Lymphoma Involving the Esophagus

1994 ◽  
Vol 103 (11) ◽  
pp. 843-848 ◽  
Author(s):  
Laura J. Orvidas ◽  
Thomas V. McCaffrey ◽  
Jean E. Lewis ◽  
Paul J. Kurtin ◽  
Thomas M. Habermann

Fewer than 1% of all lymphomas involve the esophagus; however, lymphoma of the esophagus represents an important cause of dysphagia. This study reviewed all cases of biopsy-proven lymphoma involving the esophagus presenting at our institution between 1945 and 1992. Twenty-seven cases were identified. Three were primary esophageal lymphomas. Eleven percent of the cases represented Hodgkin's disease. Eighty-nine percent were non-Hodgkin's lymphoma. Eighty-nine percent of the patients experienced dysphagia. Eleven lymphomas (41%) were located at the gastroesophageal junction, while the other 17 were in the esophagus proper. Seven of these cases occurred at relapse. Three had mediastinal adenopathy with secondary esophageal involvement Morbidity included tracheoesophageal fistula in 22%, and surgical repair was performed in half of these cases. Vocal cord paralysis occurred in 22%, with minimal sequelae. Esophageal stricture was present in 30%, usually necessitating dilation. The presentation, diagnosis, and management of this problem are multidisciplinary.

PEDIATRICS ◽  
1991 ◽  
Vol 87 (1) ◽  
pp. 39-43
Author(s):  
Frank C. Chaten ◽  
Steven E. Lucking ◽  
Edwin S. Young ◽  
John J. Mickell

During an 18-month period in a pediatric intensive care unit, nine patients with vocal cord paralysis were identified using flexible bronchoscopy. When tracheally extubated, each child was found to have stridor. The children ranged in age from 17 days to 5½ years. Two patients had unilateral paralysis, but neither required tracheostomy. Seven patients displayed bilateral abductor vocal cord paralysis. Of these, six patients required tracheostomy. Surgical injury to the recurrent laryngeal nerve was the probable cause in two patients. The other seven patients had neurologic disorders with documented or suspected increases of intracranial pressure. Four of the seven patients with bilateral abductor vocal cord paralysis regained cord mobility within 4 months. Both children with unilateral cord paralysis have no stridor and vocalize well 1 year later. Cord paralysis in the setting of intracranial hypertension probably results from compression or ischemia of the vagus nerve before it exits the skull. Early visualization of the larynx should be done in patients who become stridulous when extubated, especially those with prior thoracic procedures or with neurologic disorders associated with intracranial hypertension.


Author(s):  
Thomas Kovesi ◽  
Federica Porcaro ◽  
Francesca Petreschi ◽  
Marilena Trozzi ◽  
Sergio Bottero ◽  
...  

Author(s):  
Federica Porcaro ◽  
Thomas Kovesi ◽  
Francesca Petreschi ◽  
Marilena Trozzi ◽  
Sergio Bottero ◽  
...  

Endoscopy ◽  
2008 ◽  
Vol 40 (S 02) ◽  
pp. E150-E150 ◽  
Author(s):  
Z. Gellad ◽  
D. Hampton ◽  
C. Tebbit ◽  
L. Puscas ◽  
D. Pavey

2003 ◽  
Vol 112 (9) ◽  
pp. 764-767 ◽  
Author(s):  
Robert G. Berkowitz

Bilateral adductor vocal cord paralysis (BAdP), presenting with features of laryngeal incompetence, is a rare form of congenital bilateral vocal cord paralysis, and only 2 small series of BAdP have previously been published. Three cases are reported here. The BAdP occurred as an isolated abnormality in 1 child, and was associated with a recognizable syndrome (Robinow's syndrome and 22q deletion) in the other 2 children. Gastrostomy tube feeding was required in 2 children, who both remain gastrostomy tube–dependent at 26 months and 10 years 9 months of age. The child with Robinow's syndrome received parenteral nutrition until 2 months, but was then able to feed orally after partial improvement in vocal cord function. The global impairment in vocal cord constrictor function observed in these 3 children is consistent with the site of lesion's being at the level of the laryngeal constrictor motoneurons in BAdP.


2004 ◽  
Vol 131 (2) ◽  
pp. P70-P70
Author(s):  
Christopher J Hartnick ◽  
Dana M Thompson ◽  
J Scott McMurray ◽  
Roger C Nuss ◽  
Benjamin E Hartley ◽  
...  

Author(s):  
Valli Rajasekaran ◽  
Srividhya G.

<p class="abstract"><strong>Background:</strong> <span lang="EN-IN">In the recent years there is resurgence of tuberculosis and the presentation of the same has changed. Vocal cord paralysis can be due to involvement of vagus anywhere along its course. The lesions in the larynx can be the earliest presentation of tuberculosis. </span></p><p class="abstract"><strong>Methods:</strong> <span lang="EN-IN">A study has been done on all patients with tuberculosis with laryngeal lesions due to tuberculosis for a period of 6 months. These lesions were followed up and the resolution of these lesions and symptoms were documented.  </span></p><p class="abstract"><strong>Results:</strong> <span lang="EN-IN">Vocal cord paralysis was the most common manifestation (66.6%). Tuberculosis of the larynx can be secondary to pulmonary tuberculosis or a primary manifestation. Vocal cord paralysis on the left side was due to mediastinal lesions and on the right side was due to apical fibrosis. </span></p><p class="abstract"><strong>Conclusions:</strong> <span lang="EN-IN">Vocal cord lesions due to active tuberculosis either nodal or parenchymal involvement usually recover completely with treatment. On the other hand, the lesions due to inactive lesions are likely to be permanent.</span></p>


2002 ◽  
Vol 53 (1) ◽  
pp. 1-5
Author(s):  
Etsuyo Tamura ◽  
Satoshi Kitahara ◽  
Naoyuki Kohno ◽  
Masami Ogura

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