Traumatic Pseudoaneurysm of the Left Ventricle

1996 ◽  
Vol 4 (4) ◽  
pp. 236-237
Author(s):  
Rajendra Kumar Premchand ◽  
Rakesh Arora ◽  
Rakesh Sudhan ◽  
Vasantha Kumar Allam ◽  
Padmanabhan Tirumalai Nallan Chakravarthi ◽  
...  
Keyword(s):  
Left Ventricle ◽  
Blunt Chest Trauma ◽  
Chest Trauma ◽  
Surgical Repair ◽  
False Aneurysm ◽  
Coronary Artery Occlusion ◽  
Artery Occlusion ◽  
Myocardial Contusion ◽  
Traumatic Pseudoaneurysm

A pseudoaneurysm of the left ventricle developed in an 11-year-old boy following a trivial blunt trauma to the chest wall. This was detected by echocardiography 3 months after the injury. An echocardiogram performed 2 weeks after the injury demonstrated intact chambers. The false aneurysm resulted from myocardial contusion with subsequent gradual rupture of the left ventricle without coronary artery occlusion. The patient underwent a successful surgical repair. This case demonstrates the need for careful follow-up of all patients sustaining blunt chest trauma.

Circumflex Coronary Artery Occlusion After Blunt Chest Trauma

Heart Disease ◽  
2003 ◽  
Vol 5 (3) ◽  
pp. 184-186 ◽  
Author(s):  
Nauman Naseer ◽  
Wilbert S. Aronow ◽  
John A. McClung ◽  
Shirin Sanal ◽  
Stephen J. Peterson ◽  
...  
Keyword(s):  
Coronary Artery ◽  
Blunt Chest Trauma ◽  
Chest Trauma ◽  
Coronary Artery Occlusion ◽  
Artery Occlusion ◽  
Circumflex Coronary Artery

scholarly journals Coronary artery occlusion following blunt chest trauma: a case report and review of the literature

CJEM ◽  
2005 ◽  
Vol 7 (02) ◽  
pp. 118-123 ◽  
Author(s):  
Gregory T. Guldner ◽  
Thomas D. Schilling
Keyword(s):  
Myocardial Infarction ◽  
Coronary Artery ◽  
Blunt Chest Trauma ◽  
Chest Trauma ◽  
Coronary Artery Occlusion ◽  
Artery Occlusion ◽  
Review Of The Literature ◽  
Laboratory Findings ◽  
Complete Occlusion ◽  
Proximal Right Coronary Artery

ABSTRACTBlunt chest trauma causing coronary artery occlusion and myocardial infarction is a rare but potentially fatal condition. We present the case of a healthy 29-year-old man who developed a myocardial infarction due to complete occlusion of the proximal right coronary artery following blunt chest trauma. A review of the literature found 63 cases of previously healthy patients under 40 years of age who developed coronary artery occlusion following blunt chest trauma; diagnosis in all cases had been proven by angiography or during autopsy. The presentation, results of electrocardiography and echocardiography and laboratory findings of these patients are described.

scholarly journals Acute coronary artery occlusion secondary to blunt chest trauma

1982 ◽  
Vol 83 (1) ◽  
pp. 122-125 ◽  
Author(s):  
Roque Pifarré ◽  
John Grieco ◽  
Abel Garibaldi ◽  
Henry J. Sullivan ◽  
Alvaro Montoya ◽  
...  
Keyword(s):  
Coronary Artery ◽  
Blunt Chest Trauma ◽  
Chest Trauma ◽  
Coronary Artery Occlusion ◽  
Artery Occlusion

scholarly journals Clinicoradiological diagnosis: Cough-induced transdiaphragmatic intercostal herniation

2020 ◽  
Vol 6 (2) ◽  
pp. 20190061
Author(s):  
Prema Mohandas ◽  
Ahmed O.A. Krim ◽  
Jerry Glenn
Keyword(s):  
Large Bowel ◽  
Blunt Chest Trauma ◽  
Chest Trauma ◽  
Surgical Repair ◽  
Penetrating Trauma ◽  
Rib Fractures ◽  
Intercostal Muscles

