Influence of Strength Training Background on Postactivation Potentiation Response

2011 ◽  
Vol 25 (9) ◽  
pp. 2496-2502 ◽  
Author(s):  
Mauro AB Batista ◽  
Hamilton Roschel ◽  
Renato Barroso ◽  
Carlos Ugrinowitsch ◽  
Valmor Tricoli
2018 ◽  
Vol 4 (63) ◽  
Author(s):  
Nerijus Masiulis ◽  
Albertas Skurvydas ◽  
Sigitas Kamandulis ◽  
Lina Kamandulienė

Repeated activation of muscle induces processes resulting in decreased performance (fatigue) as well as enhanced performance (postactivation potentiation, (PAP)). This implies that at any time during contraction, fatiguing effects are being countered by potentiation effects, and vise versa. Therefore, which of the processes will be prevalent during and after explosive strength training is not clear.The purpose of this investigation was to study the acute neuromuscular responses to one explosive strength training session. Eleven healthy untrained men (aged 22—35 years) performed explosive strength training session of six sets (fi ve repetitions each) of the unilateral isometric contractions at an angle of 90 degrees in the knee. The contractility of the muscle was monitored via the electrically evoked contractions at 1, 20, and 50 Hz (P 1, P 20, and P 50, re-spectively) before (Ini), after the fi rst and sixth sets as well as during the 5 and 30 min recovery period (A 5 and A 30, respectively). Contraction time (CT) and relaxation time (RT) of a single twitch (P1) of quadriceps was registered. Maximal voluntary contraction (MVC) force as well force developed during 100 ms (MVC 0-100ms ) was also determined. The ratio of P 20 / P 50 kinetics after exercise was used for the evaluation of low-frequency fatigue (LFF). There was statistically signifi cant repetition effect observed on MVC (p = 0.045) and MVC 0-100ms  (p = 0.012). After the fi rst set there was a signifi cant increase in muscle force induced by very low (1 Hz) and low (20 Hz) stimulation frequencies and did not change during all explosive strength training session (p < 0.05). The ratio of P 20 / P 50 recorded after the fi rst set increased signifi cantly (p < 0.05), however 30-min after the explosive strength training session it was signifi cantly decrease in P 20 / P50 ratio compared to its Ini level (p < 0.05). The present study showed that potentia-tion increases P 20 / P 50 ratio during the explosive strength training session, however the subsequent (after 30 min of recovery) decline in P 20 / P 50 ratio is an outcome of diminishing infl uence of potentiation on the background of persistent LFF. Therefore, when muscles are potentiated, it may seem as if no LFF is present.Keywords: explosive strength training, low-frequency fatigue, maximum voluntary contraction, post-activation potentiation.


2005 ◽  
Vol 39 (2) ◽  
pp. 23
Author(s):  
NORRA MACREADY
Keyword(s):  

2020 ◽  
Vol 90 (1-2) ◽  
pp. 113-123
Author(s):  
Ines Schadock ◽  
Barbara G. Freitas ◽  
Irae L. Moreira ◽  
Joao A. Rincon ◽  
Marcio Nunes Correa ◽  
...  

Abstract. β-hydroxy-β-methyl butyrate (HMB) is a bioactive metabolite derived from the amino acid leucine, usually applied for muscle mass increase during physical training, as well as for muscle mass maintenance in debilitating chronic diseases. The hypothesis of the present study is that HMB is a safe supplement for muscle mass gain by strength training. Based on this, the objective was to measure changes in body composition, glucose homeostasis and hepatic metabolism of HMB supplemented mice during strength training. Two of four groups of male mice (n = 6/group) underwent an 8-week training period session (climbing stairs) with or without HMB supplementation (190 mg/kgBW per day). We observed lower body mass gain (4.9 ± 0.43% versus 1.2 ± 0.43, p < 0.001) and increased liver mass (40.9 ± 0.9 mg/gBW versus 44.8 ± 1.3, p < 0.001) in the supplemented trained group compared with the non-supplemented groups. The supplemented trained group had an increase in relative adipose tissue mass (12.4 ± 0.63 mg/gBW versus 16.1 ± 0.88, P < 0.01) compared to the non-supplemented untrained group, and an increase in fasting blood glucose (111 ± 4.58 mg/dL versus 122 ± 3.70, P < 0.05) and insulin resistance (3.79 ± 0.19 % glucose decay/min versus 2.45 ± 0.28, P < 0.05) comparing with non-supplemented trained group. Adaptive heart hypertrophy was observed only in the non-supplemented trained group (4.82 ± 0.05 mg/gBW versus 5.12 ± 0.13, P < 0.05). There was a higher hepatic insulin-like growth factor-1 expression (P = 0.002) in supplemented untrained comparing with non-supplemented untrained group. Gene expression of gluconeogenesis regulatory factors was increased by training and reduced by HMB supplementation. These results confirm that HMB supplementation associated with intensive training protocol drives changes in glucose homeostasis and liver metabolism in mice.


1991 ◽  
Vol 10 (1) ◽  
pp. 33-62 ◽  
Author(s):  
Nicholas A. DiNubile
Keyword(s):  

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