Arbitrary visuomotor mapping in the grip-lift task: dissociation of performance deficits in right and left middle cerebral artery stroke

Neuroscience ◽  
2012 ◽  
Vol 210 ◽  
pp. 128-136 ◽  
Author(s):  
D. Bensmail ◽  
A.-S. Sarfeld ◽  
M. Ameli ◽  
G.R. Fink ◽  
D.A. Nowak
2020 ◽  
Vol 43 (3) ◽  
pp. 86-89
Author(s):  
Andrew T. Connor ◽  
Alex Crawford ◽  
Rebecca J. Levy ◽  
Lauren M. Schneider ◽  
Seth A. Hollander ◽  
...  

2017 ◽  
Vol 11 (4) ◽  
pp. 459-461 ◽  
Author(s):  
Breno José Alencar Pires Barbosa ◽  
Marcelo Houat de Brito ◽  
Júlia Chartouni Rodrigues ◽  
Gabriel Taricani Kubota ◽  
Jacy Bezerra Parmera

ABSTRACT. A 75-year-old right-handed woman presented to the emergency department with simultanagnosia and right unilateral optic ataxia. Moreover, the patient had agraphia, acalculia, digital agnosia and right-left disorientation, consistent with complete Gerstmann's syndrome. This case highlights the concurrence of Gerstmann's syndrome and unilateral optic ataxia in the acute phase of a left middle cerebral artery stroke.


2016 ◽  
Vol 48 (1) ◽  
pp. 72-74 ◽  
Author(s):  
Zeke M. Campbell ◽  
Peter W. Kaplan ◽  
Jonathan C. Edwards

Subclinical rhythmic electrographic discharges in adults (SREDA) is a well-known benign EEG phenomenon. However, the occurrence of SREDA is rare, and atypical forms are even more elusive, with only few cases reported in the literature. Herein, we describe a case of a 77-year-old woman with a left middle cerebral artery stroke and paroxysms of rhythmic, sharply contoured activity over the right central head region, mimicking focal seizures on EEG, that were determined to represent atypical SREDA. To our knowledge, no case of SREDA with a contralateral structural cerebral abnormality has been described, and its occurrence offers some limited insight as to the mechanisms underlying this mysterious entity.


Neurocase ◽  
2005 ◽  
Vol 11 (6) ◽  
pp. 416-426 ◽  
Author(s):  
Helmut Hildebrandt ◽  
Cathleen Schütze ◽  
Markus Ebke ◽  
Freimuth Brunner-Beeg ◽  
Paul Eling

2021 ◽  
pp. 174749302098455
Author(s):  
Nick A Weaver ◽  
Angelina K Kancheva ◽  
Jae-Sung Lim ◽  
J Matthijs Biesbroek ◽  
Irene MC Huenges Wajer ◽  
...  

Background Post-stroke cognitive impairment can occur after damage to various brain regions, and cognitive deficits depend on infarct location. The Mini-Mental State Examination (MMSE) is still widely used to assess post-stroke cognition, but it has been criticized for capturing only certain cognitive deficits. Along these lines, it might be hypothesized that cognitive deficits as measured with the MMSE primarily involve certain infarct locations. Aims This comprehensive lesion-symptom mapping study aimed to determine which acute infarct locations are associated with post-stroke cognitive impairment on the MMSE. Methods We examined associations between impairment on the MMSE (<5th percentile; normative data) and infarct location in 1198 patients (age 67 ± 12 years, 43% female) with acute ischemic stroke using voxel-based lesion-symptom mapping. As a frame of reference, infarct patterns associated with impairments in individual cognitive domains were determined, based on a more detailed neuropsychological assessment. Results Impairment on the MMSE was present in 420 patients (35%). Large voxel clusters in the left middle cerebral artery territory and thalamus were significantly (p < 0.01) associated with cognitive impairment on the MMSE, with highest odds ratios (>15) in the thalamus and superior temporal gyrus. In comparison, domain-specific impairments were related to various infarct patterns across both hemispheres including the left medial temporal lobe (verbal memory) and right parietal lobe (visuospatial functioning). Conclusions Our findings indicate that post-stroke cognitive impairment on the MMSE primarily relates to infarct locations in the left middle cerebral artery territory. The MMSE is apparently less sensitive to cognitive deficits that specifically relate to other locations.


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