Optimal control of a diffusive eco-epidemiological predator–prey model

2020 ◽  
Vol 13 (07) ◽  
pp. 2050065
Author(s):  
Xuebing Zhang ◽  
Guanglan Wang ◽  
Honglan Zhu

In this study, we investigate the optimal control problem for a diffusion eco-epidemiological predator–prey model. We applied two controllers to this model. One is the separation control, which separates the uninfected prey from the infected prey population, and the other is used as a treatment control to decrease the mortality caused by the disease. Then, we propose an optimal problem to minimize the infected prey population at the final time and the cost cause by the controls. To do this, by the operator semigroup theory we prove the existence of the solution to the controlled system. Furthermore, we prove the existence of the optimal controls and obtain the first-order necessary optimality condition for the optimal controls. Finally, some numerical simulations are carried out to support the theoretical results.

Symmetry ◽  
2021 ◽  
Vol 13 (5) ◽  
pp. 785
Author(s):  
Hasan S. Panigoro ◽  
Agus Suryanto ◽  
Wuryansari Muharini Kusumawinahyu ◽  
Isnani Darti

In this paper, we consider a fractional-order eco-epidemic model based on the Rosenzweig–MacArthur predator–prey model. The model is derived by assuming that the prey may be infected by a disease. In order to take the memory effect into account, we apply two fractional differential operators, namely the Caputo fractional derivative (operator with power-law kernel) and the Atangana–Baleanu fractional derivative in the Caputo (ABC) sense (operator with Mittag–Leffler kernel). We take the same order of the fractional derivative in all equations for both senses to maintain the symmetry aspect. The existence and uniqueness of solutions of both eco-epidemic models (i.e., in the Caputo sense and in ABC sense) are established. Both models have the same equilibrium points, namely the trivial (origin) equilibrium point, the extinction of infected prey and predator point, the infected prey free point, the predator-free point and the co-existence point. For a model in the Caputo sense, we also show the non-negativity and boundedness of solution, perform the local and global stability analysis and establish the conditions for the existence of Hopf bifurcation. It is found that the trivial equilibrium point is a saddle point while other equilibrium points are conditionally asymptotically stable. The numerical simulations show that the solutions of the model in the Caputo sense strongly agree with analytical results. Furthermore, it is indicated numerically that the model in the ABC sense has quite similar dynamics as the model in the Caputo sense. The essential difference between the two models is the convergence rate to reach the stable equilibrium point. When a Hopf bifurcation occurs, the bifurcation points and the diameter of the limit cycles of both models are different. Moreover, we also observe a bistability phenomenon which disappears via Hopf bifurcation.


1995 ◽  
Vol 32 (01) ◽  
pp. 274-277
Author(s):  
John Coffey

A new stochastic predator-prey model is introduced. The predator population X(t) is described by a linear birth-and-death process with birth rate λ 1 X and death rate μ 1 X. The prey population Y(t) is described by a linear birth-and-death process in which the birth rate is λ 2 Y and the death rate is . It is proven that and iff


2018 ◽  
Vol 23 (9) ◽  
pp. 3817-3836
Author(s):  
Yang Lu ◽  
◽  
Xia Wang ◽  
Shengqiang Liu ◽  
◽  
...  

2020 ◽  
Vol 554 ◽  
pp. 124136 ◽  
Author(s):  
Chengdai Huang ◽  
Heng Liu ◽  
Xiaoping Chen ◽  
Minsong Zhang ◽  
Ling Ding ◽  
...  

2015 ◽  
Vol 08 (02) ◽  
pp. 1550015 ◽  
Author(s):  
Krishna Pada Das ◽  
J. Chattopadhyay

Disease in ecological systems plays an important role. In the present investigation we propose and analyze a predator–prey mathematical model in which both species are affected by infectious disease. The parasite is transmitted directly (by contact) within the prey population and indirectly (by consumption of infected prey) within the predator population. We derive biologically feasible and insightful quantities in terms of ecological as well as epidemiological reproduction numbers that allow us to describe the dynamics of the proposed system. Our observations indicate that predator–prey system is stable without disease but high infection rate drive the predator population toward extinction. We also observe that predation of vulnerable infected prey makes the disease to eradicate into the community composition of the model system. Local stability analysis of the interior equilibrium point near the disease-free equilibrium point is worked out. To study the global dynamics of the system, numerical simulations are performed. Our simulation results show that for higher values of the force of infection in the prey population the predator population goes to extinction. Our numerical analysis reveals that predation rates specially on susceptible prey population and recovery of infective predator play crucial role for preventing the extinction of the susceptible predator and disease propagation.


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