Patterns in the management of superficial vein thrombosis

2016 ◽  
Vol 32 (3) ◽  
pp. 207-213 ◽  
Author(s):  
Christos Karathanos ◽  
Konstantinos Spanos ◽  
Vasileios Lachanas ◽  
Athanasios Athanasoulas ◽  
Athanasios D Giannoukas
Keyword(s):  
Duplex Ultrasound ◽  
Superficial Vein ◽  
Electronic Survey ◽  
Vein Thrombosis ◽  
Mailing Lists ◽  
Superficial Vein Thrombosis ◽  
Venous Diseases ◽  
Elastic Stockings ◽  
Inflammatory Drugs ◽  
Current Guidelines

Objective To highlight current practice patterns in management of superficial vein thrombosis. Methods An electronic survey was conducted using the mailing lists of the Mediterranean League of Angiology and Vascular Surgery and European Venous Forum regarding superficial vein thrombosis diagnosis, investigation, and treatment. Results The response rate was 41% (175/430) and the majority of the participants were vascular surgeons practicing in a hospital. More experienced physicians considered superficial vein thrombosis as a medical issue of moderate seriousness and performed duplex ultrasound for confirmation of diagnosis. Elastic stockings were recommended by 87% of the physicians, while 57% prescribed nonsteroidal anti-inflammatory drugs. Eighty six percent advised anticoagulation, although a large disparity was shown regarding regime, dose, and duration. Thrombophilia test was regularly suggested by 19% of the physicians. Ligation of the saphenofemoral junction was the treatment of choice by those who suggested intervention in the acute phase of superficial vein thrombosis. Conclusions A great disparity exists in the management of superficial vein thrombosis. Current guidelines have not been adopted by physicians; more focused training is needed for those involved in the management of venous diseases.

scholarly journals Controversies in venous thromboembolism: to treat or not to treat superficial vein thrombosis

Hematology ◽  
2017 ◽  
Vol 2017 (1) ◽  
pp. 223-230 ◽  
Author(s):  
Jan Beyer-Westendorf
Keyword(s):  
Clinical Decision ◽  
Superficial Vein ◽  
Compression Stockings ◽  
Surgical Interventions ◽  
Vein Thrombosis ◽  
Superficial Vein Thrombosis ◽  
Inflammatory Drugs ◽  
Deep Vein ◽  
Current Guidelines ◽  
Insight Into

Abstract The management of superficial vein thrombosis (SVT) is poorly defined and remains controversial overall. SVT has long been considered a benign, self-limited disease, but recent studies show that SVT carries a nonnegligible risk for recurrence, deep vein thrombosis, or pulmonary embolism. Current guidelines recommend the use of low-molecular-weight heparin or fondaparinux, but results of several surveys indicate that the majority of patients with SVT receive nonanticoagulant therapy only, which includes compression stockings or bandages, nonsteroidal anti-inflammatory drugs, topical application of heparin gel, or surgical interventions. However, several recent observational and interventional studies provide better insight into the optimal treatment of patients with SVT who are at different risks for thromboembolic complications. This educational review summarizes the available evidence and aims to provide practical guidance based on a clinical decision pathway.

scholarly journals Duplex Ultrasound Screening for Deep and Superficial Vein Thrombosis in COVID‐19 Patients

2021 ◽  
Author(s):  
Yale Tung‐Chen ◽  
Ruth Calderón ◽  
Cristina Marcelo ◽  
Francesco Deodati ◽  
María Mateos ◽  
...  
Keyword(s):  
Duplex Ultrasound ◽  
Ultrasound Screening ◽  
Superficial Vein ◽  
Vein Thrombosis ◽  
Superficial Vein Thrombosis

Treating Superficial Venous Thrombophlebitis

2008 ◽  
Vol 6 (8) ◽  
pp. 760-765 ◽  
Author(s):  
Jason T. Lee ◽  
Maziyar A. Kalani
Keyword(s):  
Clinical Presentation ◽  
Low Molecular Weight ◽  
Superficial Vein ◽  
Low Molecular Weight Heparins ◽  
Surgical Interventions ◽  
Superficial Thrombophlebitis ◽  
Compression Bandages ◽  
Elastic Stockings ◽  
Inflammatory Drugs ◽  
Valvular Insufficiency

