Effects of Cell Density on Cell Growth in a Clone of Mouse Liver Cells2

1957 ◽  
Keyword(s):  
Cell Growth ◽  
Cell Density ◽  
Mouse Liver ◽  
Liver Cells

scholarly journals Inhibition of sterol biosynthesis in L cells and mouse liver cells by 15-oxygenated sterols.

1977 ◽  
Vol 252 (24) ◽  
pp. 8975-8980 ◽  
Author(s):  
G.J. Schroepfer ◽  
E.J. Parish ◽  
H.W. Chen ◽  
A.A. Kandutsch
Keyword(s):  
Mouse Liver ◽  
Liver Cells ◽  
Sterol Biosynthesis ◽  
L Cells

Protection from oxidative damage in mouse liver cells

1992 ◽  
Vol 64-65 ◽  
pp. 581-587 ◽  
Author(s):  
Andrew W. Harman ◽  
Gregory M. Adamson ◽  
Scott G. Shaw
Keyword(s):  
Oxidative Damage ◽  
Mouse Liver ◽  
Liver Cells

Lowered antioxidant enzymes in spontaneously transformed embryonic mouse liver cells in culture

1993 ◽  
Vol 14 (7) ◽  
pp. 1457-1463 ◽  
Author(s):  
Yi Sun ◽  
Larry W. Oberley ◽  
Terry D. Oberley ◽  
James H. Elwell ◽  
Elaine Sierra-Rivera
Keyword(s):  
Antioxidant Enzymes ◽  
Mouse Liver ◽  
Liver Cells ◽  
Embryonic Mouse ◽  
Cells In Culture

Direct preparation and G-banding of chromosomes of mouse liver cells

1986 ◽  
Vol 164 (3) ◽  
pp. 177-181 ◽  
Author(s):  
Håvard Danielsen ◽  
Mette Lier ◽  
Anton Brøgger ◽  
Albrecht Reith
Keyword(s):  
Mouse Liver ◽  
Liver Cells ◽  
Direct Preparation

Mitotic catastrophe as a prestage to necrosis in mouse liver cells treated withHelicobacter pullorumsonicates

2009 ◽  
Vol 270 (8) ◽  
pp. 921-928 ◽  
Author(s):  
Liesbeth M. Ceelen ◽  
Freddy Haesebrouck ◽  
Katharina D'Herde ◽  
Dmitri V. Krysko ◽  
Herman Favoreel ◽  
...  
Keyword(s):  
Mouse Liver ◽  
Liver Cells ◽  
Mitotic Catastrophe

Effect of acute exposure to PFOA on mouse liver cells in vivo and in vitro

2017 ◽  
Vol 24 (31) ◽  
pp. 24201-24206 ◽  
Author(s):  
Xinmou Wu ◽  
Minqing Liang ◽  
Zhao Yang ◽  
Min Su ◽  
Bin Yang
Keyword(s):  
Mouse Liver ◽  
Liver Cells ◽  
Acute Exposure

scholarly journals CHANGES OF 8-OXO-2'-DEOXYGUANOSINE LEVEL IN MOUSE LIVER CELLS DNA IN CASE OF ACUTE TOXIC STRESS

2016 ◽  
Vol 11 (6) ◽  
pp. 68-74
Author(s):  
N. V. Marmiy ◽  
D. S. Esipov
Keyword(s):  
Carbon Tetrachloride ◽  
Liver Tissue ◽  
Mouse Liver ◽  
Pathological Change ◽  
Liver Cells ◽  
Antioxidant Systems ◽  
Toxic Stress ◽  
Laboratory Mice ◽  
Short Term ◽  
Initial Injection

The changes of the 8-oxo-2'-deoxyguanosine (8-oxo-dG)/dG ratio in the DNA of laboratory mice hepatocytes under the influence of toxic stress were studied. It was shown that the injection of carbon tetrachloride causes the growth of 8-oxo-dG level. A rapid increase in the level of 8-oxo-dG in DNA occurs during the first day of the experiment at short-term toxic stress. Subsequently, 48 hours after the initial injection, the level of 8-oxo-dG decreases to the control values. This change in the value of the biomarker can be attributed to the activation of the reparative and antioxidant systems. The subsequent injection results again in an increase of 8-oxo-dG level, and the latter only increases thereafter. This reflects the exhaustion of the reparative potential of the organism and accompanies the progress of inflammation and pathological change of the liver tissue.

Amino acid uptake regulation by cell growth in cultured hepatocytes isolated from fetal and adult rats

1992 ◽  
Vol 12 (2) ◽  
pp. 135-141 ◽  
Author(s):  
S. Leoni ◽  
S. Spagnuolo ◽  
M. Massimi ◽  
F. Terenzi ◽  
L. Conti Devirgiliis
Keyword(s):  
Amino Acid ◽  
Cell Growth ◽  
Growth Inhibition ◽  
Cell Density ◽  
Amino Acid Uptake ◽  
Growth Stimulation ◽  
Acid Uptake ◽  
Adult Rats ◽  
Dependent Growth ◽  
Differentiation Stage

Amino acid uptake mediated by system A was studied in cultured fetal and adult hepatocytes, subjected to growth stimulation by EGF and insulin, or to growth inhibition by high cell density. The mitogenic stimulation induced a strong transport increase only in fetal cells, while the cell density-dependent growth inhibition, probably mediated by molecules present on adult hepatocyte membranes, provoked the decrease of amino acid uptake only in the adult cells. The results indicate that the different modulation of amino acid transport by cell growth is dependent on the age and the differentiation stage of hepatocytes.

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