Obesity and the Cardiovascular Health Effects of Fine Particulate Air Pollution: a Review

2014 ◽  
Vol 2014 (1) ◽  
pp. 1502
Author(s):  
Scott Weichenthal* ◽  
Jane A Hoppin ◽  
Francois Reeves
Obesity ◽  
2014 ◽  
Vol 22 (7) ◽  
pp. 1580-1589 ◽  
Author(s):  
Scott Weichenthal ◽  
Jane A. Hoppin ◽  
Francois Reeves

BMJ ◽  
2019 ◽  
pp. l6609 ◽  
Author(s):  
Matthew Loxham ◽  
Donna E Davies ◽  
Stephen T Holgate

2006 ◽  
Vol 56 (10) ◽  
pp. 1368-1380 ◽  
Author(s):  
Judith C. Chow ◽  
John G. Watson ◽  
Joe L. Mauderly ◽  
Daniel L. Costa ◽  
Ronald E. Wyzga ◽  
...  

1997 ◽  
Vol 4 (6) ◽  
pp. 323-328
Author(s):  
Patrick A Hessel ◽  
John R Goldsmith ◽  
H-Erich Wichmann ◽  
William E Wilson ◽  
Colin L Soskolne

The health effects of particulate air pollution were highlighted at the Eighth Annual Conference of the International Society for Environmental Epidemiology, held in Edmonton, Alberta in August 1996. Despite consistent evidence for adverse respiratory and cardiovascular health effects related to particulate air pollution, there are significant gaps in the knowledge of the mechanisms whereby particulate air pollution affects human health. Questions regarding the appropriate measure of dose for assessing exposures relevant to health outcomes and the methods used to analyze dose-response data remain unanswered. Health effects have been demonstrated across the range of exposures that have been examined, and further research in low exposure settings is necessary to explore the lower end of the dose-response curve. Although a significant body of literature has been generated, comprehensive risk assessments have not been undertaken. Examination of the chronic effects of particulate air pollution and identification of high risk populations are necessary. Although there are significant unanswered questions regarding the health effects of particulate air pollution, the available information suggests that particulate air pollution at levels consistent with current standards is associated with measurable health effects.


Circulation ◽  
2021 ◽  
Vol 143 (Suppl_1) ◽  
Author(s):  
Fan He ◽  
Julio Fernandez-Mendoza ◽  
Jeff D Yanosky ◽  
Vernon M Chinchilli ◽  
Laila Al-shaar ◽  
...  

Introduction: Sleep deprivation and large sleep variability are potential risk factors for obesity and cardiovascular diseases. While it is plausible that particulate air pollution may contribute to these unfavorable sleep patterns, very few studies have been conducted to assess the association between fine particulate (PM 2.5 ) air pollution and objectively measured sleep duration and its variability in an adolescent U.S. population. Hypothesis: We tested the hypothesis that higher individual-level short-term PM 2.5 exposure is associated with shorter sleep duration and higher sleep variability among adolescents. Methods: We analyzed the available data collected from 421 adolescents who participated in the follow-up examination of the population-based Penn State Child Cohort (PSCC) study. To estimate individual-level short-term PM 2.5 exposure, a personal nephelometer (Thermo pDR-1200) was used to measure real-time PM 2.5 concentration for 24 hours from the study participants. The 24-hour mean PM 2.5 concentration was used to quantify the short-term PM 2.5 exposure. To obtain objectively-measured habitual sleep duration (HSD) and habitual sleep variability (HSV), an actigraphy (GT3X+) was used to collect sleep data for 7 consecutive nights, including 1 night in parallel with the PM 2.5 monitoring and 6 nights thereafter. HSD and HSV were calculated as the intra-individual mean and standard deviation (SD) of the 7-night sleep duration, respectively. Participants with < 5 nights (70% of 7 nights) of data were excluded from the analyses. The associations between the individual-level PM 2.5 exposure and HSD/HSV were evaluated by using multi-variable adjusted linear regression models, controlling for age, race, sex, BMI percentile, environmental temperature, and relative humidity. Results: The mean (SD) age of the study population was 16.9 (2.2) years. The study sample consisted of 54% males and 78% whites. The 24-hour mean (SD) of PM 2.5 concentration was 16.9 (26.8) μg/m 3 , while the average HSD and HSV were 7.0 (0.9) hours and 1.2 (0.6) hours, respectively. We observed that a 10 μg/m 3 increase in the 24-hour mean PM 2.5 was associated with significantly lower HSD [β (SE): -0.06 (0.03) hours, p=0.02] and larger HSV [β (SE): 0.04 (0.02) hours, p=0.04]. The effect sizes were approximately 7% of their respective SDs. Conclusion: Individual-level short-term PM 2.5 exposure is associated with objective-measured shorter sleep duration and higher night-to-night sleep variability among U.S. adolescents. These observed associations suggest that particulate air pollution exposure in early life may impact habitual sleep pattern, which may in turn be associated with the risks of obesity and cardiovascular diseases in later life.


2007 ◽  
Vol 115 (1) ◽  
pp. 13-19 ◽  
Author(s):  
Bart Ostro ◽  
Wen-Ying Feng ◽  
Rachel Broadwin ◽  
Shelley Green ◽  
Michael Lipsett

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