scholarly journals PULMONARY ARTERIAL BLOOD FLOW THROUGH AN ACUTELY ATELECTATIC LUNG

1961 ◽  
Vol 42 (5) ◽  
pp. 599-606 ◽  
Author(s):  
Rudolph C. Camishion ◽  
Yoshinori Ota ◽  
Vincent D. Cuddy ◽  
John H. Gibbon
Keyword(s):  
Blood Flow ◽  
Arterial Blood Flow ◽  
Arterial Blood ◽  
Flow Through ◽  
Pulmonary Arterial

scholarly journals Changes in bronchial and pulmonary arterial blood flow with progressive tension pneumothorax

1996 ◽  
Vol 81 (4) ◽  
pp. 1664-1669 ◽  
Author(s):  
Paula Carvalho ◽  
Jacob Hildebrandt ◽  
Nirmal B. Charan
Keyword(s):  
Blood Flow ◽  
Gas Exchange ◽  
Tension Pneumothorax ◽  
Blood Gases ◽  
Left Pulmonary Artery ◽  
Arterial Blood Flow ◽  
Mechanically Ventilated ◽  
Arterial Blood ◽  
Flow Through ◽  
Pulmonary Arterial

Carvalho, Paula, Jacob Hildebrandt, and Nirmal B. Charan.Changes in bronchial and pulmonary arterial blood flow with progressive tension pneumothorax. J. Appl. Physiol. 81(4): 1664–1669, 1996.—We studied the effects of unilateral tension pneumothorax and its release on bronchial and pulmonary arterial blood flow and gas exchange in 10 adult anesthetized and mechanically ventilated sheep with chronically implanted ultrasonic flow probes. Right pleural pressure (Ppl) was increased in two steps from −5 to 10 and 25 cmH2O and then decreased to 10 and −5 cmH2O. Each level of Ppl was maintained for 5 min. Bronchial blood flow, right and left pulmonary arterial flows, cardiac output (Q˙t), hemodynamic measurements, and arterial blood gases were obtained at the end of each period. Pneumothorax resulted in a 66% decrease inQ˙t, bronchial blood flow decreased by 84%, and right pulmonary arterial flow decreased by 80% at Ppl of 25 cmH2O ( P < 0.001). At peak Ppl, the majority ofQ˙t was due to blood flow through the left pulmonary artery. With resolution of pneumothorax, hemodynamic parameters normalized, although abnormalities in gas exchange persisted for 60–90 min after recovery and were associated with a decrease in total respiratory compliance.

The selective closure of feline carotid arteriovenous anastomoses (AVAs) by GR43175

Cephalalgia ◽  
1989 ◽  
Vol 9 (9_suppl) ◽  
pp. 41-46
Author(s):  
Marion J Perren ◽  
Wasyl Feniuk ◽  
Patrick Pa Humphrey
Keyword(s):  
Blood Pressure ◽  
Blood Flow ◽  
Peripheral Resistance ◽  
Haemodynamic Effects ◽  
Arterial Blood Flow ◽  
Arteriovenous Anastomoses ◽  
Arterial Blood ◽  
Flow Through ◽  
Carotid Arterial ◽  
Dose Dependent

The haemodynamic effects of the selective 5-HT1-like agonist GR43175 have been compared with that of ergotamine in anaesthetized cats. Both GR43175 (30–1000 μg/kg intravenously) and ergotamine (0.3–30 μg/kg intravenously) caused a dose-dependent reduction in the proportion of cardiac output passing through arteriovenous anastomoses (AVAs). However, unlike GR43175, the effect of ergotamine (30 μg/kg intravenously) was associated with marked increases in diastolic blood pressure and total peripheral resistance. In further studies, the effect of GR43175 on the distribution of blood flow within the carotid bed has been examined. GR43175 caused a reduction in total carotid arterial blood flow which was entirely due to a reduction in flow through carotid AVAs. These results demonstrate that GR43175, unlike ergotamine, has a highly selective vasoconstrictor action on AVAs within the cranial circulation of anaesthetized cats. Such a mechanism may be important in its antimigraine activity.

Pulmonary blood flow, pressure and resistance following tetraethylammonium and aminophylline

1961 ◽  
Vol 200 (2) ◽  
pp. 287-291 ◽  
Author(s):  
M. Harasawa ◽  
S. Rodbard
Keyword(s):  
Blood Flow ◽  
Pulmonary Vascular Resistance ◽  
Vascular Resistance ◽  
Systemic Blood Pressure ◽  
Arterial Blood Flow ◽  
Tetraethylammonium Chloride ◽  
Blood Flows ◽  
Arterial Blood ◽  
Pulmonary Arterial ◽  
Flow Pressure

The effects of tetraethylammonium chloride (TEAC) and aminophylline on the pulmonary vascular resistance were studied in thoracotomized dogs. Pulmonary arterial blood flow and pressure, and systemic blood pressure were measured simultaneously. Both drugs showed marked hypotensive effects on the systemic vessels. In every instance pulmonary arterial pressures and blood flows were reduced by TEAC given via the pulmonary artery and increased by aminophylline. However, the calculated pulmonary vascular resistance remained essentially unchanged in all experiments. These data challenge the concept that the pulmonary vessels respond to these drugs by active vasodilatation

Regional Abnormalities in Pulmonary Arterial Blood Flow During Acute Asthmatic Attacks

Radiology ◽  
1967 ◽  
Vol 88 (1) ◽  
pp. 142-144 ◽  
Author(s):  
Fred Mishkin ◽  
Henry N. Wagner
Keyword(s):  
Blood Flow ◽  
Arterial Blood Flow ◽  
Arterial Blood ◽  
Pulmonary Arterial
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