A rare complication following general anaesthesia for gynaecological laparotomy: postoperative visual loss due to bilateral occipital lobe infarction

2013 ◽  
Vol 30 ◽  
pp. 240-241
Author(s):  
C. Araz ◽  
Z. Kayhan ◽  
M. Gürler ◽  
C. Küçükerdönmez
Keyword(s):  
General Anaesthesia ◽  
Visual Loss ◽  
Rare Complication ◽  
Occipital Lobe ◽  
Postoperative Visual Loss

scholarly journals Post Appendectomy Blindness, Extremely rare complication after abdominal surgery

2021 ◽  
pp. 189
Author(s):  
Keyword(s):  
Laparoscopic Appendectomy ◽  
Visual Loss ◽  
Rare Complication ◽  
Occipital Lobe ◽  
Occipital Cortex ◽  
Underlying Disease ◽  
Young Female ◽  
Ischemic Optic Neuropathy ◽  
Ocular Surgery ◽  
Perioperative Visual Loss

Perioperative visual loss (POVL) is a drastic complication that can result after ocular and more commonly non-ocular surgery, mostly reported after spine and cardiac bypass procedures. Despite the rarity of such complication, it has been reported following the abdominal surgeries. Overall, the most common cause of POVL is ischemic optic neuropathy but any pathology to the optical system from the cornea to the occipital lobe can lead to this rare phenomenon. Here, we are reporting the second case in the literature of post-operative visual loss after laparoscopic appendectomy. A young female, with no underlying disease, underwent laparoscopic appendectomy after septic shock secondary to acute appendicitis. Postoperatively, patient complained of complete blindness after extubation. Neurologic examination revealed bilateral near complete blindness, and hemodynamic ischemic stroke in the occipital cortex.

scholarly journals Postoperative visual loss associated with spine surgery

2005 ◽  
Vol 15 (4) ◽  
pp. 479-484 ◽  
Author(s):  
Brian Gill ◽  
James E. Heavner
Keyword(s):  
Spine Surgery ◽  
Visual Loss ◽  
Postoperative Visual Loss

scholarly journals Acute Visual Loss as a First Sign of Hyperhomocysteinaemia

1970 ◽  
Vol 12 (3) ◽  
pp. 172-174
Author(s):  
Aggeliki Kolea ◽  
Aggelos Baltatzidis ◽  
Vasileios Margaritis ◽  
Belal Almoghrabi ◽  
Irini Kaldi ◽  
...  
Keyword(s):  
Magnetic Resonance Imaging ◽  
Magnetic Resonance ◽  
Visual Field ◽  
Genetic Analysis ◽  
Visual Loss ◽  
Methylenetetrahydrofolate Reductase ◽  
Occipital Lobe ◽  
Field Testing ◽  
Resonance Imaging ◽  
Acute Visual Loss

A 38-year-old man presented with an acute right homonymous visual field defect due to occipital lobe infarct caused by hyperhomocysteinaemia. Visual field testing, magnetic resonance imaging, laboratory studies, and genetic analysis were carried out. On magnetic resonance imaging, a left occipital lesion with bright signal on the diffusion-weighted and fluid-attenuated inversion recovery images suggested a diagnosis of an acute infarct. Blood tests revealed raised homocysteine of 52.08 μmol/L (reference range, <15 μmol/L) and genetic analysis showed the patient to be homozygote to 5, 10-methylenetetrahydrofolate reductase deficiency. Hyperhomocysteinaemia is a rare causes of acute visual loss due to cerebral ischaemia and should always be suspected and investigated with the appropriate tests to diagnose the condition and limit further vision deterioration.

scholarly journals SUCCESSFUL TREATMENT OF TRAUMATIC ORBITAL APEX SYNDROME WITH LOW DOSE STEROIDS - A CASE REPORT

2020 ◽  
pp. 214-215
Author(s):  
Tharini Senthamizh ◽  
Subashini Kaliaperumal
Keyword(s):  
Visual Loss ◽  
Low Dose ◽  
Rare Complication ◽  
Signs And Symptoms ◽  
Complete Recovery ◽  
Dramatic Improvement ◽  
Orbital Apex ◽  
Orbital Apex Syndrome ◽  
Surgical Evacuation ◽  
Displaced Fracture

