the ascites, thrombus, and portal vein to aorta (PV/Ao) ratio. Therefore, the dog was additionally prescribed rivaroxaban and low-dose prednisolone for the portal vein thrombus and hypoalbuminemia due to lymphoplasmacytic enteritis, respectively. Following the treatment, the PV/Ao ratio decreased because of a decrease in the thrombus and the ascites disappeared completely with an elevation of albumin concentration (1.9 g/dL). Conclusions This case report demonstrated that oral administration of rivaroxaban combined with low-dose glucocorticoid was effective management for PVT in a dog with PLE.

The Seemingly Benign Abdomen with an Underlying Insidious Pathology: A Rare Presentation of Portal Vein Thrombosis

2016 ◽  
Vol 111 ◽  
pp. S1349-S1350
Author(s):  
Joseph Dulaney ◽  
Leena Chacko. Saline ◽  
David Wes Powers ◽  
Javier Sobrado
Keyword(s):  
Portal Vein ◽  
Portal Vein Thrombosis ◽  
Rare Presentation ◽  
Vein Thrombosis
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