scholarly journals Patent urachus and bladder outflow obstruction—chance or consequence? A study of a cohort of patients with complete patent urachus presenting to a tertiary urological center and a review of literature

2021 ◽  
Vol 17 (1) ◽  
Author(s):  
Heba Taher ◽  
Sharmila Ramnarine ◽  
Naima Smeulders ◽  
Divyesh Desai ◽  
Imran Mushtaq ◽  
...  

Abstract Background To explore the association between patent urachus and bladder outflow obstruction (BOO). A retrospective review of patient records over a 35-year period (1983–2018) with complete patent urachus was performed. Antenatal ultrasound findings were noted, and postnatal investigations included ultrasound (US), micturating cystourethrogram (MCUG), functional nuclear medicine scans (MAG3, DTPA, and DMSA), and serum creatinine. Associated anomalies and management in all patients were analyzed. Results Sixty-six patients with all types of urachal remnants were identified of whom only 16 had a patent urachus. All presented clinically with a discharging umbilicus, 10/16 confirmed on MCUG and 4 had umbilical cord cysts on antenatal US. Twenty-five percent had associated bladder outlet obstruction (BOO): etiologies included atresia of posterior urethra, congenital urethral hypoplasia, urethral atresia with prune belly syndrome, and sacrococcygeal teratoma. Vesicoureteral reflux (VUR) was confirmed in 37%, and four of them had bladder outlet obstruction (BOO). Conclusion With patent urachus, bladder outflow obstruction occurs in the minority. Based on our findings, we commend US and cystogram to document VUR. The isolated PU should be treated nonoperatively up to a year of age. Renal function should be checked with the finding of VUR. The etiopathogenesis of the condition remains uncertain.

2013 ◽  
Vol 32 (3) ◽  
pp. 257-258
Author(s):  
Zaheer Hasan ◽  
Bindey Kumar ◽  
Prem Kumar

Bladder neck and posterior urethra are common sites for obstructive uropathy in children. Diverticula of posterior urethra are rare cause of obstruction in children. A six year old boy presented with features of bladder outflow obstruction since birth. Ultrasound findings were suggestive of posterior urethral valve. Micturating cystourethrogram and endoscopic examinations revealed posterior urethral diverticulum which was placed dorsally. Diverticulectomy and reconstruction of urethra was performed by midline perineal incision. This report emphasizes that a posterior urethral diverticulum may be considered in those cases where features are suggestive of posterior urethral valve bladder outflow obstruction. DOI: http://dx.doi.org/10.3126/jnps.v32i3.5914 J. Nepal Paediatr. SocVol.32(3) 2012 257-258


Author(s):  
Christopher R. Chapple ◽  
Altaf Mangera

Bladder outflow obstruction (BOO) may occur due to several underlying causes in both men and women. It is not possible to diagnose bladder outlet obstruction on a history alone. It can be suspected based on the use of a flow rate but can only be diagnosed using pressure flow urodynamics. In this chapter, we discuss the aetiology, pathophysiology, and investigation of BOO. We emphasize the importance of a complete history, examination, and investigations with investigations such as flow rate and voiding cystometry, in addition to standard tests including urinalysis and a bladder diary. The management of the underlying disorder responsible for the BOO symptoms is discussed in the relevant chapters separately.


2021 ◽  
Vol 10 (21) ◽  
pp. 4920
Author(s):  
Francisco E. Martins ◽  
Henriette Veiby Holm ◽  
Nicolaas Lumen

Bladder outlet obstruction following treatment of pelvic cancer, predominantly prostate cancer, occurs in 1–8% of patients. The high incidence of prostate cancer combined with the long-life expectancy after treatment has increased concerns with cancer survivorship care. However, despite increased oncological cure rates, these adverse events do occur, compromising patients’ quality of life. Non-traumatic obstruction of the posterior urethra and bladder neck include membranous and prostatic urethral stenosis and bladder neck stenosis (also known as contracture). The devastated bladder outlet can result from benign conditions, such as neurogenic dysfunction, trauma, iatrogenic causes, or more frequently from complications of oncologic treatment, such as prostate, bladder and rectum. Most posterior urethral stenoses may respond to endoluminal treatments such as dilatation, direct vision internal urethrotomy, and occasionally urethral stents. Although surgical reconstruction offers the best chance of durable success, these reconstructive options are fraught with severe complications and, therefore, are far from being ideal. In patients with prior RT, failed reconstruction, densely fibrotic and/or necrotic and calcified posterior urethra, refractory incontinence or severe comorbidities, reconstruction may not be either feasible or recommended. In these cases, urinary diversion with or without cystectomy is usually required. This review aims to discuss the diagnostic evaluation and treatment options for patients with bladder outlet obstruction with a special emphasis on patients unsuitable for reconstruction of the posterior urethra and requiring urinary diversion.


1990 ◽  
Vol 25 (3) ◽  
pp. 342-345 ◽  
Author(s):  
Ricardo Gonzalez ◽  
Yuri Reinberg ◽  
Barbara Burke ◽  
Thomas Wells ◽  
Robert L. Vernier

Author(s):  
Aron Chakera ◽  
William G. Herrington ◽  
Christopher A. O’Callaghan

Oliguria can be caused by any factor that affects renal function, or the free passage of urine down the urinary tract. Complete anuria most commonly occurs in men as a consequence of bladder outlet obstruction from an enlarged prostate. It can also arise in patients who have a single functioning kidney which then becomes obstructed or loses its vascular supply. Oliguria occurs commonly in hospitalized patients, is usually secondary to impaired renal perfusion, and is often predictable. The elderly and more unwell patients, for example, those in critical care settings, are most at risk. The presence of oliguria tends to reflect the severity of the underlying disease processes. The commonest cause of complete anuria is bladder outflow obstruction from an enlarged prostate. This may be precipitated by prostatitis or constipation in a patient with benign prostatic hypertrophy. In catheterized patients, a blocked catheter must be excluded.


2005 ◽  
Vol 173 (4S) ◽  
pp. 394-394
Author(s):  
Martha A. Hass ◽  
Robert M. Levin ◽  
William Connors ◽  
Alma Birnboim

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