scholarly journals Successful Laparoscopic Management of Ruptured Tubal Pregnancy with an Ipsilateral Ectopic Pelvic Kidney

2014 ◽  
Vol 2014 ◽  
pp. 1-4 ◽  
Author(s):  
Jimmy Belotte ◽  
Jim Belotte ◽  
Mitchell Alexis ◽  
Awoniyi O. Awonuga ◽  
Tina Jessica Aguin

Objective.To report a case of successful laparoscopic management of a left ruptured tubal pregnancy in the setting of an ipsilateral ectopic pelvic kidney.Method.Case report was prepared at Wayne State University/Detroit Medical Center. The patient is a young woman gravida 2 para 0 in her twenties who presented with severe abdominal pain and vaginal bleeding. She had a plateaued beta HCG and ultrasonographic findings suggestive of ectopic left tubal pregnancy along with an ectopic ipsilateral pelvic kidney. The IRB approval is not needed, as this is a case report. The informed consent could not be obtained, as the patient was not reachable.Result.Multiple intraperitoneal adhesions, left ruptured ampullary ectopic pregnancy and left retroperitoneal pelvic mass consistent with ipsilateral ectopic pelvic kidney.Conclusion.Laparoscopic management of tubal pregnancy can be safely performed in the setting of an ipsilateral ectopic pelvic kidney.

2018 ◽  
Vol 159 (2) ◽  
pp. 403-403

Stachler RJ, Francis DO, Schwartz SR, et al. Clinical practice guideline: hoarseness (dysphonia) (update). Otolaryngol Head Neck Surg. 2018;158:S1-S42. (Original DOI: 10.1177/0194599817751030) Stachler RJ, Francis DO, Schwartz SR, et al. Clinical practice guideline: hoarseness (dysphonia) (update) executive summary. Otolaryngol Head Neck Surg. 2018;158:409-426. (Original DOI: 10.1177/0194599817751031) Krouse HJ, Reavis CW, Stachler RJ, Francis DO, O’Connor S. Plain language summary: hoarseness (dysphonia). Otolaryngol Head Neck Surg. 2018;158:427-431. (Original DOI: 10.1177/0194599817751137) In the March 2018 clinical practice guideline and associated articles David O. Francis and Seth R. Schwartz’s affiliations were listed incorrectly. Dr Francis should have been affiliated with University of Wisconsin, Madison, Wisconsin, USA. Dr Schwartz should have been affiliated with Virginia Mason Medical Center, Seattle, Washington, USA. Additionally, in the Plain Language Summary the location of Wayne State University was listed as Allen Park, Michigan; it should have been Detroit, Michigan. These have been updated in the online issues.


Author(s):  
Ulun Uluğ ◽  
Ali İhsan Gönenç ◽  
Özlem Dülger ◽  
Latif Küpelioğlu ◽  
Güray Kılıç

<span><span>Leiomyoma is one of the rarest solid tumours of the ovary. W</span></span>e report a case of a 35 years old woman suffering from irregular menstruel periods and solid unilateral ovarian mass managed by laparoscopic removal. Medical history of the patient is remarkable because of coexisting <span>pelvic kidney and a history of infertility necessiating IVF treatment including gonadotropin stimulation</span>


2005 ◽  
Vol 2 (4) ◽  
pp. 319-321 ◽  
Author(s):  
Suleyman Engin Akhan ◽  
Ilknur Çitil ◽  
Samet Topuz ◽  
Cem Iyibozkurt

Blood ◽  
2011 ◽  
Vol 118 (21) ◽  
pp. 2132-2132
Author(s):  
Abay Taddesse ◽  
Indryas Woldie ◽  
Panchali Khana ◽  
Paul S. Swerdlow ◽  
Jerry Chu ◽  
...  

Abstract Abstract 2132 Studies have provided conflicting evidence on the etiology of hypogonadism in patients with sickle cell disease. A cross-sectional study was conducted on adult men with SCD to determine whether hypogonadism in these patients is due to primary testicular failure or secondary pituitary / hypothalamic dysfunction and to look for association between hypogonadism and serum ferritin levels. The study protocol was approved by Wayne State University / Detroit Medical Center Human investigation committee. A total of 19 male patients with SCD 18 years or older following at the benign hematology clinic of Detroit Medical Center between Sep 21 to Dec 21; 2011 consented to participate in the study. Blood sample was drawn between 9AM – 11 AM and sent to the laboratory for hormonal assay. A previous pilot study (completed few months earlier) of 15 adult male patients with SCD aged 18years or older at the same site was also included in the analysis. The pilot study was approved by Wayne State University / Detroit Medical Center Human investigation committee. Serum samples from 15 patients with sickle cell disease who had blood work during their routine clinic visit at the benign hematology clinic was assessed for testosterone, FSH and LH levels. Thus a total of 34 male patients with SCD were included in the final analysis. Hormone measurements were done for total testosterone (using immunoassay), FSH and LH using two site sandwich immunoassay by chemilumino method (Siemens, Centaur XP). Additional information on body mass index (BMI), hemoglobin, total bilirubin, ferritin and creatinine levels was collected from the Electronic Medical Record (EMR). Hypogonadism was defined as serum total testosterone level of ≤250 ng/dL. Hypogonadal patients with high FSH and LH were considered as having primary/peripheral hypogonadism where as those with low FSH and LH or inappropriately normal (normal FSH and LH despite low serum testosterone) were considered to have secondary/central hypogonadism. FSH level between 1.5–12.4mIU/ml and LH level of 2–14mIU/ml was considered as normal for male adults. Stata 11.1 was used for statistical analysis. Kruskal-Wallis tests were used to assess the statistical significance of differences in hormone and ferritin levels; no adjustment was made for multiple comparisons. Almost all patients in the study have homozygous SCD-SS genotype except two patients with SCD-SC and one patient with SCD-Sβ+ thalassemia. Additional characteristics of the study population are shown in table 1. Eight of 34 men with SCD (23.6 %) were found to have hypogonadism. Hypogonadal patients were found to have significantly low LH (p=0.006) and FSH (p= 0.01) levels indicating central etiology (Figure 1 and Figure 2). There was no significant difference between hypogonadal and normogonadal with respect to ferritin level (p=0.71) (Figure 3). Our study provides evidence for a central etiology to hypogonadism in male patients with SCD. In this small study ferritin level was not significantly related to hypogonadism. Disclosures: No relevant conflicts of interest to declare.


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