Massive Extra-Abdominal Umbilical Vein Varix: A Case Report

2021 ◽  
pp. 1-4
Author(s):  
Josef Jackson ◽  
Eumenia Castro ◽  
Michael A. Belfort ◽  
Alireza A. Shamsirshaz ◽  
Ahmed A. Nassr ◽  
...  
Keyword(s):  
Case Report ◽  
Umbilical Cord ◽  
Neonatal Period ◽  
Umbilical Vein ◽  
Fetal Distress ◽  
Fetal Anemia ◽  
Pathologic Examination ◽  
Fetal Demise ◽  
Increased Risk ◽  
Umbilical Vein Varix

Umbilical vein varices are rare umbilical cord anomalies that typically occur intra-abdominally. Extra-abdominal umbilical vein varices are exceedingly rare and usually diagnosed postnatally on gross pathologic examination. Umbilical vein varices have been associated with increased risk of fetal anemia, cardiac abnormalities, and intrauterine fetal demise. This case report discusses a patient who presented with a massive extra-abdominal umbilical vein varix, whose infant was ultimately delivered due to fetal distress and died in the neonatal period. This report also discusses associated fetal conditions and guidelines for antenatal testing and surveillance of known umbilical vein varices.

Severe fetal anemia as a consequence of extra‐abdominal umbilical vein varix: A case report and review of the literature

2020 ◽  
Vol 61 (1) ◽  
pp. 4-8
Author(s):  
Shingo Io ◽  
Eiji Kondoh ◽  
Yoshiki Iemura ◽  
Sachiko Minamiguchi ◽  
Yoshitsugu Chigusa ◽  
...  
Keyword(s):  
Case Report ◽  
Umbilical Vein ◽  
Fetal Anemia ◽  
Review Of The Literature ◽  
Umbilical Vein Varix

scholarly journals Spontaneous umbilical cord hematoma causing still birth: a case report in Madagascar

2018 ◽  
Vol 7 (5) ◽  
pp. 2031
Author(s):  
Lyn Z. A. Rabetsimamanga ◽  
Hary F. Rabarikoto ◽  
Eddie B. Rekoronirina ◽  
Hery R. Andrianampanalinarivo
Keyword(s):  
Case Report ◽  
Umbilical Cord ◽  
Fetal Heart ◽  
Fetal Distress ◽  
Fetal Hypoxia ◽  
Female Fetus ◽  
Still Birth ◽  
Heart Monitoring ◽  
Short Time ◽  
Uncommon Complication

Spontaneous umbilical cord hematoma is uncommon complication during delivery. It is responsible for severe fetal distress or death. We report a case of a 28 year-old primigravida Malagasy woman at the 37th weeks of gestation. She was admitted in the delivery room for beat oscillating on the fetal heart monitoring at the beginning of labour. Cesarean section was indicated for acute fetal distress in monitoring at dilation 5cm of the cervix traduced by some decelerations.  But she gives birth to a still born female fetus by vaginal way short time after. A 5,5 cm hematoma was discovered on umbilical cord. This still birth may be due to anoxia during acute compression of the umbilical vessels by the hematoma. So, placental and cord examinations in cases of unexplained fetal hypoxia and stillbirth are very important.

Fetal Vascular Malperfusion Due To Long and Hypercoiled Umbilical Cords Resulting in Recurrent Second Trimester Pregnancy Loss: A Case Series and Literature Review

2020 ◽  
pp. 109352662096206
Author(s):  
Jonathan C Slack ◽  
Theonia K Boyd
Keyword(s):  
Umbilical Cord ◽  
Rare Event ◽  
Case Series ◽  
Second Trimester ◽  
Fetal Demise ◽  
Flow Restriction ◽  
Intrauterine Fetal Demise ◽  
Increased Risk ◽  
Second Trimester Pregnancy ◽  
Umbilical Cords

