scholarly journals cis-Hydroxyplatination of Diethyl Maleate:  Modeling the Intermediates in a Catalytic Alkene-Hydration Cycle with Organoplatinum(II)−Hydroxo ComplexesJ.Am.Chem. Soc.1995,117, 8335−8340

1996 ◽  
Vol 118 (27) ◽  
pp. 6528-6528 ◽  
Author(s):  
Martin A. Bennett ◽  
Hong Jin ◽  
Sihai Li ◽  
Louis M. Rendina ◽  
Anthony C. Willis
Keyword(s):  
Diethyl Maleate ◽  
Alkene Hydration

Phase equilibria of (water–carboxylic acid–diethyl maleate) ternary liquid systems at 298.15K

2011 ◽  
Vol 303 (2) ◽  
pp. 168-173 ◽  
Author(s):  
Süheyla Çehreli ◽  
Tugba Gündogdu
Keyword(s):  
Carboxylic Acid ◽  
Phase Equilibria ◽  
Diethyl Maleate ◽  
Ternary Liquid Systems ◽  
Liquid Systems

Hetero-Diels-Alder Reactions of Enaminothione with Electrophilic Olefins. Synthesis of 2-Furyl Substituted 2H-Thiopyrans

2002 ◽  
Vol 57 (6) ◽  
pp. 637-644 ◽  
Author(s):  
Krystyna Bogdanowicz-Szwed ◽  
Artur Budzowski
Keyword(s):  
Acetic Acid ◽  
Maleic Anhydride ◽  
Acetic Anhydride ◽  
Alder Reaction ◽  
Diels Alder ◽  
Diethyl Maleate ◽  
Diels Alder Reaction

AbstractThe hetero-Diels-Alder reaction of 1-(2-furyl)-3-(dimethylamino)-2-propene-1-thione (diene) with maleic and fumaric acids, and β-nitrostyrenes yielded 3,4-dihydro-2H-thiopyran derivatives. Treatment of some of those cycloadducts with acetic acid caused elimination of dimethylamine yielding stable 2H-thiopyrans. Reaction of the diene with maleic anhydride furnished a cycloadduct which underwent spontaneous rearrangement to form an N,N-dimethylamide derivative. Cycloadditions of the diene to maleimide, N-phenylmaleimide, diethyl maleate, fumarate and butenolide, carried out in the presence of acetic anhydride,were followed by elimination of dimethylamine, afforded stable 2H-thiopyran derivatives.

Metabolism of aflatoxin B1 in cultured mouse hepatocytes: Comparison with rat and effects of cyclohexene oxide and diethyl maleate

1979 ◽  
Vol 50 (3) ◽  
pp. 429-436 ◽  
Author(s):  
Gary M. Decad ◽  
Kathleen K. Dougherty ◽  
Dennis P.H. Hsieh ◽  
James L. Byard
Keyword(s):  
Aflatoxin B1 ◽  
Diethyl Maleate ◽  
Cyclohexene Oxide ◽  
Mouse Hepatocytes

Free radical initiation mechanisms in the polymerisation of diethyl fumarate and diethyl maleate studied by the nitroxide trapping technique

1994 ◽  
Vol 30 (11) ◽  
pp. 1259-1267 ◽  
Author(s):  
W. Ken Busfield ◽  
Ian D. Jenkins ◽  
Kirstin Heiland
Keyword(s):  
Free Radical ◽  
Diethyl Maleate ◽  
Radical Initiation ◽  
Free Radical Initiation

Diethyl Maleate

2007 ◽  
pp. 332-332
Keyword(s):  
Diethyl Maleate

Chloroform‐induced multiple forms of ornithine decarboxylase: Differential sensitivity of forms to enhancement by diethyl maleate and inhibition by ODC‐antizyme

1989 ◽  
Vol 27 (1) ◽  
pp. 57-64 ◽  
Author(s):  
Russell E. Savage ◽  
Kirk Nofzinger ◽  
Cynthia Bedell ◽  
Anthony B. DeAngelo ◽  
Michael A. Pereira
Keyword(s):  
Ornithine Decarboxylase ◽  
Differential Sensitivity ◽  
Diethyl Maleate ◽  
Multiple Forms

Role of glutathione in gastric mucosal cytoprotection

1984 ◽  
Vol 247 (3) ◽  
pp. G296-G304 ◽  
Author(s):  
A. Robert ◽  
D. Eberle ◽  
N. Kaplowitz
Keyword(s):  
Gastric Injury ◽  
Minor Role ◽  
Mucosal Protection ◽  
Diethyl Maleate ◽  
Thiol Compounds ◽  
Acute Ethanol ◽  
Endogenous Release ◽  
A Minor ◽  
Mucosal Cytoprotection

Exogenous thiol compounds have been reported to protect the stomach from ethanol-induced necrotic lesions. The gastric mucosa contains high levels of an endogenous thiol, glutathion (GSH). Because of the known role of glutathione in protecting against hepatic injury, its role in gastric mucosal cytoprotection was of interest. By use of an animal model for acute gastric injury from ethanol, a close parallel relation between depletion of endogenous mucosal GSH and induction of mucosal protection was demonstrated. Surprisingly, mucosal protection varied inversely with the level of mucosal GSH obtained after treatment with specific GSH-depleting agents (diethyl maleate and cyclohexene-1-one). Depletion of gastric mucosal GSH was associated with an increase in the mucosal content of prostaglandins 6-keto F1 alpha and F2 alpha but not E2. The protective effect induced by GSH-depleting agents was partially reversed by indomethacin in some but not all studies. Although GSH depletors increased gastric juice volume, protection with these agents persisted after the volume and mucosal GSH had returned to control levels and also was not reversed by increasing the dose of ethanol threefold to overcome a possible dilutional effect. We conclude that, contrary to apparent predictions, depletion of endogenous gastric GSH protects the stomach from acute ethanol-induced injury. Although the mechanism of this protection is unknown, a mediation by endogenous release of prostaglandins seems to play a minor role since diethyl maleate was protective even in indomethacin-treated animals.

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