scholarly journals Fibrodysplastic implications for transvenous embolization of a high-flow pelvic arteriovenous malformation in Osler-Weber-Rendu syndrome

2015 ◽  
Vol 1 (1) ◽  
pp. 16-19 ◽  
Author(s):  
Naiem Nassiri ◽  
Jones Thomas ◽  
Saum Rahimi
Keyword(s):  
Arteriovenous Malformation ◽  
High Flow ◽  
Transvenous Embolization

Adenosine-Induced Cardiac Arrest for Transvenous Embolization of Midbrain Arteriovenous Malformation

2019 ◽  
Vol 18 (6) ◽  
pp. E184-E190 ◽  
Author(s):  
Jaims Lim ◽  
Hussain Shallwani ◽  
Kunal Vakharia ◽  
Adnan H Siddiqui
Keyword(s):  
Arteriovenous Malformation ◽  
Transarterial Embolization ◽  
Posterior Wall ◽  
Sudden Onset ◽  
High Flow ◽  
Neurological Deficits ◽  
Transvenous Embolization ◽  
Blurred Vision ◽  
Systemic Hypotension ◽  
The Right

Abstract BACKGROUND AND IMPORTANCE Few studies describe the use of adenosine-induced cardiac systole for treatment of cerebrovascular pathologies. We describe a midbrain arteriovenous malformation (AVM) treated with transvenous embolization using adenosine-induced asystole to achieve transient systemic hypotension with the purpose of furthering discussion on the technique and operative considerations for adenosine use in endovascular AVM treatments. CLINICAL PRESENTATION A 29-yr-old man presented with sudden onset of severe bilateral headache, blurred vision, and numbness on the right side of his face and tongue. Noncontrast head computed tomography revealed fourth ventricle hemorrhage. Diagnostic cerebral angiography revealed a high-flow midbrain AVM with a posterior wall perforator from the basilar artery terminus and a draining vein into the straight sinus. Transarterial AVM embolization was successful. The patient was discharged with no residual neurological deficits but returned 1 wk later with slurred speech and left-sided dysmetria. Repeat angiography revealed partial AVM filling. Attempts at transarterial embolization were unsuccessful. Thus, transvenous AVM embolization with adenosine-induced cardiac asystole and systemic hypotension was performed. A total of 60 mg of adenosine was administered, followed by 2 additional doses of 60 and 40 mg; and complete cardiac asystole with a mean arterial pressure of 40 mmHg was maintained, resulting in successful embolization of the AVM. No residual filling was visualized on postembolization arterial angiography runs. The patient was neurologically stable and discharged on postoperative day 2. CONCLUSION With appropriate and safe dosing, adenosine-induced asystole and systemic hypotension may be a feasible, safe option to reduce flow and assist endovascular transvenous embolization of high-flow AVMs.

An intrathoracic arteriovenous malformation discovered as an extremely uncommon reason of neonatal congestive cardiac failure

2009 ◽  
Vol 19 (5) ◽  
pp. 530-533 ◽  
Author(s):  
Sigrun R. Hofmann ◽  
Matthias Weise ◽  
Katharina I. Nitzsche
Keyword(s):  
Arteriovenous Malformation ◽  
Cardiac Failure ◽  
Arteriovenous Malformations ◽  
Neonatal Period ◽  
High Flow ◽  
Congestive Cardiac Failure ◽  
Vein Of Galen ◽  
Prenatal Detection ◽  
First Case

AbstractCongenital arteriovenous malformations are rare causes of congestive cardiac failure in neonates. The most common sites are in the head and liver, but other sites include the thorax, the abdomen and the limbs. The onset of failure is usually not in the immediate neonatal period, but later on in life, albeit that lesions such as the arteriovenous malformation of the vein of Galen, and other arteriovenous malformations in different locations which produce high flow can present early. We describe here the first case, to the best of our knowledge, of prenatal detection of an intrathoracic arteriovenous malformation producing neonatal cardiac failure, which was successfully treated by surgery postnatally.

