Acquired zinc deficiency in a premature breast-fed infant

1993 ◽  
Vol 28 (3) ◽  
pp. 499-501 ◽  
Author(s):  
Laura J. Buehning ◽  
Robert W. Goltz
Keyword(s):  
2021 ◽  
Vol 13 (3) ◽  
pp. 444-449
Author(s):  
Giovanna D’Amico ◽  
Corinne De Laet ◽  
Guillaume Smits ◽  
Deborah Salik ◽  
Guillaume Deprez ◽  
...  

We present a case of a transient acquired zinc deficiency in a breast-fed, 4-month-old-male prematurely born infant, with acrodermatitis enteropathica-like symptoms such as crusted, eroded, erythemato-squamous eruption in periorificial and acral patterns. The laboratory investigations showed low zinc levels in the infant’s and the mother’s serum and in the mother’s milk; genetic analysis did not show any mutation in the SLC39A4 gene, involved in acrodermatitis enteropathica. Acquired zinc deficiency is often found in premature infants because of their increased requirement, the low serum and milk zinc levels in breastfeeding women being also an important risk factor, as in this case. A prompt zinc supplementation is essential for the good prognosis of the disease.


2006 ◽  
Vol 166 (3) ◽  
pp. 189-194 ◽  
Author(s):  
Antonia Kienast ◽  
Bernhard Roth ◽  
Christiane Bossier ◽  
Christina Hojabri ◽  
Peter H. Hoeger
Keyword(s):  

1995 ◽  
Vol 36 (3) ◽  
pp. 157-159 ◽  
Author(s):  
Karen M Stapleton ◽  
Edward O'Loughlin ◽  
John P Relic

2010 ◽  
Vol 16 (6) ◽  
Author(s):  
Sambasiviah C Murthy ◽  
Malleshappa M Udagani ◽  
Ashok V Badakali ◽  
Bhuvaneshwari C Yelameli
Keyword(s):  

Nutrition ◽  
2011 ◽  
Vol 27 (10) ◽  
pp. 1087-1089 ◽  
Author(s):  
Nadia El Fékih ◽  
Kharfi Monia ◽  
Sebastien Schmitt ◽  
Iméne Dorbani ◽  
Sébastien Küry ◽  
...  

1995 ◽  
Vol 154 (1) ◽  
pp. 71-75 ◽  
Author(s):  
F. Heinen ◽  
D. Matern ◽  
W. Pringsheim ◽  
J. U. Leititis ◽  
M. Brandis

PEDIATRICS ◽  
1992 ◽  
Vol 89 (6) ◽  
pp. 1094-1095
Author(s):  
VIKRAM KHOSHOO ◽  
JOHN KJARSGAARD ◽  
BERNICE KRAFCHICK ◽  
STANLEY H. ZLOTKIN

Zinc is an essential trace mineral for all mammals. The activities of many enzymes, including those needed for protein synthesis, are zinc dependent. It is therefore not surprising that zinc deficiency is cinically expressed in rapidly growing tissues. Zinc deficiency commonly presents with specific skin lesions, diarrhea, growth failure, alopecia, irritability, and anorexia. Acquired zinc deficiency is an uncommon entity. It has most often been described in patients receiving total parenteral nutrition with inadequate or no zinc in the nutrient mixture.1,2 It has also been described in breast-fed premature babies3,4 in whom symptoms become apparent by 4 to 5 months of age.


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