Carinal Resection and Reconstruction for an Obstructing Inflammatory Myofibroblastic Tumor in a Child

2019 ◽  
Vol 11 (2) ◽  
pp. 226-228
Author(s):  
James Nawalaniec ◽  
Bhargava Mullapudi ◽  
Wen Jiang ◽  
Matthew Brigger ◽  
Howaida El-Said ◽  
...  
Keyword(s):  
Inflammatory Myofibroblastic Tumor ◽  
Soft Tissue Tumor ◽  
Pediatric Population ◽  
Lung Parenchyma ◽  
Left Main ◽  
Main Stem Bronchus ◽  
Extensive Resection ◽  
Inflammatory Myofibroblastic Tumors ◽  
Left Main Stem Bronchus ◽  
Fibroblastic Cells

Inflammatory myofibroblastic tumor (IMT) is a rare soft tissue tumor characterized by proliferation of fibroblastic cells associated with an inflammatory infiltrate. Inflammatory myofibroblastic tumors have a predilection for the pediatric population and are usually found in the lung parenchyma but rarely at the carina. They rarely metastasize but can be locally destructive. Surgical resection is the cornerstone of therapy, which results in excellent survival despite risk of local recurrence. We present the case of a nine-year-old girl with an IMT mass at the carina and obstructing the left main stem bronchus, requiring extensive resection and reconstruction.

scholarly journals Pediatric endobronchial inflammatory myofibroblastic tumor: a case report and review of the literature

2016 ◽  
Vol 6 (4) ◽  
Author(s):  
Leilei Liu ◽  
Xianglei Kong ◽  
Xiaoqian Lu ◽  
Dianbo Cao
Keyword(s):  
Inflammatory Myofibroblastic Tumor ◽  
Soft Tissue Tumor ◽  
Anatomical Site ◽  
Age Group ◽  
Review Of The Literature ◽  
Left Main ◽  
Main Stem Bronchus ◽  
Endobronchial Lesion ◽  
The Central Nervous System ◽  
Left Main Stem Bronchus

Inflammatory myofibroblastic tumor (IMT) belongs to the group of soft tissue tumor and could occur at any anatomical site from the central nervous system to gastrointestinal tract. The lung and abdomen are commonly affected sites, however, pulmonary IMT is predominantly located within the parenchyma rather than presenting as endobronchial lesion. IMTs may occur in any age group, but they are observed most commonly in children and adolescents. Here, we present a case of IMT arising from the left main stem bronchus in a 10-year-old girl.

scholarly journals Inflammatory Myofibroblastic Tumor of the Left Main Stem Bronchus

2006 ◽  
Vol 1 (7) ◽  
pp. 726-728 ◽  
Author(s):  
Salvatore Privitera ◽  
David M. Hwang ◽  
Gail E. Darling
Keyword(s):  
Inflammatory Myofibroblastic Tumor ◽  
Main Stem ◽  
Left Main ◽  
Main Stem Bronchus ◽  
Left Main Stem ◽  
Left Main Stem Bronchus

Rupture of the Left Main-stem Bronchus with a Polyvinylchloride Double-lumen Tube

CHEST Journal ◽  
1983 ◽  
Vol 83 (6) ◽  
pp. 928-929 ◽  
Author(s):  
Nelson A Burton ◽  
Stephen M. Fall ◽  
Thomas Lyons ◽  
Geoffrey M. Graeber
Keyword(s):  
Main Stem ◽  
Double Lumen Tube ◽  
Left Main ◽  
Double Lumen ◽  
Main Stem Bronchus ◽  
Lumen Tube ◽  
Left Main Stem ◽  
Left Main Stem Bronchus

PULMONARY EMPHYSEMA AND OTHER CARDIORESPIRATORY LESIONS AS PART OF THE MARFAN ABIOTROPHY

PEDIATRICS ◽  
1964 ◽  
Vol 33 (3) ◽  
pp. 356-366
Author(s):  
Robert P. Bolande ◽  
Arthur S. Tucker
Keyword(s):  
Connective Tissue ◽  
Left Atrium ◽  
Pulmonary Emphysema ◽  
Left Lung ◽  
Left Main ◽  
Main Stem Bronchus ◽  
Giant Left Atrium ◽  
Left Main Stem Bronchus ◽  
The Right ◽  
Chronic Pulmonary Emphysema