High flow arteriovenous malformation with transverse-sigmoid sinus stenosis and congestive venopathy mimicking DAVF

2022 ◽  
pp. 1-4
Author(s):  
Dinesh D. Menon ◽  
Dnyandeep Patil ◽  
Bopanna K. M. ◽  
Ullas V. Acharya ◽  
Paritosh Pandey
Keyword(s):  
Arteriovenous Malformation ◽  
High Flow ◽  
Sigmoid Sinus ◽  
Sinus Stenosis

scholarly journals Primary free fibula reconstruction in life-threatening haemorrhage from high flow arteriovenous malformation of mandible

2018 ◽  
Vol 51 (02) ◽  
pp. 222-230
Author(s):  
Parvathi Ravula ◽  
Srikanth Rangachari ◽  
Rammurti Susarla ◽  
Laxman Sambari ◽  
Srinivas Saraswathi Jammula
Keyword(s):  
Arteriovenous Malformation ◽  
Curative Resection ◽  
High Flow ◽  
External Carotid ◽  
Primary Reconstruction ◽  
Free Fibula Flap ◽  
Definitive Therapy ◽  
Free Fibula ◽  
Life Threatening ◽  
Selective Embolisation

ABSTRACT Background: High flow arteriovenous malformation (AVM) of the mandible is rare, but it can present as a life-threatening emergency with severe intraoral bleeding for the first time. The gold standard of treatment for an AVM of the mandible is selective embolisation combined with resection and subsequent reconstructions. With the advent of advanced multidisciplinary techniques aimed at definitive therapy, surgical resection and primary reconstruction can provide an ideal anatomical and functional cure. There are no previous reports on primary resection and reconstruction for life-threatening haemorrhage from high flow AVM of the mandible. Aim: We discuss our approach aimed at definitive therapy in life-threatening intraoral bleeding from large high flow AVM of the mandible. Subjects and Methods: Four patients were managed for life-threatening intraoral bleeding during 2015–2017. Compression was applied over the bleeding point before the airway could be secured by endotracheal tube. Under general anaesthesia, the external carotid artery (ECA) was temporarily occluded using an umbilical tape loop ligature to control the bleeding. Emergency selective embolisation was done, followed by curative resection and primary mandible reconstruction using free fibula flap. Outcome assessed. Results: Temporary occlusion of the ECA successfully controlled the bleeding immediately and facilitated selective embolisation and definitive therapy. All the four cases were successfully reconstructed with a good outcome. There was no recurrence during the follow-up period. Conclusion: In life-threatening intraoral bleeding from large high flow AVM of the mandible, emergency selective embolisation followed by curative resection and primary reconstruction is safe in achieving an ideal cure.

scholarly journals Transvenous embolization for a huge pelvic arteriovenous malformation associated with prominent outflow veins

2020 ◽  
Vol 15 (3) ◽  
pp. 285-291 ◽  
Author(s):  
Atsushi Ugajin ◽  
Hiroyuki Fujii ◽  
Akifumi Fujita ◽  
Hiroyasu Nakamura ◽  
Akira Fujisaki ◽  
...  
Keyword(s):  
Arteriovenous Malformation ◽  
Transvenous Embolization

Transvenous embolization for a purely intraorbital arteriovenous fistula

2005 ◽  
Vol 103 (4) ◽  
pp. 756-759 ◽  
Author(s):  
Jun Deguchi ◽  
Makoto Yamada ◽  
Ryusuke Ogawa ◽  
Toshihiko Kuroiwa
Keyword(s):  
Arteriovenous Malformation ◽  
Arteriovenous Fistula ◽  
Visual Function ◽  
Clinical Management ◽  
Ophthalmic Artery ◽  
Transvenous Embolization ◽  
Content Type ◽  
Arteriovenous Fistulas ◽  
Fine Print

✓ Purely intraorbital arteriovenous fistulas (AVFs) are rare, and their clinical management is controversial. The authors successfully treated a patient with an intraorbital AVF by transvenous embolization alone. An accurate distinction between an arteriovenous malformation (AVM), which is characterized by the existence of a nidus, and an AVF, which has no nidus, is important and requires superselective ophthalmic artery angiography. Treatment of an intraorbital AVF by transvenous embolization can improve visual function.

scholarly journals Embolization of High-Flow Arteriovenous Malformation

2016 ◽  
Vol 33 (02) ◽  
pp. 157-160 ◽  
Author(s):  
Christopher Funaki ◽  
Brian Funaki
Keyword(s):  
Arteriovenous Malformation ◽  
High Flow
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