scholarly journals Ocular Flutter in Hyperosmolar Hyperglycemic State during Treatment of Malignant Cerebral Edema

2020 ◽  
Vol 38 (4) ◽  
pp. 293-297
Author(s):  
Yeoung Deok Seo ◽  
Seongho Park
Keyword(s):  
Cerebral Infarction ◽  
Movement Disorder ◽  
Eye Movement ◽  
Cerebral Edema ◽  
Clinical Settings ◽  
Metabolic State ◽  
Ocular Flutter ◽  
New Information ◽  
Hyperosmolar Hyperglycemic State

Ocular flutter (OF) is a rare eye movement disorder with horizontal saccadic oscillations without inter-saccadic intervals. The OF can occur in various clinical settings. A 83-year-old female showed the OF in hyperosmolar hyperglycemic state (HHS) following malignant cerebral infarction. The pathophysiology of the OF is still unclear, but this case provides a new information that the OF can occur in the context of toxic-metabolic state such as HHS.

scholarly journals Ocular Flutter in Parainfectious Meningoencephalitis: A Case Report

2021 ◽  
Vol 20 (3) ◽  
pp. 113-117
Author(s):  
In-Ho Yoon ◽  
Seung-Hoon Yun ◽  
Bong-Hui Kang
Keyword(s):  
Case Report ◽  
Movement Disorder ◽  
Eye Movement ◽  
Paraneoplastic Syndrome ◽  
Immunoglobulin Therapy ◽  
Metabolic Dysfunction ◽  
Ocular Flutter ◽  
Clinical Conditions

Ocular flutter is a rare, horizontal eye movement disorder characterized by intermittent bursts of conjugate horizontal saccades without intersaccadic intervals. It can occur in various clinical conditions such as metabolic dysfunction, infection and paraneoplastic syndrome. Herein, a 50-year-old male showed ocular flutter in parainfectious meningoencephalitis and immunoglobulin therapy led to an improvement of symptoms. This case can improve the understanding of the pathological mechanisms of ocular flutter.

Bifurcation theory explains waveform variability in a congenital eye movement disorder

2008 ◽  
Vol 26 (2) ◽  
pp. 321-329 ◽  
Author(s):  
Andrea K. Barreiro ◽  
Jared C. Bronski ◽  
Thomas J. Anastasio
Keyword(s):  
Movement Disorder ◽  
Eye Movement ◽  
Bifurcation Theory

CONTROLLED TRIAL OF GLYCEROL VERSUS DEXAMETHASONE IN THE TREATMENT OF CEREBRAL (EDEMA IN ACUTE CEREBRAL INFARCTION

The Lancet ◽  
1975 ◽  
Vol 305 (7915) ◽  
pp. 1049-1051 ◽  
Author(s):  
V. Gilsanz ◽  
J. Buencuerpo ◽  
J.L. Rebollar ◽  
M.T. Chantres
Keyword(s):  
Cerebral Infarction ◽  
Cerebral Edema ◽  
Controlled Trial ◽  
Acute Cerebral Infarction

Repeated Hypothermia for Rebound Cerebral Edema after Therapeutic Hypothermia in Malignant Cerebral Infarction

2013 ◽  
Vol 28 (3) ◽  
pp. 221 ◽  
Author(s):  
Jeong-Ho Hong ◽  
Jin-Heon Jeong ◽  
Jun Young Chang ◽  
Min-Ju Yeo ◽  
Han-Yeong Jeong ◽  
...  
Keyword(s):  
Cerebral Infarction ◽  
Therapeutic Hypothermia ◽  
Cerebral Edema

Seizure-induced Cerebral Edema Mimicking Multiple Cerebral Infarction

2015 ◽  
Vol 33 (1) ◽  
pp. 54-56 ◽  
Author(s):  
Tae-Won Yang ◽  
Wonsik Nam ◽  
Seungnam Son ◽  
Soo-Kyoung Kim ◽  
Heeyoung Kang ◽  
...  
Keyword(s):  
Cerebral Infarction ◽  
Cerebral Edema ◽  
Multiple Cerebral Infarction

Web based decision support system for eye movement disorder diagnosis

2016 ◽  
Vol 10 (1) ◽  
pp. 59-68
Author(s):  
He Xu ◽  
Yan Xu ◽  
Hongpeng Yu ◽  
Xunda Lv ◽  
Ozoemena A. Ani
Keyword(s):  
Decision Support ◽  
Decision Support System ◽  
Movement Disorder ◽  
Eye Movement ◽  
Support System ◽  
Web Based

The roles of MMP-9/TIMP-1 in cerebral edema following experimental acute cerebral infarction in rats

2013 ◽  
Vol 550 ◽  
pp. 168-172 ◽  
Author(s):  
Dan-Dong Li ◽  
Jin-Ning Song ◽  
Huan Huang ◽  
Xiao-Ye Guo ◽  
Ji-Yang An ◽  
...  
Keyword(s):  
Cerebral Infarction ◽  
Cerebral Edema ◽  
Acute Cerebral Infarction

scholarly journals Two Cases of Oculopharyngeal Muscular Dystrophy in Brothers with Ptosis and Eye Movement Disorder

2020 ◽  
Vol 61 (5) ◽  
pp. 575-581
Author(s):  
Seulki Bang ◽  
Sanghyu Nam ◽  
Jin San Lee ◽  
Sung-Hye Park ◽  
Min Seok Kang ◽  
...  
Keyword(s):  
Muscular Dystrophy ◽  
Movement Disorder ◽  
Eye Movement ◽  
Oculopharyngeal Muscular Dystrophy

scholarly journals Pardon the interruption: saccadic inhibition enables the rapid deployment of alternate oculomotor plans

2018 ◽  
Author(s):  
Emilio Salinas ◽  
Terrence R. Stanford
Keyword(s):  
Eye Movement ◽  
Visual Information ◽  
Oculomotor System ◽  
Sudden Onset ◽  
Physiological Data ◽  
Alternative Plan ◽  
New Information ◽  
Saccadic Inhibition ◽  
Simulation Results ◽  
Rapid Deployment

Diverse psychophysical and neurophysiological results show that oculomotor networks are continuously active, such that plans for making the next eye movement are always ongoing. So, when new visual information arrives unexpectedly, how are those plans affected? At what point can the new information start guiding an eye movement, and how? Here, based on modeling and simulation results, we make two observations that are relevant to these questions. First, we note that many experiments, including those investigating the phenomenon known as “saccadic inhibition,” are consistent with the idea that sudden-onset stimuli briefly interrupt the gradual rise in neural activity associated with the preparation of an impending saccade. And second, we show that this stimulus-driven interruption is functionally adaptive, but only if perception is fast. In that case, putting on hold an ongoing saccade plan toward location A allows the oculomotor system to initiate a concurrent, alternative plan toward location B (where a stimulus just appeared), deliberate (briefly) on the priority of each target, and determine which plan should continue. Based on physiological data, we estimate that the actual advantage of this strategy, relative to one in which any plan once initiated must be completed, is of several tens of milliseconds.

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