Differential transcriptional profile of the Wnt pathway in sporadic colorectal cancers with and without microsatellite instability

2008 ◽  
Vol 6 (9) ◽  
pp. 13-14
Author(s):  
P. Ortega ◽  
A. Morán ◽  
C. De Juan ◽  
C. Frías ◽  
T. Fernández-Marcelo ◽  
...  
2008 ◽  
Vol 14 (4) ◽  
pp. 995-1001 ◽  
Author(s):  
Paloma Ortega ◽  
Alberto Morán ◽  
Carmen de Juan ◽  
Cristina Frías ◽  
Susana Hernández ◽  
...  

2001 ◽  
Vol 120 (5) ◽  
pp. A295-A295
Author(s):  
D CHANG ◽  
A GOEL ◽  
L RICCIARDIELLO ◽  
C ARNOLD ◽  
C BOLAND

2014 ◽  
Vol 146 (5) ◽  
pp. S-628
Author(s):  
Young Baek Kim ◽  
Sun-Young Lee ◽  
Jeong Hwan Kim ◽  
In Kyung Sung ◽  
Hyung Seok Park ◽  
...  

2011 ◽  
Vol 459 (1) ◽  
pp. 55-63 ◽  
Author(s):  
Jeong Mo Bae ◽  
Mi Jung Kim ◽  
Jung Ho Kim ◽  
Jae Moon Koh ◽  
Nam-Yun Cho ◽  
...  

2013 ◽  
Vol 145 (3) ◽  
pp. 540-543.e22 ◽  
Author(s):  
Alexandra E. Gylfe ◽  
Johanna Kondelin ◽  
Mikko Turunen ◽  
Heikki Ristolainen ◽  
Riku Katainen ◽  
...  

2016 ◽  
Vol 29 (04) ◽  
pp. 336-344 ◽  
Author(s):  
Jean Ashburn ◽  
Thomas Plesec ◽  
Matthew Kalady

AbstractColorectal serrated polyps are intermediate lesions in the serrated neoplastic pathway, which account for up to 30% of colorectal cancers. This pathway is biologically distinct from the adenoma-to-carcinoma sequence, with associated cancers exhibiting mutations in the BRAF oncogene, DNA promoter hypermethylation, and microsatellite instability. An evolving understanding of these unique lesions has led to the development of a more accurate classification, improved endoscopic identification, and tailored clinical management guidelines. This article reviews serrated polyps and serrated polyposis syndrome.


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