Secondary in-hospital epidemiological investigation after an outbreak of Pseudomonas aeruginosa ST357

2020 ◽  
Vol 26 (3) ◽  
pp. 257-265 ◽  
Author(s):  
Toshihito Mihara ◽  
Takeshi Kimura ◽  
Kyoko Momiyama ◽  
Atsushi Kainuma ◽  
Koichi Akiyama ◽  
...  
2006 ◽  
Vol 27 (7) ◽  
pp. 767-770 ◽  
Author(s):  
Rosa Fanci ◽  
Patrizia Pecile ◽  
Enrico Casalone ◽  
Alessio Mengoni ◽  
Elena Tamburini ◽  
...  

We report the epidemiological investigation of an outbreak ofPseudomonas aeruginosainfection in 6 patients who shared, during different periods, the same 2 rooms of a bone marrow transplantation unit. Phenotypic and molecular analysis of isolates from patients and from the environment strongly suggested a single, environmental source of infection.


2012 ◽  
Vol 12 (1) ◽  
pp. 152 ◽  
Author(s):  
Annalisa Ballarini ◽  
Giovanna Scalet ◽  
Malgorzata Kos ◽  
Nina Cramer ◽  
Lutz Wiehlmann ◽  
...  

1981 ◽  
Vol 9 (3) ◽  
pp. 260-265 ◽  
Author(s):  
D. V. Seal ◽  
Janet M. Strangeways

An epidemiological investigation of Pseudomonas aeruginosa in an Intensive Care Neurosurgical Unit has shown that there were epidemic, endemic and endogenous types present simultaneously. These pseudomonads were cultured from purulent sputa postoperatively and sometimes caused systemic disease. The epidemic type was traced to a ventilator and a nebulizer whilst the endemic and endogenous types were not found in environmental sites. Effective decontamination of equipment and the use of bacterial filters where possible are essential in preventing the spread of infection. Staff hygiene remains important, particularly hand washing with antiseptic soap preparations.


2008 ◽  
Vol 43 (4) ◽  
pp. 335-338 ◽  
Author(s):  
R Fanci ◽  
B Bartolozzi ◽  
S Sergi ◽  
E Casalone ◽  
P Pecile ◽  
...  

1999 ◽  
Vol 37 (6) ◽  
pp. 2071-2073 ◽  
Author(s):  
I. Pujana ◽  
L. Gallego ◽  
G. Martín ◽  
F. López ◽  
J. Canduela ◽  
...  

PCR fingerprinting was used for the epidemiological investigation of 64 Pseudomonas aeruginosa isolates collected from 16 chronic bronchiectasis patients without cystic fibrosis: 56% of the patients harbored one clone, 12.5% carried a single major type with minor variants, and 31.5% carried two clones. Only a minority of the acquisitions of antibiotic resistance was related to the acquisition of exogenous strains. Mucoid and nonmucoid sets of isolates did not display any consistent differences in their patterns. The genetic similarity among the clones ranged from 10 to 69%. Cross-infection or common-source exposure did not appear to have occurred.


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