Intracerebral haemorrhage in older people

2020 ◽  
pp. 141-164
Author(s):  
Zhe Kang Law ◽  
Nikola Sprigg

Chapter 10 refers to ‘Intracerebral haemorrhage in older people’. The chapter has been written by the investigators of the TICH-2 trial, one of the largest trial of intracerebral haemorrhage in the world. The authors review the aetiology, epidemiology, risk factors and in particular the causes of intracerebral haemorrhage in older people (including hypertensive arteriopathy, cerebral amyloid angiopathy, cerebral small vessel disease, anticoagulant-related bleeds, haemorrhagic transformation of infarcts). Less common causes such as haemorrhagic brain metastasis, cortical vein thrombosis, arteriovenous malformation, cavernomas, dural arteriovenous fistulas, aneurysms, and septic or mycotic aneurysms are also discussed. Evolution of neuroimaging patterns after a bleed, approach to investigations and management are discussed in detail, in addition to important trial evidence.

2021 ◽  
pp. 197140092110490
Author(s):  
Skander Sammoud ◽  
Nadia Hammami ◽  
Dhaker Turki ◽  
Fatma Nabli ◽  
Samia Ben Sassi ◽  
...  

Pial arteriovenous fistulas (AVFs) are rare neurovascular malformations. They differ from arteriovenous malformations (AVMs) in that they involve single or multiple feeding arteries, draining directly into a dilated cortical vein with no intervening nidus. Pial and dural AVFs differ in blood supply, as the first originate from pial or cortical arteries and the latter from outside the dural leaflets. Unlike dural AVFs, most of the pial AVFs are supratentorial. The vast majority are congenital, manifesting during infancy. Acquired pial AVFs are significantly rarer and occur after vasculopathy, head trauma, brain surgery, or cerebral vein thrombosis. We describe a unique case of an acquired pial AVF in a 50-year-old man secondary to a cortical vein thrombosis manifesting as a focal-onset seizure with secondary generalization. A cerebral digital subtraction angiography revealed a low-flow pial AVF fed by a postcentral branch of the left middle cerebral artery draining to the superior sagittal sinus via a cortical vein. It also showed a collateral venous circulation adjacent to the previously thrombosed left parietal vein. There was no evidence of an associated dural AVF or venous varix. Endovascular treatment was scheduled three months later, but the angiogram preceding the embolization showed spontaneous and complete closure of the malformation. To our knowledge, this is the first case illustrating acquired pure pial AVF unaccompanied by a dural component following cortical vein thrombosis, eventually resulting in an unprompted closure.


2007 ◽  
Vol 34 (S 2) ◽  
Author(s):  
J Linn ◽  
T Pfefferkorn ◽  
S Michl ◽  
M Wiesmann ◽  
S Hartz ◽  
...  

BMC Neurology ◽  
2021 ◽  
Vol 21 (1) ◽  
Author(s):  
Hongfeng Wen ◽  
Di Jin ◽  
Yu Chen ◽  
Bin Cui ◽  
Tianyi Xiao

Abstract Background Cerebral venous thrombosis (CVT) occurs rarely in the general population and is frequently associated with confused clinical findings and delayed diagnosis. Isolated cerebellar cortical vein thrombosis is a very rare phenomenon. Case presentation This report describes a case with CVT, which is manifested as space-occupying lesions of the cerebellar hemisphere and mimics a cerebellar tumor at the beginning. The diagnosis of CVT was finalized given the laboratory and brain biopsy findings. The etiology may be related to polycythemia vera with Janus Kinase 2 V617F mutation. Conclusion Isolated cerebellar vein thrombosis should be considered when swelling and enhancing cerebellar lesions are detected. Polycythemia vera, especially with a positive JAK2 V617F mutation, may be a rare risk factor for CVT.


2013 ◽  
Vol 61 (2) ◽  
pp. 173 ◽  
Author(s):  
PedroTadao Hamamoto Filho ◽  
RobertoColichio Gabarra ◽  
GabrielPereira Braga ◽  
LucileneSilva Ruiz e Resende ◽  
Rodrigo Bazan ◽  
...  

BMC Neurology ◽  
2016 ◽  
Vol 16 (1) ◽  
Author(s):  
Nobuhiko Arai ◽  
Masanao Tabuse ◽  
Akiyoshi Nakamura ◽  
Hiromichi Miyazaki

1987 ◽  
Vol 7 (2) ◽  
pp. 54
Author(s):  
D. Younker ◽  
M. M. Jones ◽  
J. Adenwala ◽  
A. Citrin ◽  
T. H. Joyce

Sign in / Sign up

Export Citation Format

Share Document