scholarly journals Wernicke Encephalopathy as a Complication of Hyperemesis Gravidarum: Case Report

2020 ◽  
Vol 42 (10) ◽  
pp. 672-675
Author(s):  
Ênio Luis Damaso ◽  
Estella Thaisa Sontag dos Reis ◽  
Felipe Alves de Jesus ◽  
Alessandra Cristina Marcolin ◽  
Ricardo de Carvalho Cavalli ◽  
...  
Keyword(s):  
Hyperemesis Gravidarum ◽  
Clinical Status ◽  
Vitamin B1 ◽  
Anterograde Amnesia ◽  
Pregnant Patient ◽  
Chronic Alcoholism ◽  
Wernicke Encephalopathy ◽  
Rapid Improvement ◽  
Vitamin B1 Deficiency ◽  
Magnetic Resonance Imaging Mri

AbstractWernicke encephalopathy (WE) is an acute neurological disorder resulting from vitamin B1 deficiency, which is common in chronic alcoholism. We report a rare case of WE due to hyperemesis gravidarum in a 25-year-old pregnant patient at 13 weeks and 5 days of gestation. Initially, the disease manifested as weakness, mental confusion, anterograde amnesia, and visual and auditory hallucinations. The diagnosis was established after the detection of suggestive findings of WE in the thalamus by magnetic resonance imaging (MRI) and a rapid improvement in the patient's clinical status subsequent to treatment with thiamine. Hyperemesis is a rare cause of WE, which makes the reported case important in the literature and reinforces the need for attention in clinical practice to rare but important complications of this common condition (hyperemesis gravidarum).

scholarly journals Fundus Findings in Wernicke Encephalopathy

2017 ◽  
Vol 8 (2) ◽  
pp. 406-409 ◽  
Author(s):  
Tal Serlin ◽  
Elad Moisseiev
Keyword(s):  
Vitamin B1 ◽  
Chronic Alcoholism ◽  
Visual Disturbance ◽  
Retinal Hemorrhage ◽  
Wernicke Encephalopathy ◽  
Optic Disc Edema ◽  
Vitamin B1 Deficiency ◽  
Postural Imbalance ◽  
Poor Nutrition ◽  
Thiamine Treatment

Wernicke encephalopathy (WE) is an acute neuropsychiatric syndrome resulting from thiamine (vitamin B1) deficiency, classically characterized by the triad of ophthalmoplegia, confusion, and ataxia. While commonly associated with chronic alcoholism, WE may also occur in the setting of poor nutrition or absorption. We present a 37-year-old woman who underwent laparoscopic sleeve gastrectomy and presented with visual disturbance with bilateral horizontal nystagmus, confusion, and postural imbalance. Fundus examination revealed bilateral optic disc edema with a retinal hemorrhage in the left eye. Metabolic workup demonstrated thiamine deficiency. Her symptoms resolved after thiamine treatment. This case raises the awareness of the possibility of posterior segment findings in WE, which are underreported in WE.

scholarly journals Wernicke’s encephalopathy: An uncommon complication from hyperemesis gravidarum

2019 ◽  
Vol 13 (4) ◽  
pp. 198-200
Author(s):  
Roshan Shah ◽  
Isabelle Malhamé ◽  
Mariam Fayek ◽  
Alisa Merolli ◽  
Niharika Mehta
Keyword(s):  
Hyperemesis Gravidarum ◽  
Vitamin B1 ◽  
Wernicke’S Encephalopathy ◽  
Nutritional Deficiencies ◽  
Wernicke's Encephalopathy ◽  
Gait Ataxia ◽  
Severe Nausea ◽  
Neurologic Manifestation ◽  
Vitamin B1 Deficiency ◽  
Uncommon Complication

Hyperemesis gravidarum is a complication of pregnancy associated with severe nausea and vomiting that can lead to fluid-electrolyte imbalances and nutritional deficiencies. Wernicke’s encephalopathy is a neurologic manifestation of acute thiamine (vitamin B1) deficiency. We describe a case of hyperemesis gravidarum presenting with gait ataxia and nystagmus which led to a diagnosis of Wernicke’s encephalopathy.

