scholarly journals SUCCESSFUL TREATMENT OF TRAUMATIC ORBITAL APEX SYNDROME WITH LOW DOSE STEROIDS - A CASE REPORT

2020 ◽  
pp. 214-215
Author(s):  
Tharini Senthamizh ◽  
Subashini Kaliaperumal

Traumatic Orbital Apex Syndrome is a rare complication of trauma presenting with visual loss, ophthalmoplegia, and anesthesia of cornea, forehead and maxillary regions. It requires immediate action as it poses great threat to permanent visual loss. The incidence of Traumatic orbital apex syndrome is very less compared to Superior Orbital fissure syndrome and traumatic optic neuropathy alone and only few cases have been reported till now. Management depends on the cause, those with displaced fracture fragments are treated by surgical decompression whereas those with edema or hematoma causing compression can be treated with steroids or surgical evacuation of hematoma. Previous reports have proved the usefulness of mega dose steroids in such cases. We report a case of Traumatic Orbital Apex Syndrome who presented with painful proptosis, visual loss, ophthalmoplegia and loss of sensation in periorbital region. Imaging confirmed hematoma causing compression of neurovascular structures and hence a trial of low dose corticosteroids was started. Our patient showed dramatic improvement in signs and symptoms with complete recovery in three weeks. Low dose steroids can be considered as an alternative to mega dose steroids to treat patients with indirect traumatic Orbital Apex Syndrome, thereby reducing the necessity of surgical evacuation in such cases.

1993 ◽  
Vol 107 (9) ◽  
pp. 840-842 ◽  
Author(s):  
Yousry El-Sayed ◽  
Hamad Al-Muhaimeid

AbstractAcute visual loss may occur in association with sinusitis either as a complication of orbital cellulitis or, less frequently, as a part of the orbital apex syndrome. We describe two cases of temporary monocular visual loss caused by sinusitis. In one case the visual loss was due to orbital cellulitis; while in the other patient it was due to incompletely developed orbital apex syndrome. This later mode of presentation is called ‘partial orbital apex syndrome’ by some authors and ‘posterior orbital cellulitis’ by others.The relationship between sinusitis and blindness is discussed.


Orbit ◽  
2015 ◽  
Vol 34 (3) ◽  
pp. 172-174 ◽  
Author(s):  
Rajeev G. Tanawade ◽  
Reshma S. Thampy ◽  
Scott Wilson ◽  
I. Chris Lloyd ◽  
Jane Ashworth

2015 ◽  
Vol 129 (6) ◽  
pp. 540-543 ◽  
Author(s):  
K Sikka ◽  
R Agrawal ◽  
K Devraja ◽  
J V Lodha ◽  
A Thakar

AbstractBackground:Animate foreign bodies in the ear are frequent occurrences in otology practice. Such foreign bodies may lead to hazardous complications.Method:This paper describes a retrospective study of six patients with a recent history of an insect in the ear who presented with various complications following intervention received elsewhere.Results:An insect was retrieved from the external auditory canal in four cases and from the antrum in two cases. The patients presented with progressive otological complications: two patients who presented with orbital apex syndrome and cavernous sinus thrombosis succumbed to the disease; three patients suffered sensorineural hearing loss; and two patients had persistent facial palsy. One patient with sigmoid sinus thrombosis, who presented early, experienced complete recovery.Conclusion:Insects in the ear can lead to hazardous complications. Animate foreign bodies should preferably be managed by a trained otologist, even in an emergency setting. Patients with delayed presentation and complications have a guarded prognosis.


2014 ◽  
Vol 7 (4) ◽  
pp. 318-322 ◽  
Author(s):  
Atsushi Imaizumi ◽  
Kunihiro Ishida ◽  
Yasunari Ishikawa ◽  
Izuru Nakayoshi

Orbital apex syndrome is an uncommon but severe ocular complication of craniomaxillofacial fracture. The optimal treatment strategy for this very rare traumatic syndrome has not been well established. We present a case in which traumatic orbital apex syndrome was caused by direct compression from the displaced fracture segments. Visual and extraocular function both improved quickly after emergency decompression surgery. This case suggests that managing the direct type of traumatic orbital apex syndrome with craniomaxillofacial fracture with a combination of urgent reduction of impinging bone and decompression of affected nerves is an effective strategy.


