scholarly journals Vein wall remodeling after deep vein thrombosis involves matrix metalloproteinases and late fibrosis in a mouse model

2005 ◽  
Vol 42 (1) ◽  
pp. 140-148 ◽  
Author(s):  
Kristopher B. Deatrick ◽  
Jonathan L. Eliason ◽  
Erin M. Lynch ◽  
Andrea J. Moore ◽  
Nicholas A. Dewyer ◽  
...  
Keyword(s):  
Deep Vein Thrombosis ◽  
Matrix Metalloproteinases ◽  
Mouse Model ◽  
Vein Wall ◽  
Vein Thrombosis ◽  
Deep Vein

scholarly journals Deep Vein Thrombosis Exhibits Characteristic Serum and Vein Wall Metabolic Phenotypes in the Inferior Vena Cava Ligation Mouse Model

2018 ◽  
Vol 55 (5) ◽  
pp. 703-713 ◽  
Author(s):  
Yeji Sung ◽  
Konstantina Spagou ◽  
Marina Kafeza ◽  
Michael Kyriakides ◽  
Brahman Dharmarajah ◽  
...  
Keyword(s):  
Deep Vein Thrombosis ◽  
Mouse Model ◽  
Inferior Vena Cava ◽  
Inferior Vena ◽  
Vena Cava ◽  
Vein Wall ◽  
Metabolic Phenotypes ◽  
Vein Thrombosis ◽  
Deep Vein

Vein wall remodeling after deep vein thrombosis involves matrix metalloproteinases

2004 ◽  
Vol 121 (2) ◽  
pp. 290-291
Author(s):  
K.B. Deatrick ◽  
J.L. Eliason ◽  
E.M. Lynch ◽  
A.J. Moore ◽  
N.A. Dewyer ◽  
...  
Keyword(s):  
Deep Vein Thrombosis ◽  
Matrix Metalloproteinases ◽  
Vein Wall ◽  
Vein Thrombosis ◽  
Deep Vein

Vein wall remodeling in patients with acute deep vein thrombosis and chronic postthrombotic changes

2017 ◽  
Vol 15 (10) ◽  
pp. 1989-1993 ◽  
Author(s):  
A. Chandrashekar ◽  
J. Garry ◽  
A. Gasparis ◽  
N. Labropoulos
Keyword(s):  
Deep Vein Thrombosis ◽  
Vein Wall ◽  
Vein Thrombosis ◽  
Acute Deep Vein Thrombosis ◽  
Deep Vein

scholarly journals Deep Vein Thrombosis Resolution Is Modulated by Monocyte CXCR2-Mediated Activity in a Mouse Model

2004 ◽  
Vol 24 (6) ◽  
pp. 1130-1137 ◽  
Author(s):  
Peter K. Henke ◽  
Andrea Varga ◽  
Sumit De ◽  
C. Barry Deatrick ◽  
Jonathon Eliason ◽  
...  
Keyword(s):  
Deep Vein Thrombosis ◽  
Mouse Model ◽  
Vein Thrombosis ◽  
Deep Vein

scholarly journals Vein wall re-endothelialization after deep vein thrombosis is improved with low-molecular-weight heparin

2008 ◽  
Vol 47 (3) ◽  
pp. 616-624 ◽  
Author(s):  
Daria K. Moaveni ◽  
Erin M. Lynch ◽  
Cathy Luke ◽  
Vikram Sood ◽  
Gilbert R. Upchurch ◽  
...  
Keyword(s):  
Molecular Weight ◽  
Deep Vein Thrombosis ◽  
Low Molecular Weight Heparin ◽  
Low Molecular Weight ◽  
Vein Wall ◽  
Vein Thrombosis ◽  
Deep Vein

scholarly journals The role of urokinase plasminogen activator and plasmin activator inhibitor-1 on vein wall remodeling in experimental deep vein thrombosis

2012 ◽  
Vol 56 (4) ◽  
pp. 1089-1097 ◽  
Author(s):  
Joe F. Baldwin ◽  
Vikram Sood ◽  
Megan A. Elfline ◽  
Cathy E. Luke ◽  
Nicholas A. Dewyer ◽  
...  
Keyword(s):  
Deep Vein Thrombosis ◽  
Plasminogen Activator ◽  
Urokinase Plasminogen Activator ◽  
Vein Wall ◽  
Vein Thrombosis ◽  
Deep Vein ◽  
Activator Inhibitor

IL-17A promotes the formation of deep vein thrombosis in a mouse model

2018 ◽  
Vol 57 ◽  
pp. 132-138 ◽  
Author(s):  
Peiwu Ding ◽  
Shaoshao Zhang ◽  
Miao Yu ◽  
Yuqian Feng ◽  
Qi Long ◽  
...  
Keyword(s):  
Deep Vein Thrombosis ◽  
Mouse Model ◽  
Vein Thrombosis ◽  
Deep Vein

The Role of Leg Compression in the Treatment of Deep Vein Thrombosis

2014 ◽  
Vol 29 (1_suppl) ◽  
pp. 66-70 ◽  
Author(s):  
Hugo Partsch
Keyword(s):  
Deep Vein Thrombosis ◽  
Bed Rest ◽  
Postthrombotic Syndrome ◽  
Vein Wall ◽  
Adjunct Treatment ◽  
Vein Thrombosis ◽  
Reduction Of Pain ◽  
Deep Vein ◽  
Good Compression

Up until today mobile outpatients are put into bed as soon as the diagnosis of deep vein thrombosis (DVT) is made. Fear of pulmonary embolism (PE) is the main rationale for bed-rest; additional arguments are pain and swelling which are expected to resolve faster during leg elevation. Based on previous work it is demonstrated that keeping mobile patients with acute DVT walking under exact anticoagulation does not increase the risk for symptomatic PE in comparison with bed-rest and that there is an immediate reduction of pain and swelling when good compression is applied and the patient is encouraged to walk. Immediate compression and walking seems also to reduce the incidence of a postthrombotic syndrome. One presumed mechanism of action of this adjunct treatment modality is the increase of shear stress in the microcirculation of the vein wall releasing anti-inflammatory and anti-coagulatory mediators.

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