scholarly journals Isolated Mammillary Bodies Damage—An Atypical Presentation of Wernicke Syndrome

2018 ◽  
Vol 8 (11) ◽  
pp. 104
Author(s):  
Samar Abbas ◽  
Halim Abboud ◽  
Moussa Chalah ◽  
Chadi Sabbagh ◽  
Samar Ayache
Keyword(s):  
Anterograde Amnesia ◽  
Complete Recovery ◽  
Resonance Imaging ◽  
Wernicke Encephalopathy ◽  
Mammillary Bodies ◽  
Fluid Attenuated Inversion Recovery ◽  
Partial Colectomy ◽  
Mri Sequences ◽  
Magnetic Resonance Imaging Mri ◽  
T2 Mri

We report atypical magnetic resonance imaging (MRI) lesions in a case of Wernicke encephalopathy. The patient presented with isolated anterograde amnesia following a partial colectomy complicated by peritonitis. Fluid-attenuated inversion recovery and T2 MRI sequences were normal. However, bilateral contrast enhancement of mammillary bodies was shown on T1 gadolinium-enhanced sequences. Blood tests revealed thiamine deficiency. The diagnosis of Wernicke encephalopathy was made and thiamine supplementation was given, resulting in complete recovery of the memory functions.

Wernicke’s encephalopathy

2017 ◽  
Vol 10 (11) ◽  
pp. 660-666
Author(s):  
Sue May Liow
Keyword(s):  
Sulfuric Acid ◽  
Late Complication ◽  
Vitamin B1 ◽  
Memory Loss ◽  
Altered Mental Status ◽  
Chronic Alcoholism ◽  
Wernicke’S Encephalopathy ◽  
Wernicke's Encephalopathy ◽  
Common Presentation ◽  
Vitamin B1 Deficiency

Wernicke’s encephalopathy occurs as a result of thiamine (vitamin B1) deficiency. It was originally described by German neurologist Karl Wernicke in 1881 who observed the three cardinal features of Wernicke’s in two men with chronic alcoholism and a young woman who had ingested sulfuric acid and suffered prolonged episodes of vomiting. The three cardinal features are ophthalmoplegia, ataxia and confusion. It is largely undiagnosed, due to its vague presentation. Patients often present with confusion or altered mental status; a common presentation in those who might be intoxicated. The classic triad of symptoms described in textbooks is rarely seen. If not promptly or adequately treated Korsakoff’s syndrome, a late complication can develop, resulting in memory loss, confabulation and, ultimately, death.

scholarly journals CONFABULATIONS IN A CHRONIC ALCOHOLIC- A SUBTLE PRESENTATION OF KORSAKOFF SYNDROME

2019 ◽  
Vol 7 (7) ◽  
pp. 58-60
Author(s):  
Yan Leyfman
Keyword(s):  
Case Report ◽  
Alcohol Abuse ◽  
Retrograde Amnesia ◽  
Vitamin B1 ◽  
Wernicke Encephalopathy ◽  
Chronic Alcoholic ◽  
Present Case Report ◽  
Personality Changes ◽  
Korsakoff Syndrome ◽  
Vitamin B1 Deficiency

Wernicke Encephalopathy is an acute neuropsychiatric condition, characterized by confusion, nystagmus, ataxia, and ophthalmoplegia, resulting from thiamine (Vitamin B1) deficiency, typically secondary to alcohol abuse. Failure to properly diagnose, which occurs in 80% of the time, can result in gradual progression to irreversible Korsakoff Syndrome, characterized by irreversible personality changes, and anterograde and retrograde amnesia. The present case report seeks to highlight this patient’s chronology to Korsakoff Syndrome and our attempted interventions.

scholarly journals MR Imaging Findings in Alcoholic and Nonalcoholic Acute Wernicke’s Encephalopathy: A Review