2019 ◽  
Vol 98 (10) ◽  
pp. 609-612 ◽  
Author(s):  
Tom Shokri ◽  
Brad E. Zacharia ◽  
Jessyka G. Lighthall

Orbital apex syndrome (OAS) is a rare ocular complication following craniomaxillofacial trauma. This traumatic syndrome is a combination of features seen in both superior orbital fissure syndrome and traumatic orbital neuropathy due to nerve impingement. Due in part to the rarity of this disorder, the optimal treatment of traumatic OAS has yet to be determined. We present a case in which traumatic OAS was caused by direct compression due to a displaced fracture segment from the superior orbit. The patient was successfully treated with a combination of emergent decompression and urgent reconstruction suggesting that this may be an effective strategy in OAS resulting from direct nerve compression as a result of craniomaxillofacial fracture.


2006 ◽  
Vol 7 (2) ◽  
pp. 130-136 ◽  
Author(s):  
R.A. Machado ◽  
R. Lanes Silveira ◽  
H.O.I Borges ◽  
A.M. Bourguignon Filho ◽  
M. Gerhardt de Oliveira

Abstract Retrobulbar hemorrhage is a rare complication that may occur after mid-face injuries or following soft and hard tissue surgery around the eyes. The cardinal signs and symptoms of retrobulbar hemorrhage are pain, diplopia, ophthalmoplegia, a progression of increasing proptosis, and decreasing visual acuity leading to blindness. The diagnosis can be confirmed with computed tomography (CT) of the orbit or with ocular ultrasound. These diagnostic images are also important to define the size of the hematoma. This report describes a traumatic retrobulbar hemorrhage. In this case there were no signs of acute visual loss, and conservative treatment was possible without surgical intervention. Citation Machado RA, Silveira RL, Borges HOI, Filho AMB, de Oliveira G. Retrobulbar Hemorrhage: A Case Report. J Contemp Dent Pract 2006 May;(7)2:130-136.


2001 ◽  
Vol 59 (3B) ◽  
pp. 806-808 ◽  
Author(s):  
Yvens B. Fernandes ◽  
Ricardo Ramina ◽  
Guilherme Borges ◽  
Luciano S. Queiroz ◽  
Marcos V.C. Maldaun ◽  
...  

We report the case of a 73-year-old female who presented facial numbness and pain in the first division of the trigeminal nerve, ptosis, diplopia and visual loss on the right side for the previous four months. The neurological, radiological and histological examination demonstrated a rare case of invasive fungal aspergillosis of the central nervous system, causing orbital apex syndrome, later transformed in temporal brain abscess. She died ten months later due to respiratory and renal failure in spite of specific antimycotic therapy.


2015 ◽  
Vol 4 (2) ◽  
pp. 177
Author(s):  
CS Sandhya ◽  
DMurali Krishna ◽  
C Jagannath ◽  
G Srinivas ◽  
K Radhika

Author(s):  
Masood Bagheri ◽  
Ahad Jafari ◽  
Sasan Jafari

2021 ◽  
pp. 32-40
Author(s):  
Yusuke Murai ◽  
Takuji Kurimoto ◽  
Sotaro Mori ◽  
Kaori Ueda ◽  
Mari Sakamoto ◽  
...  

We report a rare case of granulomatosis with polyangiitis (GPA) presenting with bilateral orbital apex syndrome (OAS). A 73-year-old woman with a history of endoscopic sinus surgery for ethmoidal sinusitis experienced a sudden decrease in visual acuity (VA) of both eyes. At the initial examination, her VA had decreased to 0.01 in the right eye and 0.03 in the left eye, and eye movement in both eyes was mildly limited in all directions. Visual field tests of both eyes showed a large central scotoma. Laboratory tests revealed an elevation of myeloperoxidase-anti-neutrophil cytoplasmic antibody. Facial computed tomography demonstrated a thickened mucosal membrane in the entire ethmoidal sinus, and the posterosuperior walls of Onodi cells filled with infiltrative lesions had thinned. Orbital magnetic resonance imaging showed severe inflammation in the orbital apex. From these clinical findings, the patient was diagnosed with GPA presenting with OAS associated with ethmoid sinusitis. Emergent endoscopic sinus surgery was performed for biopsy and debridement of the ethmoidal and sphenoid sinusitis to decompress the optic nerve. One day after endoscopic sinus surgery, the patient’s VA and visual field were improved, and steroid pulse therapy was commenced postoperatively. Four days later, VA had recovered to 1.0 in both eyes, and eye movement and visual field had were improved. Although OAS is a rare manifestation, early surgical treatment should be considered when the orbital lesion presents as risk of rapid deterioration of visual function in patients with GPA.


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