Seven cases of Marfan's syndrome are reviewed clinically, radiologically, and pathologically. Six of the seven cases showed evidence of pulmonary dysaeration: (a) Two of the cases showed compression of the left main-stem bronchus by a giant left atrium with atelectasis of the left lung and compensatory emphysema of the right lung. (b) Two of the cases showed evidence of diffuse chronic pulmonary emphysema. Three cases had bilateral apical bullae. (c) One of the cases developed pneumothorax. The lungs of the children with the Marfan syndrome show precocious maturation of the elastic stroma of the alveolar septae. The pathogenesis of emphysema is discussed in relationship to the Marfan abiotrophy of connective tissue.

Fiberbronchoscopic guidance was used to properly place the BB into the left main stem bronchus

ASVIDE ◽  
2021 ◽  
Vol 8 ◽  
pp. 038-038
Author(s):  
Hui Jia ◽  
Wen-Fei Tan ◽  
Hong Ma ◽  
Yong Cui
Keyword(s):  
Main Stem ◽  
Left Main ◽  
Main Stem Bronchus ◽  
Left Main Stem ◽  
Left Main Stem Bronchus

Short-length ligamentum arteriosum as a cause of congenital narrowing of the left main stem bronchus

2016 ◽  
Vol 51 (12) ◽  
pp. 1356-1361 ◽  
Author(s):  
Oliviero Sacco ◽  
Francesco Santoro ◽  
Elena Ribera ◽  
Gian Michele Magnano ◽  
Giovanni A. Rossi
Keyword(s):  
Short Length ◽  
Main Stem ◽  
Left Main ◽  
Main Stem Bronchus ◽  
Ligamentum Arteriosum ◽  
Left Main Stem ◽  
Left Main Stem Bronchus

Expiratory flow limitation in dogs with regional changes in lung mechanical properties

1988 ◽  
Vol 64 (1) ◽  
pp. 162-173 ◽  
Author(s):  
S. N. Mink ◽  
H. Greville ◽  
A. Gomez ◽  
J. Eng
Keyword(s):  
Mechanical Properties ◽  
Wave Speed ◽  
Main Stem ◽  
Left Main ◽  
Main Stem Bronchus ◽  
Left Main Stem ◽  
In Series ◽  
Left Main Stem Bronchus ◽  
Expiratory Flow ◽  
The Right

We examined maximum expiratory flow (Vmax) in two canine preparations in which regional changes in lung mechanical properties were produced. In one experiment serial bronchial obstructions were made to determine whether flow-limiting sites (choke points, CP) would occur in series. With the right lung tied off, constrictions were placed at the left lower lobar bronchus (LLL) and left main-stem bronchus. On deflation from total lung capacity, the obstructed LLL and nonobstructed left upper lobe (LUL) emptied into the obstructed left main-stem bronchus. Although a CP common to both lobes was identified at the main-stem obstruction, which limited total Vmax, we questioned whether there was also a CP at the lobar obstruction that fixed LLL flow. In that case the rate of LLL emptying would not be dependent on the presence of the common (i.e., central) CP and thus the flow contribution of the LUL. We found that when the LUL was removed, the LLL increased its rate of emptying. Thus a lobar CP did not fix LLL flow and CP did not occur in series. In a second experiment emphysema was produced in the left lung to reduce lung recoil, whereas the right lung was normal. CP were identified at approximately lobar bronchi of each lung, and the lungs were emptied at different rates. A CP common to both lungs was not identified. Our results indicate that in localized lung disease, if flows from the different regions are high enough, then wave speed is reached in proximal airways, and a CP occurs centrally rather than peripherally. On the other hand, if flows are low, then wave speed is reached peripherally and a CP common to all lung regions does not occur.

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