2014 ◽  
Vol 2014 ◽  
pp. 1-12 ◽  
Author(s):  
Gaetana Manzo ◽  
Angela De Gennaro ◽  
Attilio Cozzolino ◽  
Antonietta Serino ◽  
Giacomo Fenza ◽  
...  
Keyword(s):  
Mr Imaging ◽  
Complex Disease ◽  
Hyperemesis Gravidarum ◽  
Vitamin B1 ◽  
Chronic Alcoholism ◽  
Sudden Onset ◽  
Wernicke’S Encephalopathy ◽  
Wernicke's Encephalopathy ◽  
Tectal Plate ◽  
Low Prevalence

Wernicke’s encephalopathy (WE) is a severe neurological syndrome caused by thiamine (vitamin B1) deficiency and clinically characterized by the sudden onset of mental status changes, ocular abnormalities, and ataxia. Apart from chronic alcoholism, the most common cause of WE, a lot of other conditions causing malnutrition and decreasing thiamine absorption such as gastrointestinal surgical procedures and hyperemesis gravidarum must be considered as predisposing factors. Due to its low prevalence and clinical heterogeneity, WE is often misdiagnosed, leading to persistent dysfunctions and, in some cases, to death. Nowadays, MR imaging of the brain, showing T2 and FLAIR hyperintensities in typical (thalami, mammillary bodies, tectal plate, and periaqueductal area) and atypical areas (cerebellum, cranial nerve nuclei, and cerebral cortex), is surely the most important and effective tool in the diagnostic assessment of WE. The aim of this paper is to propose a state of the art of the role of MR imaging in the early diagnosis of this complex disease.

Neglected woman with hyperemesis gravidarum leading to Wernicke encephalopathy

2020 ◽  
Vol 13 (12) ◽  
pp. e238545
Author(s):  
Papa Dasari ◽  
Smitha Priyadarshini
Keyword(s):  
Grey Matter ◽  
Hyperemesis Gravidarum ◽  
Intravenous Fluids ◽  
State Of Health ◽  
Wernicke Encephalopathy ◽  
Periaqueductal Grey ◽  
Mri Brain ◽  
Good State ◽  
Periaqueductal Grey Matter ◽  
History Of

A teenage primigravida at 13 weeks of gestation presented with hyperemesis gravidarum of 45 days and a history of giddiness and inability to walk due to involuntary movements of limbs and eyes since 2 days. She was treated with intravenous fluids, thiamine and antiemetics. MRI brain showed hyperintensities in bilateral dorsomedial thalami, periaqueductal grey matter in T2-weighted and FLAIR images. A diagnosis of Wernicke encephalopathy was made and she was managed in intensive care unit and received injection thiamine as per the guidelines and her weakness and ataxia improved over 3 weeks and she was discharged at 17 weeks of pregnancy in good state of health.

scholarly journals Acute Mesenteric Vein Thrombosis in a Pregnant Patient at 10 Weeks Gestation: A Case Report

Diagnostics ◽  
2021 ◽  
Vol 11 (8) ◽  
pp. 1348
Author(s):  
Ying-Ying Chen ◽  
Sheng-Mao Wu ◽  
Russell Oliver Kosik ◽  
Yi-Chien Hsieh ◽  
Tzu-I Wu ◽  
...  
Keyword(s):  
Acute Abdominal Pain ◽  
Clinical Manifestations ◽  
Pregnant Patient ◽  
Resonance Imaging ◽  
Advanced Imaging ◽  
Mesenteric Vein Thrombosis ◽  
Mesenteric Vein ◽  
Vein Thrombosis ◽  
Management Perspective ◽  
Magnetic Resonance Imaging Mri

Acute abdominal pain during pregnancy is challenging, both from a diagnostic and management perspective. A non-localized, persistent pain out of proportion to physical examination is a sign that advanced imaging may be necessary. Mesenteric venous thrombosis in a pregnant patient is extremely rare, but if diagnosis is delayed, can be potentially fatal to both the mother and the fetus. We present here a pregnant patient in the tenth week of gestation with classic clinical manifestations of mesenteric vein thrombosis and the corresponding findings on magnetic resonance imaging (MRI) and computed tomography (CT).

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