scholarly journals Symptom Dimensions of Attention Deficit Hyperactivity Disorder and Nicotine Withdrawal Symptoms

2012 ◽  
Vol 31 (4) ◽  
pp. 363-375 ◽  
Author(s):  
Katherine J. Ameringer ◽  
Adam M. Leventhal
2016 ◽  
Vol 16 (1) ◽  
Author(s):  
Michael Liebrenz ◽  
Carl Erik Fisher ◽  
Romilda Nellen ◽  
Anja Frei ◽  
Anne-Catherine Biechl ◽  
...  

2007 ◽  
Vol 38 (7) ◽  
pp. 1057-1066 ◽  
Author(s):  
B. C. Haberstick ◽  
D. Timberlake ◽  
C. J. Hopfer ◽  
J. M. Lessem ◽  
M. A. Ehringer ◽  
...  

BackgroundA variety of methodologies and techniques converge on the notion that adults and children with attention deficit hyperactivity disorder (ADHD) have similar deficits, but there is limited knowledge about whether adult retrospective reports reflect similar genetic and environmental influences implicated in childhood ADHD.MethodDSM-IV ADHD symptoms were collected retrospectively from 3896 young adults participating in the National Longitudinal Study of Adolescent Health. Responses from this genetically informative sample of same- and opposite-sex twins and siblings were used to determine the magnitude of genetic and environmental influences. Possible gender differences in these effects were also examined. The degree of familial specificity of the genetic and environmental influences on the Inattentive and Hyperactive-Impulsive symptom dimensions was also determined.ResultsAdditive genetic effects contributed moderately to DSM-IV Inattentive, Hyperactive-Impulsive and Combined ADHD subtypes (heritability estimates of 0.30–0.38). Individual-specific influences accounted for the remaining proportion of the variance. Both genetic and individual-specific environmental effects contributed to the covariation of Inattentive and Hyperactive-Impulsive symptomologies.ConclusionsResults from our genetic analyses agree with previous findings based on self-assessment of current and retrospectively reported ADHD symptoms in adolescents and adults. Large individual-specific environmental influences as identified here suggest that current questionnaires used for retrospective diagnoses may not provide the most accurate reconstruction of the etiological influences on childhood ADHD in general population samples.


2017 ◽  
Vol 32 (14) ◽  
pp. 1083-1091 ◽  
Author(s):  
Ozalp Ekinci ◽  
Çetin Okuyaz ◽  
Semra Erdoğan ◽  
Serkan Gunes ◽  
Nuran Ekinci ◽  
...  

Author(s):  
Lauren E Oddo ◽  
Samuel F Acuff ◽  
Melanie B Arenson ◽  
Assaf Oshri ◽  
Andrea Chronis-Tuscano ◽  
...  

Abstract Aims Contemporary theories of attention-deficit/hyperactivity disorder (ADHD) and alcohol use disorder (AUD) emphasize core dysfunctions in reward-related processes and behaviors as pathognomonic characteristics. However, to date, it is unclear which domains of reward functioning are unique to ADHD versus AUD symptom dimensions, and which represent underlying shared correlates. Methods The current study employed secondary data analyses from a large community sample of emerging adults (N = 602; 57.3% female) and novel transdiagnostic modeling (i.e. bi-factor confirmatory factor analyses and structural equation modeling) of ADHD, AUD and shared symptom dimensions to identify unique and common reward-related dimensions: environmental suppressors, reward probability, hedonic capacity, proportionate substance-related reinforcement and delay discounting. Results The presence of environmental suppressors was the only reward-related construct that correlated with the underlying ADHD-AUD shared dimension. The AUD symptom dimension was uniquely associated with proportionate substance-related reinforcement, whereas the ADHD symptom dimension was uniquely associated with limited reward probability. No significant associations were found for delay discounting or hedonic capacity. Conclusions These novel findings highlight specific aspects of reward-related functioning in ADHD, AUD and shared symptom dimensions. In so doing, this work meaningfully advances theoretical conceptualizations of these two commonly co-occurring presentations and suggests future directions for research on transdiagnostic correlates. Future longitudinal studies should include clinical samples with diagnoses of AUD and ADHD to further identify underlying correlates over time.


2012 ◽  
Vol 121 (4) ◽  
pp. 991-1010 ◽  
Author(s):  
Erik G. Willcutt ◽  
Joel T. Nigg ◽  
Bruce F. Pennington ◽  
Mary V. Solanto ◽  
Luis A. Rohde ◽  
...  

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