Catalytic Conversion of Hydrocarbons - Catalytic Promoting Effect of Antimony Tetroxide in the Aromatization of Hydrocarbons with a Chromia-Alumina Catalyst

1946 ◽  
Vol 38 (1) ◽  
pp. 61-64 ◽  
Author(s):  
F. E. Fisher ◽  
H. C. Watts ◽  
G. E. Harris ◽  
C. M. Hollenbeck

1983 ◽  
Vol 14 (10) ◽  
Author(s):  
H. FUJIE ◽  
K. MORI ◽  
K. TAMURA ◽  
S. OKADA ◽  
S. MATSUOKA ◽  
...  


1990 ◽  
Vol 80 (1) ◽  
pp. 109-113 ◽  
Author(s):  
Ester P. Lorences ◽  
Gordon J. McDougall ◽  
Stephen C. Fry


1966 ◽  
Vol 16 (8) ◽  
pp. 239-244
Author(s):  
S. K. Bhattacharyya ◽  
B. N. Avasthi




1954 ◽  
Vol 16 (4) ◽  
pp. 365-368 ◽  
Author(s):  
Antti Telkkä ◽  
A. N. Kuusisto


2010 ◽  
Vol 31 (9) ◽  
pp. 1185-1188
Author(s):  
Changshui TONG ◽  
Xiaoxia TONG ◽  
Menggui JIN ◽  
Nianjun YE


2014 ◽  
Vol 18 (9) ◽  
pp. 1149-1158 ◽  
Author(s):  
Young-Byung Yi ◽  
Jin-Woo Lee ◽  
Chung-Han Chung


2020 ◽  
Vol 20 ◽  
Author(s):  
Wenbin Wu ◽  
Yangmei Zhang ◽  
Xiaowu Li ◽  
Xiang Wang ◽  
Yuan Yuan

Objective: The purpose of this study was to explore the mechanism of the miR-375/XPR1 axis in esophageal squamous cell carcinoma (ESCC) and provide a new idea for targeted therapy of ESCC. Methods: Differentially expressed genes in GEO and TCGA databases were analyzed by bioinformatics. The expression levels of miR-375 and XPR1 mRNA were detected by qRT-PCR. Protein expression of XPR1 was detected by western blot. Bioinformatics analysis and dual luciferase assay were conducted to confirm the targeting relationship between miR-375 and XPR1. The viability, proliferation, migration and invasion of cells in each treatment group were detected by CCK-8, colony formation, wound healing and Transwell assays. Results: Significantly down-regulated miR-375 and remarkably up-regulated XPR1 were observed in ESCC tissue and cells. Overexpression of miR-375 inhibited proliferation, invasion and migration of ESCC cells, and greatly reduced the promoting effect of XPR1 overexpression on cell proliferation, migration and invasion. Dual luciferase assay confirmed that miR-375 targeted and inhibited XPR1 expression in ESCC. Conclusion: These results demonstrate the regulatory role of the miR-375/XPR1 axis in ESCC cells and provide a new potential target for the precise treatment of patients with ESCC.



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