Case report: In utero diagnosis of a vein of Galen aneurysm using colour flow Doppler

Vein of Galen aneurysm is a rare pathology, representing less than 1% of intracranial vascular malformations. We report on a 65 years-old man who experienced a generalized tonic-clonic seizure. Brain imaging showed a large calcified expanding mass in the pineal region, confirming the diagnosis of a vein of Galen aneurysm. Because of the spontaneous thrombosis of the malformation, there was no need for microsurgical or endovascular treatment and he is been regularly followed since that.

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Confirmation of t1-bright vein of galen aneurysm spontaneous thrombosis by subtraction magnetic resonance venography: a case report

Acta Radiologica ◽

10.1080/02841850903055611 ◽

2009 ◽

Vol 50 (7) ◽

pp. 812-815

Author(s):

M. Irfan ◽

B. Lohman ◽

A. M. McKinney

Keyword(s):

Case Report ◽

Magnetic Resonance ◽

Contrast Medium ◽

Digital Subtraction Angiography ◽

Magnetic Resonance Venography ◽

Two Dimensional ◽

Digital Subtraction ◽

Vein Of Galen ◽

Spontaneous Thrombosis ◽

Vein Of Galen Aneurysm

Spontaneous thrombosis of a vein of Galen aneurysmal malformation (VOGM) is rare. We describe a 2-month-old patient with a patent VOGM and hydrocephalus, also confirmed patent at 6 months, but with subsequent lack of filling on preembolization catheter digital subtraction angiography (DSA) at 9 months’ age. Due to the presence of T1- and T2-bright signal, noncontrast T1-weighted images (T1WI), T2-weighted images (T2WI), two-dimensional (2D) time-of-flight (TOF) magnetic resonance venography (MRV), and postcontrast T1WI were ambiguous for patency. However, subtracting the pre- from the postcontrast MRV images confirmed closure compared to subtracted images at 6 months’ age. The factors contributing to thrombosis were likely a combination of a disproportionately small straight sinus, ventriculostomy, and contrast medium from DSA.

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In utero diagnosis of a vein of Galen aneurysm by ultrasound

Neuroradiology ◽

10.1007/bf00327500 ◽

1984 ◽

Vol 26 (5) ◽

pp. 417-418 ◽

Cited By ~ 15

Author(s):

Dianne Bernice Mendelsohn ◽

Y. Hertzanu ◽

A. Butterworth

Keyword(s):

In Utero ◽

Vein Of Galen ◽

Vein Of Galen Aneurysm

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EP03.09: Prenatal 3D ultrasound diagnosis of vein of Galen aneurysm (VGAM): case report

Ultrasound in Obstetrics and Gynecology ◽

10.1002/uog.15536 ◽

2015 ◽

Vol 46 ◽

pp. 193-193

Author(s):

M.J. Kudla ◽

A. Beczkowska-Kielek ◽

K. Kutta

Keyword(s):

Case Report ◽

3D Ultrasound ◽

Ultrasound Diagnosis ◽

Vein Of Galen ◽

Vein Of Galen Aneurysm

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Intracranial Pial Arteriovenous Fistula Mimicking a Vein of Galen Aneurysm with Hydrocephalus Managed with Endovascular Method: Case Report

Iranian Journal of Neurosurgery ◽

10.29252/irjns.3.2.67 ◽

2017 ◽

Vol 3 (2) ◽

pp. 67-63

Author(s):

Jorge Valderrama ◽

Ricardo Garcia ◽

Guru Satyarthee ◽

Willem Calderon ◽

Samer Hoz ◽

...

Keyword(s):

Case Report ◽

Arteriovenous Fistula ◽

Vein Of Galen ◽

Vein Of Galen Aneurysm ◽

Pial Arteriovenous Fistula

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Fetal supraventricular tachycardia: detection by routine auscultation and successful in-utero management. Case report

BJOG An International Journal of Obstetrics & Gynaecology ◽

10.1111/j.1471-0528.1988.tb06517.x ◽

1988 ◽

Vol 95 (10) ◽

pp. 1073-1076 ◽

Cited By ~ 9

Author(s):

SHEILA MACPHAIL ◽

S. A. WALKINSHAW

Keyword(s):

Case Report ◽

Supraventricular Tachycardia ◽

In Utero

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Successful Surgical and Anesthetic Management of Vein of Galen Aneurysm in a Neonate in Congestive Heart Failure

Neurosurgery ◽

10.1227/00006123-198805000-00018 ◽

1988 ◽

Vol 22 (5) ◽

pp. 908-910 ◽

Cited By ~ 5

Author(s):

Jane Matjasko ◽

Walker Robinson ◽

Daniel Eudaily

Keyword(s):

Heart Failure ◽

Congestive Heart Failure ◽

Cardiac Failure ◽

Growth And Development ◽

Anesthetic Management ◽

Vascular Supply ◽

Vein Of Galen ◽

Vein Of Galen Malformation ◽

Vein Of Galen Aneurysm

Abstract A 12-day-old infant in intractable cardiac failure due to a vein of Galen malformation was treated successfully with serial ligation of the majority of the vessels feeding the malformation. Despite some residual vascular supply to the malformation, the congestive heart failure has disappeared and growth and development have been normal over a 3-year follow-up period.

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