FACTORS INFLUENCING TRENDS OF POLYCHLORINATED NAPHTHALENES AND OTHER DIOXIN-LIKE COMPOUNDS IN LAKE TROUT (SALVELINUS NAMAYCUSH) FROM LAKE ONTARIO, NORTH AMERICA (1979–2004)

2009 ◽  
Vol 28 (5) ◽  
pp. 921 ◽  
Author(s):  
Sarah B. Gewurtz ◽  
Rocsana Lega ◽  
Patrick W. Crozier ◽  
D. Michael Whittle ◽  
Laila Fayez ◽  
...  



Author(s):  
Alexander Gatch ◽  
Dimitry Gorsky ◽  
Zy Biesinger ◽  
Eric Bruestle ◽  
Kelley Lee ◽  
...  


2010 ◽  
Vol 408 (7) ◽  
pp. 1725-1730 ◽  
Author(s):  
Charles P. Madenjian ◽  
Michael J. Keir ◽  
D. Michael Whittle ◽  
George E. Noguchi


2013 ◽  
Vol 32 (6) ◽  
pp. 1376-1381 ◽  
Author(s):  
Gregg T. Tomy ◽  
Ed Sverko ◽  
Vince Palace ◽  
Bruno Rosenberg ◽  
Robert McCrindle ◽  
...  




1992 ◽  
Vol 49 (6) ◽  
pp. 1086-1096 ◽  
Author(s):  
U. Borgmann ◽  
D. M. Whittle

Lake trout (Salvelinus namaycush) ingestion rates in a bioenergetics and contaminant dynamics model were estimated directly from contaminant concentrations in lake trout and their prey, rather than from the sum of growth and predicted metabolism. Elimination rates for PCB and DDE, but not for mercury, were dependent on either body mass or lipid content. Concentrations in lake trout responded rapidly to changes in concentration of their prey. This was due primarily to growth dilution and not contaminant elimination, especially for DDE and PCB. Changes in lipid concentrations, therefore, have only minor effects on final concentrations in lake trout, and it is not appropriate to lipid normalize PCB or DDE concentrations when examining trends in whole-body concentrations for this species. Concentrations of PCBs and lipids have declined in lake trout from 1977 to 1988. The drop in PCB concentrations is probably not caused primarily by the lowered lipid concentrations but is the result of either a reduction in feeding rates and improved growth efficiencies, a reduction in PCB concentrations in alewife (Alosa pseudoharengus), or an undocumented change in prey selection. Models based on chemical kinetics across the gastrointestinal tract are more consistent with observed data than models based on a constant contaminant assimilation rate and direct excretion.



1988 ◽  
Vol 45 (8) ◽  
pp. 1406-1410 ◽  
Author(s):  
Roger A. Bergstedt ◽  
Clifford P. Schneider

During 1982–85, 89 dead fake trout (Salvelinus namaycush) were recovered with bottom trawls in U.S. waters of Lake Ontario: 28 incidentally during four annual fish-stock assessment surveys and 61 during fall surveys for dead fish. During the assessment surveys, no dead lake trout were recovered in April–June, one was recovered in August, and 27 were recovered in October or November, implying that most mortality from causes other than fishing occurred in the fall. The estimated numbers of dead lake trout between the 30- and 100-m depth contours in U.S. waters ranged from 16 000 (0.08 carcass/ha) in 1983 to 94 000 (0.46 carcass/ha) in 1982. Of 76 carcasses fresh enough to enable recognition of sea lamprey (Petromyzon marinus) wounds, 75 bore fresh wounds. Assuming that sea lamprey wounding rates on dead fish were the same as on live ones of the same length range (430–740 mm), the probability of 75 of the 76 dead lake trout bearing sea lamprey wounds was 3.5 × 10−63 if death was independent of sea lamprey attack, thus strongly implicating sea lampreys as the primary cause of death of fish in the sample. The recovery of only one unwounded dead lake trout also suggested that natural mortality from causes other than sea lamprey attacks is negligible.





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