Traumatic Orbital Apex Syndrome is a rare complication of trauma presenting with visual loss, ophthalmoplegia, and anesthesia of cornea, forehead and maxillary regions. It requires immediate action as it poses great threat to permanent visual loss. The incidence of Traumatic orbital apex syndrome is very less compared to Superior Orbital fissure syndrome and traumatic optic neuropathy alone and only few cases have been reported till now. Management depends on the cause, those with displaced fracture fragments are treated by surgical decompression whereas those with edema or hematoma causing compression can be treated with steroids or surgical evacuation of hematoma. Previous reports have proved the usefulness of mega dose steroids in such cases. We report a case of Traumatic Orbital Apex Syndrome who presented with painful proptosis, visual loss, ophthalmoplegia and loss of sensation in periorbital region. Imaging confirmed hematoma causing compression of neurovascular structures and hence a trial of low dose corticosteroids was started. Our patient showed dramatic improvement in signs and symptoms with complete recovery in three weeks. Low dose steroids can be considered as an alternative to mega dose steroids to treat patients with indirect traumatic Orbital Apex Syndrome, thereby reducing the necessity of surgical evacuation in such cases.

Progressive visual loss and severe retinal degeneration in a captive Kodiak bear (Ursus arctos mittendorfi)

2020 ◽  
Vol 8 (3) ◽  
pp. e001115
Author(s):  
Kelly A Caruso ◽  
Seth Koch ◽  
Benjamin David Reynolds ◽  
Paul Massimo Giannoni McCarthy ◽  
Cameron J Whittaker
Keyword(s):  
Electrical Activity ◽  
Retinal Degeneration ◽  
General Anaesthesia ◽  
Outer Layer ◽  
Visual Loss ◽  
Ursus Arctos ◽  
Hepatic Neoplasm ◽  
Vascular Attenuation

A 27-year-old female Kodiak bear displayed signs of progressive visual loss over an 18-year period. The bear was examined under general anaesthesia, where fixed mydriatic pupils, a diffusely hyper-reflective retina with vascular attenuation was observed. An electroretinogram was performed bilaterally, revealing no electrical activity associated with either retina and confirming a diagnosis of severe bilateral retinal degeneration. The bear was euthanased eight months later due to a diagnosis of a metastasised hepatic neoplasm. Histopathology of the bear’s eyes revealed marked outer layer retinal degeneration. The aetiology of this retinal degeneration could not be confirmed.

Postoperative Visual Loss

2017 ◽  
Author(s):  
Lorri A. Lee
Keyword(s):  
Risk Factors ◽  
Associated Factors ◽  
Visual Loss ◽  
Venous Pressure ◽  
Mechanism Of Injury ◽  
Initial Management ◽  
Surgical Emergencies ◽  
Fluid Shifts ◽  
Prolonged Hypotension ◽  
Postoperative Visual Loss

Postoperative visual loss (POVL) has multiple diagnoses and contributory causes including emboli, direct globe compression, prolonged elevation of venous pressure in the head with associated large fluid shifts, prolonged hypotension, periorbital trauma, preexisting ophthalmologic anatomic risk factors, and many other associated factors. It frequently results in permanent injury and severe disability. Though any loss of vision postoperatively should prompt an emergent or urgent ophthalmologic consultation, some of the rarer causes of POVL are considered true medical or surgical emergencies. This chapter briefly discusses these rarer causes and primarily focuses on the more common POVL diagnoses, including the mechanism of injury, appropriate assessment, and initial management.

scholarly journals A case for routine tonometry to avoid postoperative visual loss

2011 ◽  
Vol 107 (4) ◽  
pp. 648-649 ◽  
Author(s):  
N.A. Usoltsev ◽  
H. Shankar ◽  
C.M. Zainer
Keyword(s):  
Visual Loss ◽  
Postoperative Visual Loss
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