Intrauterine Growth Retardation

1986 ◽  
Vol 8 (4) ◽  
pp. 107-114
Author(s):  
Joseph B. Warshaw

Intrauterine growth retardation can result from a variety of environmental and genetic influences on fetal growth. The sequelae of intrauterine growth retardation resulting from impairment of nutrient flow include low birth weight with sparing of brain growth, polycythemia, and hypoglycemia resulting from decreased storage fuels and defective gluconeogenesis. Available data suggest that the vast majority of nutritionally growthretarded infants have normal postnatal development without significant difference in IQ or neurologic scores from normal infants. The outcome of infants in whom there is decreased growth potential relates to the condition underlying growth retardation and may, of course, result in later severe handicap. Infants with nutritional intrauterine growth retardation may exhibit decreased fetal size and sparing of brain growth as an adaptive stage in the presence of lower oxygen and substrate availability. Under such conditions, a smaller size may decrease substrate and oxygen needs and improve outcome. All infants with intrauterine growth retardation, however, require follow-up and careful developmental evaluation.

PEDIATRICS ◽  
1985 ◽  
Vol 76 (6) ◽  
pp. 998-999
Author(s):  
JOSEPH B. WARSHAW

Intrauterine growth retardation can result from a variety of environmental or genetic influences on fetal growth.1 The sequelae of intrauterine growth retardation resulting from impairment of nutrient flow from mother to fetus are well known and include low birth weight with sparing of brain growth, polycythemia, and hypoglycemia resulting from decreased storage fuels and defective gluconeogenesis. Despite the generally held assumption that nutritionally related intrauterine growth retardation (either maternal malnutrition or impaired uteroplacental blood flow) represents a serious pathologic insult to the fetus, the available data suggest that the vast majority of infants with intrauterine growth retardation have normal development without significant differences in IQ or neurologic scores from normal infants.2


2000 ◽  
Vol 15 (12) ◽  
pp. 781-786 ◽  
Author(s):  
Yael Leitner ◽  
Aviva Fattal-Valevski ◽  
Ronny Geva ◽  
Haim Bassan ◽  
Edith Posner ◽  
...  

1999 ◽  
Vol 14 (11) ◽  
pp. 724-727 ◽  
Author(s):  
Aviva Fattal-Valevski ◽  
Yael Leitner ◽  
Miriam Kutai ◽  
Edith Tal-Posener ◽  
Abraham Tomer ◽  
...  

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