Transdiaphragmatic intercostal herniation can occur following blunt or penetrating trauma and is usually associated with rib fractures. It is uncommon and only sporadically reported in literature. We report a case of cough-induced intercostal herniation containing large bowel, on a background of sustaining a blunt chest trauma 25 years prior to presentation. The patient was treated by reducing the hernia followed by surgical repair of the diaphragm and intercostal muscles defect. He was discharged without further complications and remained well at follow-up.

scholarly journals Left Anterior Descending Coronary Artery Occlusion Secondary to Blunt Chest Trauma

Circulation ◽  
2009 ◽  
Vol 119 (14) ◽  
pp. 1975-1976 ◽  
Author(s):  
Sachin S. Goel ◽  
James E. Harvey ◽  
Marc Penn ◽  
Venu Menon
Keyword(s):  
Coronary Artery ◽  
Blunt Chest Trauma ◽  
Chest Trauma ◽  
Coronary Artery Occlusion ◽  
Artery Occlusion

Emergency PTCA for coronary artery occlusion after blunt chest trauma

1990 ◽  
Vol 119 (6) ◽  
pp. 1408-1410 ◽  
Author(s):  
Martin Sigmund ◽  
Silvia Nase-Hüppmeier ◽  
Rainer Uebis ◽  
Peter Hanrath
Keyword(s):  
Coronary Artery ◽  
Blunt Chest Trauma ◽  
Chest Trauma ◽  
Coronary Artery Occlusion ◽  
Artery Occlusion

Abstract 12: EMMPRIN-Targeted Magnetic Nanoparticles for in vivo Visualization and Regression of Acute Myocardial Infarction in a Model of Acute Coronary Artery Occlusion

2015 ◽  
Vol 35 (suppl_1) ◽  
Author(s):  
Irene Cuadrado ◽  
Maria Jose Garcia Miguel ◽  
Irene Herruzo ◽  
Mari Carmen Turpin ◽  
Ana Martin ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Extracellular Matrix ◽  
Coronary Artery ◽  
Left Ventricle ◽  
Infarct Size ◽  
Coronary Artery Occlusion ◽  
Artery Occlusion ◽  
Gadolinium Enhancement

Extracellular matrix metalloproteinase inducer EMMPRIN, is highly expressed in patients with acute myocardial infarction (AMI), and induces activation of several matrix metalloproteinases (MMPs), including MMP-9 and MMP-13. To prevent Extracellular matrix degradation and cardiac cell death we targeted EMMPRIN with paramagnetic/fluorescent micellar nanoparticles with an EMMPRIN binding peptide AP9 conjugated (NAP9), or an AP9 scramble peptide as a negative control (NAPSC). NAP9 binds to endogenous EMMPRIN as detected by confocal microscopy of cardiac myocytes and macrophages incubated with NAP and NAPSC in vitro, and in vivo in mouse hearts subjected to left anterior descending coronary artery occlusion (IV injection 50mγ/Kg NAP9 or NAP9SC). Administration of NAP9 at the same time or 1 hour after AMI reduced infarct size over a 20% respect to untreated and NAPSC injected mice, recovered left ventricle ejection fraction (LVEF) similar to healthy controls, and reduced EMMPRIN downstream MMP9 expression. In magnetic resonance scans of mouse hearts 2 days after AMI and injected with NAP9, we detected a significant gadolinium enhancement in the left ventricle respect to non-injected mice and to mice injected with NAPSC. Late gadolinium enhancement assays exhibited NAP9-mediated left ventricle signal enhancement as early as 30 minutes after nanoprobe injection, in which a close correlation between the MRI signal enhancement and left ventricle infarct size was detected. Taken together, these results point EMMPRIN targeted nanoprobes as a new tool for the treatment of AMI.

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