scholarly journals Fundus Findings in Wernicke Encephalopathy

2017 ◽  
Vol 8 (2) ◽  
pp. 406-409 ◽  
Author(s):  
Tal Serlin ◽  
Elad Moisseiev
Keyword(s):  
Vitamin B1 ◽  
Chronic Alcoholism ◽  
Visual Disturbance ◽  
Retinal Hemorrhage ◽  
Wernicke Encephalopathy ◽  
Optic Disc Edema ◽  
Vitamin B1 Deficiency ◽  
Postural Imbalance ◽  
Poor Nutrition ◽  
Thiamine Treatment

Wernicke encephalopathy (WE) is an acute neuropsychiatric syndrome resulting from thiamine (vitamin B1) deficiency, classically characterized by the triad of ophthalmoplegia, confusion, and ataxia. While commonly associated with chronic alcoholism, WE may also occur in the setting of poor nutrition or absorption. We present a 37-year-old woman who underwent laparoscopic sleeve gastrectomy and presented with visual disturbance with bilateral horizontal nystagmus, confusion, and postural imbalance. Fundus examination revealed bilateral optic disc edema with a retinal hemorrhage in the left eye. Metabolic workup demonstrated thiamine deficiency. Her symptoms resolved after thiamine treatment. This case raises the awareness of the possibility of posterior segment findings in WE, which are underreported in WE.

scholarly journals Wernicke Encephalopathy as a Complication of Hyperemesis Gravidarum: Case Report

2020 ◽  
Vol 42 (10) ◽  
pp. 672-675
Author(s):  
Ênio Luis Damaso ◽  
Estella Thaisa Sontag dos Reis ◽  
Felipe Alves de Jesus ◽  
Alessandra Cristina Marcolin ◽  
Ricardo de Carvalho Cavalli ◽  
...  
Keyword(s):  
Hyperemesis Gravidarum ◽  
Clinical Status ◽  
Vitamin B1 ◽  
Anterograde Amnesia ◽  
Pregnant Patient ◽  
Chronic Alcoholism ◽  
Wernicke Encephalopathy ◽  
Rapid Improvement ◽  
Vitamin B1 Deficiency ◽  
Magnetic Resonance Imaging Mri

AbstractWernicke encephalopathy (WE) is an acute neurological disorder resulting from vitamin B1 deficiency, which is common in chronic alcoholism. We report a rare case of WE due to hyperemesis gravidarum in a 25-year-old pregnant patient at 13 weeks and 5 days of gestation. Initially, the disease manifested as weakness, mental confusion, anterograde amnesia, and visual and auditory hallucinations. The diagnosis was established after the detection of suggestive findings of WE in the thalamus by magnetic resonance imaging (MRI) and a rapid improvement in the patient's clinical status subsequent to treatment with thiamine. Hyperemesis is a rare cause of WE, which makes the reported case important in the literature and reinforces the need for attention in clinical practice to rare but important complications of this common condition (hyperemesis gravidarum).

Wernicke’s encephalopathy

2017 ◽  
Vol 10 (11) ◽  
pp. 660-666
Author(s):  
Sue May Liow
Keyword(s):  
Sulfuric Acid ◽  
Late Complication ◽  
Vitamin B1 ◽  
Memory Loss ◽  
Altered Mental Status ◽  
Chronic Alcoholism ◽  
Wernicke’S Encephalopathy ◽  
Wernicke's Encephalopathy ◽  
Common Presentation ◽  
Vitamin B1 Deficiency

Wernicke’s encephalopathy occurs as a result of thiamine (vitamin B1) deficiency. It was originally described by German neurologist Karl Wernicke in 1881 who observed the three cardinal features of Wernicke’s in two men with chronic alcoholism and a young woman who had ingested sulfuric acid and suffered prolonged episodes of vomiting. The three cardinal features are ophthalmoplegia, ataxia and confusion. It is largely undiagnosed, due to its vague presentation. Patients often present with confusion or altered mental status; a common presentation in those who might be intoxicated. The classic triad of symptoms described in textbooks is rarely seen. If not promptly or adequately treated Korsakoff’s syndrome, a late complication can develop, resulting in memory loss, confabulation and, ultimately, death.

scholarly journals CONFABULATIONS IN A CHRONIC ALCOHOLIC- A SUBTLE PRESENTATION OF KORSAKOFF SYNDROME

2019 ◽  
Vol 7 (7) ◽  
pp. 58-60
Author(s):  
Yan Leyfman
Keyword(s):  
Case Report ◽  
Alcohol Abuse ◽  
Retrograde Amnesia ◽  
Vitamin B1 ◽  
Wernicke Encephalopathy ◽  
Chronic Alcoholic ◽  
Present Case Report ◽  
Personality Changes ◽  
Korsakoff Syndrome ◽  
Vitamin B1 Deficiency

Wernicke Encephalopathy is an acute neuropsychiatric condition, characterized by confusion, nystagmus, ataxia, and ophthalmoplegia, resulting from thiamine (Vitamin B1) deficiency, typically secondary to alcohol abuse. Failure to properly diagnose, which occurs in 80% of the time, can result in gradual progression to irreversible Korsakoff Syndrome, characterized by irreversible personality changes, and anterograde and retrograde amnesia. The present case report seeks to highlight this patient’s chronology to Korsakoff Syndrome and our attempted interventions.

Comparative studies on vitamin B1 deficiency in whole blood of chronically haemodialysed patients: chromatographic, fluorimetric and PCA study

2021 ◽  
pp. 122880
Author(s):  
Paweł B. Rudnicki-Velasquez ◽  
Hanna Storoniak ◽  
Karolina Jagiełło ◽  
Joanna Kreczko–Kurzawa ◽  
Magdalena Jankowska ◽  
...  
Keyword(s):  
Whole Blood ◽  
Comparative Studies ◽  
Vitamin B1 ◽  
Vitamin B1 Deficiency

ADULT SCURVY ASSOCIATED WITH VITAMIN-B1 DEFICIENCY

The Lancet ◽  
1938 ◽  
Vol 231 (5990) ◽  
pp. 1385-1387 ◽  
Author(s):  
J. Bruce Young
Keyword(s):  
Vitamin B1 ◽  
Vitamin B1 Deficiency

scholarly journals Wernicke’s encephalopathy: An uncommon complication from hyperemesis gravidarum

2019 ◽  
Vol 13 (4) ◽  
pp. 198-200
Author(s):  
Roshan Shah ◽  
Isabelle Malhamé ◽  
Mariam Fayek ◽  
Alisa Merolli ◽  
Niharika Mehta
Keyword(s):  
Hyperemesis Gravidarum ◽  
Vitamin B1 ◽  
Wernicke’S Encephalopathy ◽  
Nutritional Deficiencies ◽  
Wernicke's Encephalopathy ◽  
Gait Ataxia ◽  
Severe Nausea ◽  
Neurologic Manifestation ◽  
Vitamin B1 Deficiency ◽  
Uncommon Complication

Hyperemesis gravidarum is a complication of pregnancy associated with severe nausea and vomiting that can lead to fluid-electrolyte imbalances and nutritional deficiencies. Wernicke’s encephalopathy is a neurologic manifestation of acute thiamine (vitamin B1) deficiency. We describe a case of hyperemesis gravidarum presenting with gait ataxia and nystagmus which led to a diagnosis of Wernicke’s encephalopathy.

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