scholarly journals THE OCCURRENCE OF PITUITRIN AND EPINEPHRIN IN FETAL PITUITARY AND SUPRARENAL GLANDS

1915 ◽  
Vol 23 (2) ◽  
pp. 435-438
Author(s):  
Carey Pratt McCord
Keyword(s):  
Suprarenal Glands

scholarly journals Origin and destiny of cholesterol in the animal organism. Part XIII―On the autolysis of liver and spleen

1922 ◽  
Vol 93 (655) ◽  
pp. 486-492
Keyword(s):  
The Body ◽  
Cholesterol Esters ◽  
Animal Organism ◽  
Suprarenal Glands ◽  
Aseptic Conditions ◽  
Normal Adults

In Part XII of this series (1921) the comparison was made of the intake and output of cholesterol in normal adults on a known diet and over periods of six days, and an average daily loss of 0·3 grm. of cholesterol was noted. The conclusion drawn was that there must be some organ in the body capable of synthesising cholesterol. A similar view has been put forward by Grigaut (1913), who expressed the opinion that this synthesis is the function of the suprarenal glands. It seemed likely that the study of the autolysis of various tissues under aseptic conditions might throw some light on this question. A number of observations bearing on this subject are described in the literature, some undertaken with the object of finding evidence of the presence of enzymes capable of hydrolysing cholesterol esters, others with the object of ascertaining whether destruction or synthesis of cholesterol took place on autolysis. The results are very conflicting.

scholarly journals CALCIFICATION OF THE SUPRARENAL GLANDS OF CATS

1926 ◽  
Vol 43 (4) ◽  
pp. 495-499 ◽  
Author(s):  
David Marine
Keyword(s):  
Cell Injury ◽  
Sequence Of Events ◽  
Suprarenal Glands ◽  
Experimental Injury ◽  
Degenerative Processes

Calcification of the fascicular zone of the cortex has been observer in 64 of 257 cats. It is always calcification and never ossification It is more common in young animals and in our experience is associated with distemper. In its severe forms it may be recognized clinically. The symptoms resemble those seen in cats surviving double suprarenalectomy for 2 to 3 weeks. The toxin producing the focal degeneration is dearly a very specific one since attempts to produce such lesions by several types of experimental injury have failed. The sequence of events appears to be similar to that present in other degenerative processes associated with calcification, namely cell injury and necrosis, deposition of calcium at first as fatty compounds which later change to carbonate and phosphate. It is suggested that this lesion should be considered in interpreting experiments in which cats are used.

scholarly journals Tumor de células esteroideas de ovario: Reporte de un caso.

2012 ◽  
Vol 16 (1) ◽  
pp. 80 ◽  
Author(s):  
Edith Jacqueline LUQUE CUBA ◽  
Freddy GARCIA RAMOS ◽  
Adolfo RECHKEMMER PRIETO ◽  
José SOLIS VILLANUEVA ◽  
Luz ROSAS VARGAS ◽  
...  
Keyword(s):  
Clinical Characteristics ◽  
Polycystic Ovary ◽  
Dexamethasone Suppression Test ◽  
Androgen Excess ◽  
Ovary Syndrome ◽  
Suprarenal Glands ◽  
Suppression Test ◽  
Common Problems ◽  
Dexamethasone Suppression ◽  
Leydig Cell Tumors

The suggestive clinical characteristics of hyperandrogenism are very common problems in women and have been related with excessive androgen production from ovaries, suprarenal glands or both. The most common identifiable cause of androgen excess is the polycystic ovary syndrome. The virilizing tumors are rare. We report the case of a postmenopausal women with virilizing signs and a left anexial mass. Testosterone 4.3ng/mL (0.2-0.95); DHEAS 56ug/dL (35-430); androstenedione: 10ng/ml (0.4-2.7); Cortisol 16ug/dL. Testosterone post dexamethasone suppression test 3.5ng/mL. Ovarian steroid cell tumors secrete great quantities of testosterone or androstenedione and differ from Leydig cell tumors in that they lack crystals of Reinke. Usually, they are benign, but 20% of malignancy has been reported. They can produce different substances. The election treatment is oophorectomy. As in our patient, the androgens levels are normalized after surgery.

scholarly journals The Control of the Blood-Pressure

1934 ◽  
Vol 28 (1) ◽  
pp. 15-28 ◽  
Author(s):  
J. H. Burn
Keyword(s):  
Blood Pressure ◽  
High Pressure ◽  
Essential Hypertension ◽  
Skeletal Muscles ◽  
Sympathetic Nerves ◽  
Nerve Supply ◽  
Normal Variation ◽  
Single Observation ◽  
Paroxysmal Hypertension ◽  
Suprarenal Glands

Present conceptions of the control of the general blood-pressure are based on the view that the sympathetic nerves are only constrictor in action and the circulating hormones, adrenaline and vasopressin, only pressor in effect. Surgical treatment of high blood-pressure and allied conditions has been directed accordingly to the removal of portions of the sympathetic system and to the removal of the suprarenal glands or tumours connected with them. Removal of portions of the sympathetic has been successful in relieving attacks of angina pectoris and removal of suprarenal tumours has relieved paroxysmal hypertension. Neither removal of portions of the sympathetic systems nor removal of the suprarenal glands has been successful in reducing continuous hypertension. It is suggested that our conceptions of the control of the blood-pressure need revision.The normal variation in blood-pressure is not sufficiently realized. It may be as low as 85 mm. or as high as 190 mm. About one in every 40 men has a blood-pressure higher than 160 mm. It is suggested that the diagnosis of essential hypertension should never be made unless it is known that the blood-pressure has been rising. A single observation of a high pressure is not enough for the diagnosis.Evidence is described that adrenaline and vasopressin may lower the blood-pressure as well as cause it to rise; similarly there is evidence that the sympathetic nerve supply to the skeletal muscles is dilator rather than constrictor in effect. Hence, all three mechanisms commonly thought of as pressor may also be depressor, and it seems more accurate to think of them as controlling the blood-pressure by raising or lowering it than simply as factors which raise it.

TUBERCULOSIS OF SUPRARENAL GLANDS (ADDISON'S DISEASE)

Endocrinology ◽  
1937 ◽  
Vol 21 (3) ◽  
pp. 401-403 ◽  
Author(s):  
HENRY M. FEINBLATT ◽  
BARNETT ALPERT
Keyword(s):  
Addison’S Disease ◽  
Addison's Disease ◽  
Suprarenal Glands
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