Background Intrauterine fetal demise due to fetal vascular malperfusion in mid-gestation is a rare occurrence. Abnormally long and hypercoiled umbilical cords are associated with an increased risk of umbilical cord blood flow restriction, which in turn can result in adverse perinatal and maternal outcomes. The factors that regulate umbilical cord development, specifically umbilical cord length and coiling, are poorly understood. Methods Maternal history, along with fetal and placental findings (post-mortem, pathological, and molecular), were reviewed for a series of 3 consecutive pregnancies that ended in second trimester intrauterine fetal demise. Results All 3 umbilical cords were exceptionally long and hypercoiled, and all placentas showed evidence of high-grade fetal vascular malperfusion. At fetopsy, all 3 fetuses were developmentally normal for gestational age and lacked congenital anomalies. Maternal medical history and antenatal testing (including an extensive work-up for maternal hypercoagulability syndromes) were normal and/or noncontributory. Conclusion Although excessively long and hypercoiled cords are generally thought of as sporadic, nongenetic events, rare examples of recurrent intrauterine fetal demise secondary to such exist have been reported. This intrafamilial clustering of a rare event is suggestive that at least a subset of hypercoiled, long umbilical cords may have an underlying genetic etiology.

Fetal intra-abdominal umbilical vein varix: a case report

Ultrasound ◽  
2013 ◽  
Vol 21 (3) ◽  
pp. 152-154
Author(s):  
Chimwemwe Kalumbi ◽  
Richard Crowson ◽  
Fatimah Soydemir ◽  
Tracy Butcher
Keyword(s):  
Case Report ◽  
Umbilical Vein ◽  
Umbilical Vein Varix

A case of extra-abdominal umbilical vein varix with severe fetal anemia

Placenta ◽  
2019 ◽  
Vol 87 ◽  
pp. 58
Author(s):  
Shingo Io ◽  
Yoshitsugu Chigusa ◽  
Haruta Mogami ◽  
Eiji Kondoh ◽  
Masaki Mandai
Keyword(s):  
Umbilical Vein ◽  
Fetal Anemia ◽  
Umbilical Vein Varix

scholarly journals Fetal Intra-Abdominal Umbilical Vein Varix: A Case Report and Literature Review

2015 ◽  
Vol 65 (6) ◽  
pp. 420-422 ◽  
Author(s):  
Loida Pamplona-Bueno ◽  
Pablo Padilla-Iserte ◽  
Blanca Ferri-Folch ◽  
Irene Juárez-Pallarés ◽  
Rosa Gómez-Portero ◽  
...  
Keyword(s):  
Case Report ◽  
Literature Review ◽  
Umbilical Vein ◽  
Umbilical Vein Varix

Leukocytic Infiltration of the Umbilical Cord in Twins

1970 ◽  
Vol 19 (1-2) ◽  
pp. 92-95 ◽  
Author(s):  
M. Thiery ◽  
A. Yo le Sian ◽  
R. Derom ◽  
R. Boelaert
Keyword(s):  
Umbilical Cord ◽  
Umbilical Vein ◽  
Clinical Signs ◽  
Neonatal Infection ◽  
Fetal Distress ◽  
Long Term Study ◽  
Leukocytic Infiltration ◽  
Born Infant ◽  
Fetal Infection ◽  
Prolonged Labor

Leukocytic infiltration of the umbilical cord, particularly of the umbilical vein, is common both after normal and abnormal delivery (Tab. I).The significance and the cause(s) of this phenomenon, however, are still highly controversed subjects (Tab. II). According to some authors (Benirschke, i960; Benirschke and Clifford 1959; Blanc 1961; and Kelsall et al, 1967), cord infiltration may be an indication of intrauterine fetal infection. As a consequence, the terms vasculitis, umbilical phlebitis and funiculitis have been introduced. Other authors (Barter, 1962, and Dominguez et al, 1960), mostly impressed by the discrepancy between the incidence of clinically evident neonatal infection and funiculitis, as well as by the significant association of cord infiltration with prolonged labor and clinical signs of fetal distress, have come to the conclusion that leukocytic infiltration of the umbilical cord must predominantly be the effect of fetal hypoxia. Dominguez et al (i960) go even further and postulate “that the examination of the umbilical cord for leukocytic infiltration affords a means for a long term study of children to determine the effect of intrauterine hypoxia on their ultimate growth and development”.One way to check the validity of the latter hypothesis, is to investigate the umbilical cords of twins. Since our group has proven that, compared to the first-born twin, the second-born infant is as a rule significantly more hypoxic at birth (Derom and Thiery, 1969), one may expect that intragroup comparison of the incidence of leukocytic infiltration of twin cords should at least permit to disprove the validity of Dominguez assumptions.

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