Superficial venous thrombophlebitis (SVT) is characterized as a localized inflammatory condition of the venous vessels underlying the skin. It arises from thrombosis of a superficial vein, and clinical presentation usually involves pain, erythema, and tenderness at the sites of inflammation. Although the condition is usually self-limited and not serious or fatal, symptomatic superficial thrombophlebitis can be debilitating, limit movement and certain capabilities, or progress to involve the deep venous system and cause pulmonary embolism. SVT is typically associated with venous valvular insufficiency, pregnancy, infection, and prothrombotic conditions, including malignancy. Currently, medical therapies comprising bedrest, elastic stockings, compression bandages, nonsteroidal anti-inflammatory drugs, and low molecular weight heparins are used to reduce the extension of inflammation and recurrence of thrombotic events in patients experiencing SVT. In patients refractory to conservative measures, surgical interventions such as phlebectomy, sclerotherapy, saphenous junction ligation, or saphenous vein stripping are potential treatments.

scholarly journals Factors predictive of venous thrombotic complications in patients with isolated superficial vein thrombosis

2003 ◽  
Vol 38 (5) ◽  
pp. 944-949 ◽  
Author(s):  
Sara Quenet ◽  
Silvy Laporte ◽  
Hervé Décousus ◽  
Alain Leizorovicz ◽  
Magali Epinat ◽  
...  
Keyword(s):  
Superficial Vein ◽  
Vein Thrombosis ◽  
Superficial Vein Thrombosis ◽  
Thrombotic Complications

Evaluation of direct oral anticoagulants in superficial-vein thrombosis – Authors' reply

2017 ◽  
Vol 4 (6) ◽  
pp. e254-e255
Author(s):  
Jan Beyer-Westendorf ◽  
Sebastian M Schellong ◽  
Horst Gerlach ◽  
Eberhard Rabe ◽  
Jeffrey I Weitz ◽  
...  
Keyword(s):  
Oral Anticoagulants ◽  
Direct Oral Anticoagulants ◽  
Superficial Vein ◽  
Vein Thrombosis ◽  
Superficial Vein Thrombosis

scholarly journals Different Risks of Thrombosis in Four Coagulation Defects Associated With Inherited Thrombophilia: A Study of 150 Families

Blood ◽  
1998 ◽  
Vol 92 (7) ◽  
pp. 2353-2358 ◽  
Author(s):  
Ida Martinelli ◽  
Pier Mannuccio Mannucci ◽  
Valerio De Stefano ◽  
Emanuela Taioli ◽  
Valentina Rossi ◽  
...  
Keyword(s):  
Protein C ◽  
Factor V Leiden ◽  
Protein S ◽  
Factor V ◽  
Superficial Vein ◽  
Protein S Deficiency ◽  
Vein Thrombosis ◽  
S Deficiency ◽  
Superficial Vein Thrombosis ◽  
Coagulation Defects

AbstractDeficiency of the naturally occurring anticoagulant proteins, such as antithrombin, protein C and protein S, and activated protein C resistance due to the factor V Leiden gene mutation is associated with inherited thrombophilia. So far, no direct comparison of the thrombotic risk associated with these genetic defects is available. In this study, we wish to compare the lifetime probability of developing thrombosis, the type of thrombotic symptoms, and the role of circumstantial triggering factors in 723 first- and second-degree relatives of 150 index patients with different thrombophilic defects. We found higher risks for thrombosis for subjects with antithrombin (risk ratio 8.1, 95% confidence interval [CI], 3.4 to 19.6), protein C (7.3, 95% CI, 2.9 to 18.4) or protein S deficiency (8.5, 95% CI, 3.5 to 20.8), and factor V Leiden (2.2, 95% CI, 1.1 to 4.7) than for individuals with normal coagulation. The risk of thrombosis for subjects with factor V Leiden was lower than that for those with all three other coagulation defects (0.3, 95% CI, 0.1 to 1.6), even when arterial and superficial vein thromboses were excluded and the analysis was restricted to deep vein thrombosis (0.3, 95% CI, 0.2 to 0.5). No association between coagulation defects and arterial thrombosis was found. The most frequent venous thrombotic manifestation was deep vein thrombosis with or without pulmonary embolism (90% in antithrombin, 88% in protein C, 100% in protein S deficiency, and 57% in factor V Leiden), but a relatively mild manifestation such as superficial vein thrombosis was common in factor V Leiden (43%). There was a predisposing factor at the time of venous thromboembolism in approximately 50% of cases for each of the four defects. In conclusion, factor V Leiden is associated with a relatively small risk of thrombosis, lower than that for antithrombin, protein C, or protein S deficiency. In addition, individuals with factor V Leiden develop less severe thrombotic manifestations, such as superficial vein thrombosis.

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