Effects of smartphone use before resistance exercise on inhibitory control, heart rate variability, and countermovement jump

Author(s):  
Dalton de Lima-Junior ◽  
Leonardo S. Fortes ◽  
Maria E. C. Ferreira ◽  
Petrus Gantois ◽  
Bruno Teixeira Barbosa ◽  
...  
Author(s):  
A. H. Beg ◽  
Noraziah Ahmad ◽  
Nawsher Khan ◽  
Ahmed N Abd Alla ◽  
Muhammad Nubli ◽  
...  

1986 ◽  
Vol 27 (4) ◽  
pp. 548-559
Author(s):  
Charles E. Short ◽  
Jean-Louis Stauffer ◽  
Gary Goldberg ◽  
Outi Vainio

2013 ◽  
Vol 34 (2) ◽  
pp. 98-108 ◽  
Author(s):  
Rodrigo Polaquini Simões ◽  
Viviane Castello-Simões ◽  
Renata Gonçalves Mendes ◽  
Bruno Archiza ◽  
Daniel Augusto dos Santos ◽  
...  

Author(s):  
Amit Frenkel ◽  
Yoav Bichovsky ◽  
Natan Arotsker ◽  
Limor Besser ◽  
Ben-Zion Joshua ◽  
...  

Background: Beta blockers, mainly propranalol, are usually administered to control heart rate in patients with thyrotoxicosis, especially when congestive heart failure presents. However, when thyrotoxicosis is not controlled, heart rate may be difficult to control even with maximal doses of propranolol. This presentation alerts physicians to the possibility of using ivabradine, a selective inhibitor of the sinoatrial pacemaker, for the control of heart rate. Case presentation: We present a 37-year-old woman with thyrotoxicosis and congestive heart failure whose heart rate was not controlled with a maximal dose of beta blockers during a thyroid storm. The addition of ivabradine, a selective inhibitor of the sinoatrial pacemaker, controlled her heart rate within 48 hours. Conclusion: Ivabradine should be considered in patients with thyrotoxicosis, including those with heart failure, in whom beta blockers are insufficient to control heart rate


1981 ◽  
Vol 240 (6) ◽  
pp. H874-H880 ◽  
Author(s):  
P. D. Gupta ◽  
M. Singh

We investigated the mechanisms underlying heart rate changes resulting from systemic hypoxia in anesthetized artificially ventilated dogs with low control heart rate (less than or equal to 93 beats/min). We observed that systemic hypoxia evoked tachycardia in intact dogs that was not significantly different from that evoked in dogs with beta-adrenergic blockade (BB). Also, tachycardia elicited in dogs with BB plus spinal section at C3 (BBSS) was significantly greater than in dogs with BBSS plus bilateral section of carotid sinus nerves. Furthermore, under various anesthetics, intracarotid injection of sodium cyanide induced a tachycardia response in dogs with low control heart rate (less than or equal to 97 beats/min) and a bradycardia response in dogs with high control heart rate (greater than or equal to 130 beats/min). These results suggest that 1) when the resting cardiac parasympathetic tone is high, systemic hypoxia evokes tachycardia which is mediated predominantly through efferent vagus nerves and 2) the stimulation of carotid chemoreceptors causes excitation of both cardioacceleratory and cardioinhibitory reflexes, the resultant response being dependent on the prevalent autonomic drive.


2007 ◽  
Vol 16 ◽  
pp. S185
Author(s):  
H. Branagan ◽  
R. Braaksma ◽  
C. Jenkins ◽  
L. Hanekom ◽  
B. Venkatesh ◽  
...  

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