Arthritis after Experimental Infection with Yersinia enterocolitica 0:3 in Rabbits

2008 ◽  
Vol 48 (1) ◽  
pp. 43-53
Author(s):  
A. Vesselinova ◽  
H. Najdenski ◽  
S. Nikolova ◽  
D. Wesselinova
Keyword(s):  
Yersinia Enterocolitica ◽  
Experimental Infection

Studies of Yersinia enterocolitica 0:3 Experimental Infection in Pigs

1998 ◽  
Vol 45 (1-10) ◽  
pp. 59-64 ◽  
Author(s):  
H. Najdenski ◽  
S. Nikolova ◽  
A. Vesselinova ◽  
P. Neikov
Keyword(s):  
Yersinia Enterocolitica ◽  
Experimental Infection

scholarly journals Differentiation between serological responses to Brucella suis and Yersinia enterocolitica serotype O[ratio ]9 after natural or experimental infection in pigs

2005 ◽  
Vol 134 (2) ◽  
pp. 347-357 ◽  
Author(s):  
G. JUNGERSEN ◽  
V. SØRENSEN ◽  
S. B. GIESE ◽  
J. A. STACK ◽  
U. RIBER
Keyword(s):  
Yersinia Enterocolitica ◽  
Experimental Infection ◽  
False Positive ◽  
Indirect Elisa ◽  
Serum Samples ◽  
Cross Sectional ◽  
O Antigen ◽  
Serotype O ◽  
Brucella Suis ◽  
Limited Period

False-positive serological reactions (FPSR) due to infections with Yersinia enterocolitica serotype O[ratio ]9 (YeO[ratio ]9) are a problem in tests for brucellosis. In the present study, FPSR in classical and novel tests for brucellosis following experimental infections of pigs with YeO[ratio ]9 were compared with responses of B. suis biovar 2-inoculated pigs. FPSR were limited to 2–9 weeks post-YeO[ratio ]9 inoculation, while B. suis-infected pigs were test-positive throughout the 21-week period of investigation. Although YeO[ratio ]9-inoculated pigs exhibited FPSR in Brucella tests for a limited period of time, the serological responses in a YeO[ratio ]9-purified O-antigen indirect ELISA did not decrease accordingly. Analysis of available cross-sectional serum samples from pig herds naturally infected with YeO[ratio ]9 or B. suis biovar 2 confirmed that the observed difference in the duration of the serological responses between the two infections could be used to discriminate between herds infected with B. suis biovar 2 and YeO[ratio ]9.

Ecological studies of Yersinia enterocolitica. II. Experimental infection with Y. enterocolitica in pigs

1984 ◽  
Vol 9 (4) ◽  
pp. 375-381 ◽  
Author(s):  
H. Fukushima ◽  
R. Nakamura ◽  
Y. Ito ◽  
K. Saito ◽  
M. Tsubokura ◽  
...  
Keyword(s):  
Yersinia Enterocolitica ◽  
Experimental Infection ◽  
Ecological Studies

Effect of the combination of clavulanic acid and cephalothin on an experimental infection with Yersinia enterocolitica in iron-overloaded mice

1988 ◽  
Vol 22 (5) ◽  
pp. 707-713 ◽  
Author(s):  
Maria Jimenez-Valera ◽  
Alfonso Ruiz-Bravo ◽  
Maria del Carmen Gómez ◽  
Alberto Ramos-Cormenzana
Keyword(s):  
Clavulanic Acid ◽  
Yersinia Enterocolitica ◽  
Experimental Infection

scholarly journals Serological and biochemical characteristics of virulence plasmid of Yersinia enterocolitica isolates from chicken in the Islamic Republic of Iran

