Arterial Duct Stenting in Congenital Heart Disease with Duct-Dependent Pulmonary Circulation

2010 ◽  
Vol 6 (3) ◽  
pp. 183-191
Author(s):  
Giuseppe Santoro ◽  
Biagio Castaldi ◽  
Gianpiero Gaio ◽  
Maria Teresa Palladino ◽  
Carola Iacono ◽  
...  
2009 ◽  
Vol 74 (7) ◽  
pp. 1072-1076 ◽  
Author(s):  
Giuseppe Santoro ◽  
Maria Teresa Palladino ◽  
Giovanbattista Capozzi ◽  
Carola Iacono ◽  
Maria Giovanna Russo ◽  
...  

2015 ◽  
Vol 8 (12) ◽  
pp. 1626-1632 ◽  
Author(s):  
Giuseppe Santoro ◽  
Gianpiero Gaio ◽  
Giovanbattista Capozzi ◽  
Luca Giugno ◽  
Maria Teresa Palladino ◽  
...  

Author(s):  
Tiffany Camp ◽  
Richard S. Figliola ◽  
Timothy A. Conover ◽  
T.-Y. Hsia

Pulmonary insufficiency is one of the consequences of congenital heart disease, and currently no permanent solution exists. The concept of a motionless diode valve to regulate the pulmonary circulation has been proposed and previously studied. The diode valve has shown the ability to regulate flow with levels of regurgitation and pressure gradient that are acceptable within the pulmonary circulation [1].


1995 ◽  
Vol 5 (2) ◽  
pp. 202-203 ◽  
Author(s):  
R. John Madar ◽  
Tim J.D. Donaldson ◽  
Stewart Hunter

the use of prostaglandians in maintaining the patency of the arterial duct in congenital heart disease is well established. Intravenous1-3 and ora12–4 administration has been used, although for acute use intravenous and possibly intraosseous5 routes are favored. Both prostaglandin E1 (alprostadil—ProstinVR: Upjohn)1,4 and prostaglandin E2 (dinoprostone—Prostin E2: Upjohn)2,3 are used for this purpose, although only prostaglandin E1 is licensed for this indication in the United Kingdom. Prostaglandin E1 costs approximately 8 times more than prostaglandin E2 (£56.96 versus £7.43 per vial).


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