scholarly journals Posttraumatic Cholesteatoma Complicated by a Facial Paralysis: A Case Report

2012 ◽  
Vol 2012 ◽  
pp. 1-3
Author(s):  
M. Chihani ◽  
A. Aljalil ◽  
M. Touati ◽  
B. Bouaity ◽  
H. Ammar

The posttraumatic cholesteatoma is a rare complication of different types of the temporal bone damage. Its diagnosis is often done after several years of evolution, sometimes even at the stage of complications. A case of posttraumatic cholesteatoma is presented that was revealed by a facial nerve paralysis 23 years after a crash of the external auditory canal underlining the importance of the otoscopic and radiological regular monitoring of the patients with a traumatism of the temporal bone.

Open Medicine ◽  
2014 ◽  
Vol 9 (2) ◽  
pp. 226-230
Author(s):  
Ljiljana Vlaški ◽  
Nada Vučković ◽  
Danijela Dragičević ◽  
Vladimir Kljajić ◽  
Slavica Seničar

Abstract


2008 ◽  
Vol 139 (2_suppl) ◽  
pp. P152-P152 ◽  
Author(s):  
Marc Cohen ◽  
Claudia Kirsch ◽  
Abie Mendelsohn ◽  
Akira Ishiyama

Objectives 1) To retrospectively review the pathophysiology and radiologic features of delayed facial palsy after stapedectomy. 2) To discuss the management strategy of this unusual problem. Methods 450 stapedectomies performed at our institution were retrospectively reviewed from 1997 to 2007. A total of 2 patients in this series developed a delayed facial paralysis postoperatively. The clinical presentation, radiographic characteristics on magnetic resonance imaging (MRI), and the management of these patients are presented. Results A total of 2 patients out of 450 stapedectomies within a 10-year period developed the rare complication of delayed facial paralysis (postoperative days 12 and 41, respectively). Both patients developed rapidly progressive complete facial nerve paralysis preceded by periauricular pain and dysguesia. No sign of infection was present in the operated ear. MRI with gadolinium of the internal auditory canal demonstrated gadolinium enhancement of the labyrinthine portion of the facial nerve as seen in Bell's palsy. Both patients were treated with oral corticosteroids and antiviral medications. Subsequent imaging revealed decreased enhancement of the facial nerve with complete resolution of facial paralysis. Conclusions In the rare complication of delayed facial paralysis after stapedectomy, MR imaging is a useful diagnostic tool to dictate the treatment strategy. Delayed facial paralysis following stapedectomy can be treated medically when the MRI scan demonstrates enhancement of the facial nerve in the labyrinthine segment.


1996 ◽  
Vol 110 (1) ◽  
pp. 91-92 ◽  
Author(s):  
Y. T. Pang ◽  
C. H. Raine

AbstractA case of facial nerve paralysis secondary to acute suppurative parotitis is described. This is a rare complication in the absence of malignant processes in the parotid.


2020 ◽  
pp. 000348942096661
Author(s):  
Austin Y. Feng ◽  
Michael C. Jin ◽  
Sandy Wong ◽  
Jon-Paul Pepper ◽  
Robert Jackler ◽  
...  

Objective: We report a case of facial nerve paralysis post-endovascular embolization of a sigmoid sinus dural arterio-venous fistula from initial presentation to current management and discuss the merits of observation versus decompression through a systematic review of relevant literature. Patient: 61 F with right facial palsy Intervention: Following a single intravenous dexamethasone injection with oral steroids over 2 months, patient was observed with no additional treatment other than Botox chemodenervation and facial rehabilitation. Outcome and Results: The patient initially presented with complete right facial palsy (HB 6/6). Post-op CT imaging indicated Onyx (ev3, Irvine, California, USA) particles present at the geniculate segment of the facial nerve. Observation was chosen over surgical intervention. At the most current follow up of 8 months, facial function has improved substantially (HB 2/6). Conclusion: Facial palsy is a serious, though rare, complication of transarterial endovascular embolization. With our case report and literature review, we highlight not only how conservative observation is the recommended treatment, but also that facial nerve recovery should be expected to reach near complete recovery, but not sooner than in 3 months.


2015 ◽  
Vol 30 (2) ◽  
pp. 65-66
Author(s):  
Ian C. Bickle

  This young adult man presented to ENT clinic with a complaint of left facial weakness and persistent left retro-auricular pain. High resolution CT of the mastoids was performed following clinical assessment. In this case, there is extensive sclerotic bony expansion with a ground-glass appearance involving the left zygoma, sphenoid and petrous temporal bone. The bony expansion is centred on the medullary bone and has an abrupt zonal transition (Figure 1).  The bone involvement encompasses almost complete bony stenosis of the left external auditory meatus down to 1-2mm with consequential fluid in the external auditory canal and middle ears (Figure 2). The bony expansion involves both the tympanic and mastoid segments of the facial canal which are stenosed.  The ossicular chain remains intact.  The left mastoid air cells are under-pneumatised and completely occupied by fluid. DISCUSSION Fibrous dysplasia (FD) is a benign congenital process that typical manifests itself as a localized defect in osteoblastic differentiation and maturation. Normal bone is replaced with haphazard fibrous tissue and immature woven bone.1 Fibrous dysplasia is predominantly a condition of children and young adults (those less than 30 years of age).  Disease growth usually halts after the third decade of life. FD may be a monostotic or polyostotic in nature and in some cases is part of a syndrome, such as McCune-Albright.2 The zygomatic maxillary complex is the most commonly reported location for fibrous dysplasia.  The temporal bone is a typical site in polyostotic disease, in up to 70%, but less often observed in monoostotic disease.  Disease of the temporal bone most typically results in hearing impairment due to bony stenosis of the external auditory canal. Facial nerve involvement is a less frequent feature, resulting in facial nerve paralysis, due to involvement of the nerve as it exits through the petrous temporal bone.2 The anatomical location of the facial nerve compression is hard to access and treat surgically.3 CT is the imaging investigation of choice giving the most exquisite bony definition.  Typical CT features (as shown in this case) are: A diffuse ground-glass appearance to the affected bone Homogeneously sclerotic bone Well-defined borders between the diseased and unaffected bone (abrupt zone of transition) Bony expansion, with overlying cortical bone intact The CT appearances apply equally to the anatomical site involved, however the combination of imaging appearances can be variable presenting a diagnostic dilemma, which may merit a confirmatory bone biopsy.


2021 ◽  
Vol 82 ◽  
pp. 105916
Author(s):  
Sharifeh Haghjoo ◽  
Sayed Hamid Mousavi ◽  
Yeganeh Farsi ◽  
Ali Ahmad Makarem Nasery ◽  
Fawzia Negin ◽  
...  

2019 ◽  
Vol 5 (1) ◽  
pp. 20180029
Author(s):  
Yaotse Elikplim Nordjoe ◽  
Ouidad Azdad ◽  
Mohamed Lahkim ◽  
Laila Jroundi ◽  
Fatima Zahrae Laamrani

Facial nerve aplasia is an extremely rare condition that is usually syndromic, namely, in Moebius syndrome. The occurrence of isolated agenesis of facial nerve is even rarer, with only few cases reported in the literature. We report a case of congenital facial paralysis due to facial nerve aplasia diagnosed on MRI, while no noticeable abnormality was detected on the temporal bone CT.


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