2000 ◽  
Vol 6 (2-3) ◽  
pp. 409-415
Author(s):  
M. Motallebi
Keyword(s):  
Human Serum ◽  
Yersinia Enterocolitica ◽  
Experimental Infection ◽  
Virulence Plasmid ◽  
Plasmid Curing ◽  
Islamic Republic ◽  
Biochemical Characteristics ◽  
Calcium Dependency ◽  
Islamic Republic Of Iran ◽  
Pathogenic Yersinia

Pathogenic Yersinia enterocolitica harbour plasmid that is essential for virulence. We studied the characteristics of virulence plasmid using serological, biochemical and bioassay tests in Y. enterocolitica isolates of chicken using plasmid curing. Plasmid-cured isogenic derivatives [2029c and 2150c] were obtained from two isolates of Y. enterocolitica [RTCC 2029 and RTCC 2150]. The results demonstrated that plasmid-bearing isolates [2029 and 2150] were human-serum-resistant when grown at 37°C, but were sensitive when grown at 25°C, whereas plasmid-cured isolates [2029c and 2150c] were sensitive when grown at both temperatures. Also autoagglutination, calcium-dependency tests and experimental infection in mice demonstrated that these phenotypes were associated with the virulence plasmid

Arthritis after Experimental Infection with Yersinia enterocolitica 0:3 in Rabbits

2001 ◽  
Vol 48 (1) ◽  
pp. 43-53 ◽  
Author(s):  
A. Vesselinova ◽  
H. Najdenski ◽  
S. Nikolova ◽  
D. Wesselinova
Keyword(s):  
Yersinia Enterocolitica ◽  
Experimental Infection

Experimental infection with Yersinia enterocolitica serovar 0:8 in Beagle dogs

1995 ◽  
Vol 47 (1-2) ◽  
pp. 71-77 ◽  
Author(s):  
Hideki Hayashidani ◽  
Ken-ichi Kaneko ◽  
Kenji Sakurai ◽  
Masuo Ogawa
Keyword(s):  
Yersinia Enterocolitica ◽  
Experimental Infection ◽  
Beagle Dogs

Experimental Infection of Mice with Yersinia enterocolitica Serotype O9 by Oral and Parenteral Routes: Spreading and Enterotropism of Virulent Yersiniae

1999 ◽  
Vol 38 (5) ◽  
pp. 257-263 ◽  
Author(s):  
Alfonso Ruiz-Bravo ◽  
Encarnacion Moreno ◽  
Antonio Sampedro ◽  
Maria Jimenez-Valera
Keyword(s):  
Yersinia Enterocolitica ◽  
Experimental Infection

Labelling of non-A, non-B hepatitis infected chimpanzee hepatocytes with nuclease-gold conjugates

1984 ◽  
Vol 42 ◽  
pp. 250-251
Author(s):  
G. D. Gagne ◽  
M. F. Miller ◽  
D. A. Peterson
Keyword(s):  
Endoplasmic Reticulum ◽  
Experimental Infection ◽  
Uranyl Acetate ◽  
Type I ◽  
Cellular Injury ◽  
Type Ii ◽  
Tubular Structures ◽  
Type Iii ◽  
Type Iv ◽  
Infected Chimpanzee

Experimental infection of chimpanzees with non-A, non-B hepatitis (NANB) or with delta agent hepatitis results in the appearance of characteristic cytoplasmic alterations in the hepatocytes. These alterations include spongelike inclusions (Type I), attached convoluted membranes (Type II), tubular structures (Type III), and microtubular aggregates (Type IV) (Fig. 1). Type I, II and III structures are, by association, believed to be derived from endoplasmic reticulum and may be morphogenetically related. Type IV structures are generally observed free in the cytoplasm but sometimes in the vicinity of type III structures. It is not known whether these structures are somehow involved in the replication and/or assembly of the putative NANB virus or whether they are simply nonspecific responses to cellular injury. When treated with uranyl acetate, type I, II and III structures stain intensely as if they might contain nucleic acids. If these structures do correspond to intermediates in the replication of a virus, one might expect them to contain DNA or RNA and the present study was undertaken to explore this possibility.

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